Paolo Bonvini
- Molecular Biology top 10%
- Oncology
- Pulmonary and Respiratory Medicine
- Pathology and Forensic Medicine top 10%
- Immunology
- Co-authors
- Angelo RosolenElisa ZorziJane B. TrepelLen NeckersEdward G. MimnaughBrunangelo FaliniPhuongmai NguyenStefano Moro
- Topics
- Ubiquitin and proteasome pathways (10 papers)Sarcoma Diagnosis and Treatment (8 papers)Lymphoma Diagnosis and Treatment (6 papers)
- Partner nations
- ItalyUnited StatesGermany
In The Last Decade
Paolo Bonvini
34 papers receiving 1.0k citations
Peers
Comparison fields: 5 of 84
- Molecular Biology 721
- Oncology 229
- Pulmonary and Respiratory Medicine 133
- Pathology and Forensic Medicine 126
- Immunology 105
Countries citing papers authored by Paolo Bonvini
This map shows the geographic impact of Paolo Bonvini's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Paolo Bonvini with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Paolo Bonvini more than expected).
Fields of papers citing papers by Paolo Bonvini
This network shows the impact of papers produced by Paolo Bonvini. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Paolo Bonvini. The network helps show where Paolo Bonvini may publish in the future.
Co-authorship network of co-authors of Paolo Bonvini
This figure shows the co-authorship network connecting the top 25 collaborators of Paolo Bonvini. A scholar is included among the top collaborators of Paolo Bonvini based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Paolo Bonvini. Paolo Bonvini is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 13 | |
| 2 | 4 | |
| 3 | 14 | |
| 4 | 3 | |
| 5 | 2 | |
| 6 | 9 | |
| 7 | 14 | |
| 8 | 8 | |
| 9 | 33 | |
| 10 | 6 | |
| 11 | 12 | |
| 12 | 21 | |
| 13 | 18 | |
| 14 | 4 | |
| 15 | 8 | |
| 16 | Geldanamycin abrogates ErbB2 association with proteasome-resistant beta-catenin in melanoma cells, increases beta-catenin-E-cadherin association, and decreases beta-catenin-sensitive transcription. | 63 |
| 17 | 12 | |
| 18 | 78 | |
| 19 | 2 | |
| 20 | 51 |
About Paolo Bonvini
Paolo Bonvini is a scholar working on Oncology, Pulmonary and Respiratory Medicine and Pathology and Forensic Medicine, having authored 34 papers that have together received 1.0k indexed citations. Recurring topics across this work include Ubiquitin and proteasome pathways (10 papers), Sarcoma Diagnosis and Treatment (8 papers) and Lymphoma Diagnosis and Treatment (6 papers). The work is most often cited by research in Molecular Biology (721 citations), Oncology (229 citations) and Pathology and Forensic Medicine (126 citations). Paolo Bonvini has collaborated with scholars based in Italy, United States and Germany. Frequent co-authors include Angelo Rosolen, Elisa Zorzi, Jane B. Trepel, Len Neckers, Edward G. Mimnaugh, Brunangelo Falini, Phuongmai Nguyen, Stefano Moro, Giorgio Cozza and Giuseppe Zagotto. Their work appears in journals such as Journal of Biological Chemistry, Blood and PLoS ONE.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.