J. Robin Highley

6.2k total citations · 1 hit paper
63 papers, 3.9k citations indexed

About

J. Robin Highley is a scholar working on Neurology, Genetics and Cognitive Neuroscience. According to data from OpenAlex, J. Robin Highley has authored 63 papers receiving a total of 3.9k indexed citations (citations by other indexed papers that have themselves been cited), including 36 papers in Neurology, 21 papers in Genetics and 17 papers in Cognitive Neuroscience. Recurrent topics in J. Robin Highley's work include Amyotrophic Lateral Sclerosis Research (31 papers), Neurogenetic and Muscular Disorders Research (20 papers) and Advanced Neuroimaging Techniques and Applications (11 papers). J. Robin Highley is often cited by papers focused on Amyotrophic Lateral Sclerosis Research (31 papers), Neurogenetic and Muscular Disorders Research (20 papers) and Advanced Neuroimaging Techniques and Applications (11 papers). J. Robin Highley collaborates with scholars based in United Kingdom, United States and Australia. J. Robin Highley's co-authors include Pamela J. Shaw, Paul G. Ince, Janine Kirby, Margaret M. Esiri, Brendan McDonald, Timothy J. Crow, Mary A. Walker, Johnathan Cooper‐Knock, Stephen B. Wharton and Brian Herron and has published in prestigious journals such as The Lancet, SHILAP Revista de lepidopterología and PLoS ONE.

In The Last Decade

J. Robin Highley

62 papers receiving 3.9k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Pathological TDP‐43 distinguishes sporadic amyotrophic lateral sclerosis from amyotrophic lateral sclerosis with SOD1 mutations

2007 761 citations Ian R. Mackenzie, Eileen H. Bigio et al. Annals of Neurology profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
J. Robin Highley United Kingdom	33	2.2k	1.3k	1.2k	761	620	63	3.9k
Gesine Paul Sweden	30	1.8k 0.8×	375 0.3×	1.3k 1.1×	443 0.6×	905 1.5×	59	4.1k
Alexander M. Rossor United Kingdom	29	1.1k 0.5×	335 0.3×	908 0.8×	438 0.6×	614 1.0×	81	3.3k
Doris Lenartz Germany	33	2.7k 1.2×	376 0.3×	549 0.5×	1.4k 1.9×	996 1.6×	60	4.9k
G. V. Sawle United Kingdom	33	3.5k 1.6×	257 0.2×	1.3k 1.1×	895 1.2×	535 0.9×	53	6.1k
Matthew D. Cykowski United States	27	787 0.4×	217 0.2×	362 0.3×	360 0.5×	318 0.5×	59	2.0k
Catarina Freitas Portugal	23	335 0.2×	263 0.2×	1.6k 1.3×	638 0.8×	1.2k 1.9×	48	3.7k
Jaap J. Plomp Netherlands	40	2.5k 1.1×	178 0.1×	2.6k 2.2×	236 0.3×	305 0.5×	91	6.1k
Paula Coutinho Portugal	31	1.6k 0.7×	326 0.3×	2.8k 2.4×	232 0.3×	928 1.5×	87	4.7k
Rebecca Schüle Germany	35	600 0.3×	581 0.5×	1.2k 1.0×	282 0.4×	936 1.5×	126	3.5k
Jørgen E. Nielsen Denmark	31	1.6k 0.7×	281 0.2×	1.6k 1.4×	149 0.2×	683 1.1×	150	3.8k

Countries citing papers authored by J. Robin Highley

Since Specialization

Citations

This map shows the geographic impact of J. Robin Highley's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by J. Robin Highley with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites J. Robin Highley more than expected).

Fields of papers citing papers by J. Robin Highley

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by J. Robin Highley. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by J. Robin Highley. The network helps show where J. Robin Highley may publish in the future.

Co-authorship network of co-authors of J. Robin Highley

This figure shows the co-authorship network connecting the top 25 collaborators of J. Robin Highley. A scholar is included among the top collaborators of J. Robin Highley based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with J. Robin Highley. J. Robin Highley is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Simpson, Julie E., Charlotte Dawson, Delphine Boche, et al.. (2025). Human amyotrophic lateral sclerosis/motor neuron disease: The disease‐associated microglial pathway is upregulated while APOE genotype governs risk and survival. Brain Pathology. 35(6). e70019–e70019. 1 indexed citations

Bauer, Claudia S., Francesca Sironi, Matthew R. Livesey, et al.. (2022). An interaction between synapsin and C9orf72 regulates excitatory synapses and is impaired in ALS/FTD. Acta Neuropathologica. 144(3). 437–464. 18 indexed citations

Shaw, Pamela J., et al.. (2021). Proteinopathies as Hallmarks of Impaired Gene Expression, Proteostasis and Mitochondrial Function in Amyotrophic Lateral Sclerosis. Frontiers in Neuroscience. 15. 783624–783624. 15 indexed citations

Wilson, L, Santiago Coelho, Alejandro F. Frangi, et al.. (2020). Tissue microarray (TMA) use in post mortem neuropathology. Journal of Neuroscience Methods. 347. 108963–108963. 5 indexed citations

Boche, Delphine, et al.. (2020). Review: Microglia in motor neuron disease. Neuropathology and Applied Neurobiology. 47(2). 179–197. 20 indexed citations

Wharton, Stephen B., Nick Verber, Bart Wagner, et al.. (2019). Combined fused in sarcoma‐positive (FUS+) basophilic inclusion body disease and atypical tauopathy presenting with an amyotrophic lateral sclerosis/motor neurone disease (ALS/MND)‐plus phenotype. Neuropathology and Applied Neurobiology. 45(6). 586–596. 6 indexed citations

Pozo, José M., Santiago Coelho, Julie E. Simpson, et al.. (2019). Quantitative histomorphometry of capillary microstructure in deep white matter. NeuroImage Clinical. 23. 101839–101839. 7 indexed citations

An, Haiyan, Antonietta Notaro, J. Robin Highley, et al.. (2019). ALS-linked FUS mutations confer loss and gain of function in the nucleus by promoting excessive formation of dysfunctional paraspeckles. Acta Neuropathologica Communications. 7(1). 7–7. 101 indexed citations

Simpson, Julie E., Lisa Watson, Heather Mortiboys, et al.. (2018). TIGAR inclusion pathology is specific for Lewy body diseases. Brain Research. 1706. 218–223. 10 indexed citations

10.

Boyd, Penelope J, Hannah K. Shorrock, Ewout J. N. Groen, et al.. (2017). Bioenergetic status modulates motor neuron vulnerability and pathogenesis in a zebrafish model of spinal muscular atrophy. PLoS Genetics. 13(4). e1006744–e1006744. 72 indexed citations

11.

Cooper‐Knock, Johnathan, Joanna J. Bury, Paul R. Heath, et al.. (2015). C9ORF72 GGGGCC Expanded Repeats Produce Splicing Dysregulation which Correlates with Disease Severity in Amyotrophic Lateral Sclerosis. PLoS ONE. 10(5). e0127376–e0127376. 77 indexed citations

12.

Cooper‐Knock, Johnathan, J. Robin Highley, Judith Hartley, et al.. (2013). Neurodegeneration caused by intronic expansions of C9ORF72 is a clinically heterogeneous but pathologically distinct disease. The Lancet. 381. S32–S32. 1 indexed citations

13.

Mackenzie, Ian R., Eileen H. Bigio, Paul G. Ince, et al.. (2007). Pathological TDP‐43 distinguishes sporadic amyotrophic lateral sclerosis from amyotrophic lateral sclerosis with SOD1 mutations. Annals of Neurology. 61(5). 427–434. 761 indexed citations breakdown →

14.

Highley, J. Robin, Mary A. Walker, Timothy J. Crow, Margaret M. Esiri, & Paul J. Harrison. (2003). Low Medial and Lateral Right Pulvinar Volumes in Schizophrenia: A Postmortem Study. American Journal of Psychiatry. 160(6). 1177–1179. 42 indexed citations

15.

Walker, Mary A., J. Robin Highley, Margaret M. Esiri, et al.. (2002). Estimated Neuronal Populations and Volumes of the Hippocampus and Its Subfields in Schizophrenia. American Journal of Psychiatry. 159(5). 821–828. 81 indexed citations

16.

Highley, J. Robin. (2002). Asymmetry of the Uncinate Fasciculus: A Post-mortem Study of Normal Subjects and Patients with Schizophrenia. Cerebral Cortex. 12(11). 1218–1224. 145 indexed citations

17.

Highley, J. Robin, Margaret M. Esiri, Brendan McDonald, et al.. (1999). The size and fibre composition of the corpus callosum with respect to gender and schizophrenia: a post-mortem study. Brain. 122(1). 99–110. 183 indexed citations

18.

Highley, J. Robin, et al.. (1999). The size and fiber composition of the anterior commissure with respect to gender and schizophrenia. Biological Psychiatry. 45(9). 1120–1127. 77 indexed citations

19.

Highley, J. Robin, et al.. (1999). Schizophrenia and temporal lobe asymmetry. The British Journal of Psychiatry. 175(2). 127–134. 93 indexed citations

20.

Chance, Steven A., J. Robin Highley, Margaret M. Esiri, & Timothy J. Crow. (1999). Fiber Content of the Fornix in Schizophrenia: Lack of Evidence for a Primary Limbic Encephalopathy. American Journal of Psychiatry. 156(11). 1720–1724. 48 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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