Thomas H. Gillingwater

15.2k total citations · 2 hit papers
183 papers, 9.6k citations indexed

About

Thomas H. Gillingwater is a scholar working on Molecular Biology, Genetics and Cellular and Molecular Neuroscience. According to data from OpenAlex, Thomas H. Gillingwater has authored 183 papers receiving a total of 9.6k indexed citations (citations by other indexed papers that have themselves been cited), including 104 papers in Molecular Biology, 80 papers in Genetics and 55 papers in Cellular and Molecular Neuroscience. Recurrent topics in Thomas H. Gillingwater's work include Neurogenetic and Muscular Disorders Research (80 papers), RNA modifications and cancer (46 papers) and Neuroscience and Neuropharmacology Research (24 papers). Thomas H. Gillingwater is often cited by papers focused on Neurogenetic and Muscular Disorders Research (80 papers), RNA modifications and cancer (46 papers) and Neuroscience and Neuropharmacology Research (24 papers). Thomas H. Gillingwater collaborates with scholars based in United Kingdom, United States and Germany. Thomas H. Gillingwater's co-authors include Thomas M. Wishart, Kevin Talbot, Richard R. Ribchester, Ewout J. N. Groen, Lyndsay M. Murray, Simon H. Parson, Derek Thomson, Gillian Hamilton, Paul Skehel and Helena Chaytow and has published in prestigious journals such as The Lancet, Nucleic Acids Research and Journal of Clinical Investigation.

In The Last Decade

Thomas H. Gillingwater

178 papers receiving 9.5k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

A Mutation in the Vesicle-Trafficking Protein VAPB Causes Late-Onset Spinal Muscular Atrophy and Amyotrophic Lateral Sclerosis

2004 757 citations Thomas H. Gillingwater et al. profile →
Wallerian degeneration of injured axons and synapses is delayed by a Ube4b/Nmnat chimeric gene

2001 501 citations Derek Thomson, Thomas H. Gillingwater et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Thomas H. Gillingwater United Kingdom	55	5.6k	3.5k	2.3k	1.8k	1.2k	183	9.6k
Christopher E. Henderson France	51	7.1k 1.3×	2.1k 0.6×	5.7k 2.5×	2.3k 1.3×	1.2k 0.9×	129	13.5k
Brian K. Kaspar United States	54	7.3k 1.3×	3.2k 0.9×	2.6k 1.1×	2.9k 1.6×	1.4k 1.2×	122	12.4k
Harley I. Kornblum United States	59	6.7k 1.2×	2.1k 0.6×	2.5k 1.1×	609 0.3×	655 0.5×	158	11.7k
Siddharthan Chandran United Kingdom	47	4.4k 0.8×	2.3k 0.6×	1.6k 0.7×	3.4k 1.9×	1.1k 0.9×	204	9.0k
John Vissing Denmark	52	5.8k 1.0×	1.1k 0.3×	2.3k 1.0×	1.8k 1.0×	1.9k 1.6×	422	10.4k
Andreas Hermann Germany	42	2.6k 0.5×	1.7k 0.5×	1.3k 0.6×	1.7k 0.9×	845 0.7×	241	5.8k
Grigori Enikolopov United States	56	6.4k 1.1×	2.7k 0.8×	3.0k 1.3×	400 0.2×	1.9k 1.5×	148	16.1k
Michael Sendtner Germany	75	10.0k 1.8×	4.0k 1.1×	8.2k 3.6×	2.6k 1.4×	1.3k 1.0×	225	20.1k
Ludwig Aigner Austria	48	4.5k 0.8×	1.5k 0.4×	3.9k 1.7×	838 0.5×	1.4k 1.1×	196	12.3k
R. Jeroen Pasterkamp Netherlands	55	5.1k 0.9×	1.3k 0.4×	4.9k 2.1×	2.2k 1.2×	840 0.7×	166	11.1k

Countries citing papers authored by Thomas H. Gillingwater

Since Specialization

Citations

This map shows the geographic impact of Thomas H. Gillingwater's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas H. Gillingwater with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas H. Gillingwater more than expected).

Fields of papers citing papers by Thomas H. Gillingwater

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas H. Gillingwater. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas H. Gillingwater. The network helps show where Thomas H. Gillingwater may publish in the future.

Co-authorship network of co-authors of Thomas H. Gillingwater

This figure shows the co-authorship network connecting the top 25 collaborators of Thomas H. Gillingwater. A scholar is included among the top collaborators of Thomas H. Gillingwater based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Thomas H. Gillingwater. Thomas H. Gillingwater is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

Faller, Kiterie M. E., et al.. (2025). Phosphoglycerate kinase 1 as a therapeutic target in neurological disease. Trends in Molecular Medicine. 31(12). 1077–1088. 1 indexed citations

Alsubaie, Najah, et al.. (2024). Distribution of neurovascular structures within the prostate gland and their relationship to complications after radical prostatectomy. The Prostate. 84(5). 491–501. 2 indexed citations

Chaytow, Helena, Yu-Ting Huang, Gillian L. Currie, et al.. (2024). Timing of SMN replacement therapies in mouse models of spinal muscular atrophy: a systematic review and meta-analysis. Brain Communications. 6(4). fcae267–fcae267. 4 indexed citations

Edwards, Ruairidh, Rachel Kline, Laura C. Graham, et al.. (2022). The mitochondrial protein Sideroflexin 3 (SFXN3) influences neurodegeneration pathways in vivo. FEBS Journal. 289(13). 3894–3914. 10 indexed citations

Kline, Rachel, Dominic Kurian, Samantha L. Eaton, et al.. (2022). An Optimized Comparative Proteomic Approach as a Tool in Neurodegenerative Disease Research. Cells. 11(17). 2653–2653. 2 indexed citations

Schmid, Michael, Luciano Brocchieri, Silvia Tornaletti, et al.. (2021). Phospho-RNA sequencing with circAID-p-seq. Nucleic Acids Research. 50(4). e23–e23. 3 indexed citations

Engler, Jan Broder, Karl Kuchler, Ross A. Jones, et al.. (2020). Motor neuron translatome reveals deregulation of SYNGR4 and PLEKHB1 in mutant TDP-43 amyotrophic lateral sclerosis models. Human Molecular Genetics. 29(16). 2647–2661. 15 indexed citations

Lauria, Fabio, Paola Bernabò, Toma Tebaldi, et al.. (2020). SMN-primed ribosomes modulate the translation of transcripts related to spinal muscular atrophy. Nature Cell Biology. 22(10). 1239–1251. 62 indexed citations

Shirran, Sally L., et al.. (2018). Neurochondrin interacts with the SMN protein suggesting a novel mechanism for spinal muscular atrophy pathology. Journal of Cell Science. 131(8). 12 indexed citations

10.

Gillingwater, Thomas H., Colin Chandler, Michael Ross, et al.. (2018). The Anatomical Society's core anatomy syllabus for undergraduate nursing. Journal of Anatomy. 232(5). 721–728. 37 indexed citations

11.

Lauria, Fabio, Toma Tebaldi, Paola Bernabò, et al.. (2018). riboWaltz: Optimization of ribosome P-site positioning in ribosome profiling data. PLoS Computational Biology. 14(8). e1006169–e1006169. 123 indexed citations

12.

Bernabò, Paola, Toma Tebaldi, Ewout J. N. Groen, et al.. (2017). In Vivo Translatome Profiling in Spinal Muscular Atrophy Reveals a Role for SMN Protein in Ribosome Biology. Cell Reports. 21(4). 953–965. 85 indexed citations

13.

Boyd, Penelope J, Hannah K. Shorrock, Ewout J. N. Groen, et al.. (2017). Bioenergetic status modulates motor neuron vulnerability and pathogenesis in a zebrafish model of spinal muscular atrophy. PLoS Genetics. 13(4). e1006744–e1006744. 72 indexed citations

14.

Henstridge, Christopher M., Emily Carroll, Makis Tzioras, et al.. (2017). Synapse loss in the prefrontal cortex is associated with cognitive decline in amyotrophic lateral sclerosis. Acta Neuropathologica. 135(2). 213–226. 85 indexed citations

15.

Baxter, Paul, Karen Bell, Philip Hasel, et al.. (2015). Synaptic NMDA receptor activity is coupled to the transcriptional control of the glutathione system. Nature Communications. 6(1). 6761–6761. 118 indexed citations

16.

Bastin, Mark E., et al.. (2013). Executive deficits, not processing speed relates to abnormalities in distinct prefrontal tracts in amyotrophic lateral sclerosis. Brain. 136(11). 3290–3304. 67 indexed citations

17.

Wright, Ann K., Thomas M. Wishart, C.A. Ingham, & Thomas H. Gillingwater. (2010). Synaptic Protection in the Brain of WldS Mice Occurs Independently of Age but Is Sensitive to Gene-Dose. PLoS ONE. 5(11). e15108–e15108. 13 indexed citations

18.

Court, Felipe A., Thomas H. Gillingwater, Diane L. Sherman, et al.. (2008). Identity, developmental restriction and reactivity of extralaminar cells capping mammalian neuromuscular junctions. Journal of Cell Science. 121(23). 3901–3911. 59 indexed citations

19.

Wishart, Thomas M., Helen N. Pemberton, Sally James, Chris J. McCabe, & Thomas H. Gillingwater. (2008). Modified cell cycle status in a mouse model of altered neuronal vulnerability (slow Wallerian degeneration; Wld s ). Genome biology. 9(6). R101–R101. 22 indexed citations

20.

Newbery, Helen J., Thomas H. Gillingwater, Permphan Dharmasaroja, et al.. (2005). Progressive Loss of Motor Neuron Function in Wasted Mice: Effects of a Spontaneous Null Mutation in the Gene for the eEF1A2 Translation Factor. Journal of Neuropathology & Experimental Neurology. 64(4). 295–303. 42 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact