Christopher P Webster

915 total citations · 1 hit paper
12 papers, 579 citations indexed

About

Christopher P Webster is a scholar working on Neurology, Cellular and Molecular Neuroscience and Genetics. According to data from OpenAlex, Christopher P Webster has authored 12 papers receiving a total of 579 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Neurology, 5 papers in Cellular and Molecular Neuroscience and 4 papers in Genetics. Recurrent topics in Christopher P Webster's work include Amyotrophic Lateral Sclerosis Research (7 papers), Autophagy in Disease and Therapy (4 papers) and Neurogenetic and Muscular Disorders Research (4 papers). Christopher P Webster is often cited by papers focused on Amyotrophic Lateral Sclerosis Research (7 papers), Autophagy in Disease and Therapy (4 papers) and Neurogenetic and Muscular Disorders Research (4 papers). Christopher P Webster collaborates with scholars based in United Kingdom, United States and Belgium. Christopher P Webster's co-authors include Kurt J. De Vos, Emma F. Smith, Pamela J. Shaw, Claudia S. Bauer, Andrew J. Grierson, Adrian Higginbottom, Guillaume M. Hautbergue, Laura Ferraiuolo, Kathrin Meyer and Matthew J. Walsh and has published in prestigious journals such as The EMBO Journal, Human Molecular Genetics and Acta Neuropathologica.

In The Last Decade

Christopher P Webster

11 papers receiving 571 citations

Hit Papers

The C9orf72 protein interacts with Rab1a and the ULK 1 co... 2016 2026 2019 2022 2016 100 200 300
What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

  1. The C9orf72 protein interacts with Rab1a and the ULK 1 complex to regulate initiation of autophagy
    2016 301 citations Christopher P Webster, Emma F. Smith et al. The EMBO Journal profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Christopher P Webster United Kingdom 10 370 221 163 162 108 12 579
Angéline Gaucherot France 4 314 0.8× 215 1.0× 110 0.7× 136 0.8× 75 0.7× 5 477
Mark A. Halloran Australia 6 464 1.3× 250 1.1× 116 0.7× 207 1.3× 125 1.2× 8 673
Valeria Gerbino Italy 9 363 1.0× 272 1.2× 123 0.8× 231 1.4× 57 0.5× 10 562
Nandini Ramesh United States 13 332 0.9× 352 1.6× 81 0.5× 176 1.1× 49 0.5× 14 566
Nathaniel Safren United States 12 312 0.8× 265 1.2× 74 0.5× 115 0.7× 76 0.7× 17 493
Katrina J. Llewellyn United States 10 173 0.5× 161 0.7× 113 0.7× 70 0.4× 59 0.5× 16 362
Laura Cox United Kingdom 6 237 0.6× 108 0.5× 100 0.6× 150 0.9× 62 0.6× 7 432
J. Gavin Daigle United States 7 291 0.8× 451 2.0× 60 0.4× 186 1.1× 62 0.6× 9 670
Kristin A. Politi United States 6 188 0.5× 326 1.5× 45 0.3× 102 0.6× 108 1.0× 6 557
Yevgeniya Abramzon United States 6 327 0.9× 216 1.0× 38 0.2× 161 1.0× 68 0.6× 7 506

Countries citing papers authored by Christopher P Webster

Since Specialization
Citations

This map shows the geographic impact of Christopher P Webster's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Christopher P Webster with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Christopher P Webster more than expected).

Fields of papers citing papers by Christopher P Webster

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Christopher P Webster. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Christopher P Webster. The network helps show where Christopher P Webster may publish in the future.

Co-authorship network of co-authors of Christopher P Webster

This figure shows the co-authorship network connecting the top 25 collaborators of Christopher P Webster. A scholar is included among the top collaborators of Christopher P Webster based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Christopher P Webster. Christopher P Webster is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

12 of 12 papers shown
1.
Webster, Christopher P, Eli Harriss, Fraser McBlane, et al.. (2024). A systematic review of immunosuppressive protocols used in AAV gene therapy for monogenic disorders. Molecular Therapy. 32(10). 3220–3259. 24 indexed citations
2.
Webster, Christopher P, Bradley Hall, Ya-Hui Lin, et al.. (2024). RuvBL1/2 reduce toxic dipeptide repeat protein burden in multiple models of C9orf72-ALS/FTD. Life Science Alliance. 8(2). e202402757–e202402757.
3.
Bauer, Claudia S., Francesca Sironi, Matthew R. Livesey, et al.. (2022). An interaction between synapsin and C9orf72 regulates excitatory synapses and is impaired in ALS/FTD. Acta Neuropathologica. 144(3). 437–464. 18 indexed citations
4.
Marchi, Paolo, et al.. (2022). Ap4b1-knockout mouse model of hereditary spastic paraplegia type 47 displays motor dysfunction, aberrant brain morphology and ATG9A mislocalization. Brain Communications. 5(1). fcac335–fcac335. 9 indexed citations
5.
Bauer, Claudia S., Christopher P Webster, Allan C. Shaw, et al.. (2022). Loss of TMEM106B exacerbates C9ALS/FTD DPR pathology by disrupting autophagosome maturation. Frontiers in Cellular Neuroscience. 16. 1061559–1061559. 9 indexed citations
6.
Karyka, Evangelia, Christopher P Webster, Paolo Marchi, et al.. (2022). SMN-deficient cells exhibit increased ribosomal DNA damage. Life Science Alliance. 5(8). e202101145–e202101145. 9 indexed citations
7.
Marrone, Lara, Paolo Marchi, Christopher P Webster, et al.. (2022). SPG15 protein deficits are at the crossroads between lysosomal abnormalities, altered lipid metabolism and synaptic dysfunction. Human Molecular Genetics. 31(16). 2693–2710. 10 indexed citations
8.
Allen, Scott P., Lydia M. Castelli, Laura Francis, et al.. (2018). Inosine reverses motor neuron toxicity observed in amyotrophic lateral sclerosis patient astrocytes with an adenosine deaminase deficiency. Biochimica et Biophysica Acta (BBA) - Bioenergetics. 1859. e23–e23. 1 indexed citations
9.
Webster, Christopher P, Emma F. Smith, Pamela J. Shaw, & Kurt J. De Vos. (2017). Protein Homeostasis in Amyotrophic Lateral Sclerosis: Therapeutic Opportunities?. Frontiers in Molecular Neuroscience. 10. 123–123. 67 indexed citations
10.
Bauer, Claudia S., et al.. (2017). Amyotrophic lateral sclerosis-associated mutant SOD1 inhibits anterograde axonal transport of mitochondria by reducing Miro1 levels. Human Molecular Genetics. 26(23). 4668–4679. 82 indexed citations
11.
Webster, Christopher P, Emma F. Smith, Claudia S. Bauer, et al.. (2016). The C9orf72 protein interacts with Rab1a and the ULK 1 complex to regulate initiation of autophagy. The EMBO Journal. 35(15). 1656–1676. 301 indexed citations breakdown →
12.
Webster, Christopher P, Emma F. Smith, Andrew J. Grierson, & Kurt J. De Vos. (2016). C9orf72 plays a central role in Rab GTPase-dependent regulation of autophagy. Small GTPases. 9(5). 399–408. 49 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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