Hideaki Kamata

8.2k total citations · 4 hit papers
71 papers, 6.8k citations indexed

About

Hideaki Kamata is a scholar working on Molecular Biology, Epidemiology and Immunology. According to data from OpenAlex, Hideaki Kamata has authored 71 papers receiving a total of 6.8k indexed citations (citations by other indexed papers that have themselves been cited), including 51 papers in Molecular Biology, 12 papers in Epidemiology and 12 papers in Immunology. Recurrent topics in Hideaki Kamata's work include NF-κB Signaling Pathways (9 papers), Genomics, phytochemicals, and oxidative stress (7 papers) and Protein Kinase Regulation and GTPase Signaling (7 papers). Hideaki Kamata is often cited by papers focused on NF-κB Signaling Pathways (9 papers), Genomics, phytochemicals, and oxidative stress (7 papers) and Protein Kinase Regulation and GTPase Signaling (7 papers). Hideaki Kamata collaborates with scholars based in Japan, United States and Spain. Hideaki Kamata's co-authors include Michael Karin, Shin Maeda, Hajime Hirata, Lufen Chang, Shinichi Honda, Jun‐Li Luo, Hyam L. Leffert, Hitoshi Yagisawa, Tomoichiro Asano and Shinichi Oka and has published in prestigious journals such as Cell, Proceedings of the National Academy of Sciences and Nucleic Acids Research.

In The Last Decade

Hideaki Kamata

70 papers receiving 6.7k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Reactive Oxygen Species Promote TNFα-Induced Death and Sustained JNK Activation by Inhibiting MAP Kinase Phosphatases

2005 1.5k citations Hideaki Kamata, Shinichi Honda et al. Cell profile →
Redox Regulation of Cellular Signalling

1999 950 citations Hideaki Kamata et al. Cellular Signalling profile →
IKKβ Couples Hepatocyte Death to Cytokine-Driven Compensatory Proliferation that Promotes Chemical Hepatocarcinogenesis

2005 948 citations Shin Maeda, Hideaki Kamata et al. Cell profile →
The E3 Ubiquitin Ligase Itch Couples JNK Activation to TNFα-induced Cell Death by Inducing c-FLIPL Turnover

2006 582 citations Lufen Chang, Hideaki Kamata et al. Cell profile →

Peers

Countries citing papers authored by Hideaki Kamata

Since Specialization

Citations

This map shows the geographic impact of Hideaki Kamata's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hideaki Kamata with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hideaki Kamata more than expected).

Fields of papers citing papers by Hideaki Kamata

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hideaki Kamata. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hideaki Kamata. The network helps show where Hideaki Kamata may publish in the future.

Co-authorship network of co-authors of Hideaki Kamata

This figure shows the co-authorship network connecting the top 25 collaborators of Hideaki Kamata. A scholar is included among the top collaborators of Hideaki Kamata based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hideaki Kamata. Hideaki Kamata is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Work	Indexed citations
1	Structural instability of IκB kinase β promotes autophagic degradation through enhancement of Keap1 binding 2018 ·PLoS ONE ·(unknown), Yoshihiro Tsuchiya, Yuki Nakao, Takafumi Suzuki, Hozumi Motohashi, Masayuki Yamamoto, Hideaki Kamata	5
2	Treatment with the SGLT2 inhibitor luseogliflozin improves nonalcoholic steatohepatitis in a rodent model with diabetes mellitus 2015 ·Diabetology & Metabolic Syndrome ·Shirong Qiang, Yusuke Nakatsu, (unknown), Midori Fujishiro, Hideyuki Sakoda, Akifumi Kushiyama, Keiichi Mori, Yasuka Matsunaga, Takeshi Yamamotoya, Hideaki Kamata, Tomoichiro Asano	97
3	Par14 Protein Associates with Insulin Receptor Substrate 1 (IRS-1), Thereby Enhancing Insulin-induced IRS-1 Phosphorylation and Metabolic Actions 2013 ·Journal of Biological Chemistry ·Jun Zhang, Yusuke Nakatsu, Takanori Shinjo, Ying Guo, Hideyuki Sakoda, Takeshi Yamamotoya, (unknown), Hirofumi Okubo, Akifumi Kushiyama, Midori Fujishiro, Toshiaki Fukushima, Yoshihiro Tsuchiya, Hideaki Kamata, Misaki Iwashita, Fusanori Nishimura, Hideki Katagiri, Shin‐Ichiro Takahashi, Hiroki Kurihara, Takafumi Uchida, Tomoichiro Asano	20
4	Valsartan restores inflammatory response by macrophages in adipose and hepatic tissues of LPS-infused mice 2012 ·Adipocyte ·Misaki Iwashita, Yusuke Nakatsu, Hideyuki Sakoda, Midori Fujishiro, Akifumi Kushiyama, Toshiaki Fukushima, (unknown), Takanori Shinjo, Hideaki Kamata, Fusanori Nishimura, Tomoichiro Asano	11
5	On–off system for PI3-kinase–Akt signaling through S -nitrosylation of phosphatase with sequence homology to tensin (PTEN) 2011 ·Proceedings of the National Academy of Sciences ·(unknown), Kumi Takasawa, Tadashi Nishiya, Hirotaka Tanaka, (unknown), (unknown), (unknown), (unknown), Kaoru Azumi, Hideaki Kamata, Tomohiro Nakamura, Hideaki Hara, Masabumi Minami, Stuart A. Lipton, Takashi Uehara	126
6	Macrophage foam cell formation is augmented in serum from patients with diabetic angiopathy 2009 ·Diabetes Research and Clinical Practice ·(unknown), Akifumi Kushiyama, Masayasu Yoneda, Yusuke Nakatsu, Ying Guo, Jun Zhang, (unknown), Machi Kanna, Hideyuki Sakoda, Hiraku Ono, Takako Kikuchi, Midori Fujishiro, Masashi Shiomi, Hideaki Kamata, Hiroki Kurihara, Masatoshi Kikuchi, Shoji Kawazu, Fusanori Nishimura, Tomoichiro Asano	12
7	AMP-activated Protein Kinase Activation Increases Phosphorylation of Glycogen Synthase Kinase 3β and Thereby Reduces cAMP-responsive Element Transcriptional Activity and Phosphoenolpyruvate Carboxykinase C Gene Expression in the Liver 2008 ·Journal of Biological Chemistry ·Nanao Horike, Hideyuki Sakoda, Akifumi Kushiyama, Hiraku Ono, Midori Fujishiro, Hideaki Kamata, Koichi Nishiyama, Yasunobu Uchijima, Yukiko Kurihara, Hiroki Kurihara, Tomoichiro Asano	171
8	The E3 Ubiquitin Ligase Itch Couples JNK Activation to TNFα-induced Cell Death by Inducing c-FLIPL Turnover breakdown → 2006 ·Cell ·Lufen Chang, Hideaki Kamata, Giovanni Solinas, (unknown), Shin Maeda, K. Venuprasad, Yun‐Cai Liu, Michael Karin	582
9	Reactive Oxygen Species Promote TNFα-Induced Death and Sustained JNK Activation by Inhibiting MAP Kinase Phosphatases breakdown → 2005 ·Cell ·Hideaki Kamata, Shinichi Honda, Shin Maeda, Lufen Chang, Hajime Hirata, Michael Karin	1526
10	The Anti-Death Machinery in IKK/NF-κB Signaling 2005 ·Journal of Clinical Immunology ·Jun‐Li Luo, Hideaki Kamata, Michael Karin	88
11	A PLCδ1-binding protein, p122RhoGAP, is localized in focal adhesions 2004 ·Biochemical Society Transactions ·Katsuhisa Kawai, Masaki Yamaga, (unknown), (unknown), Hideaki Kamata, Hajime Hirata, Yoshimi Homma, Hitoshi Yagisawa	37
12	Redox regulation of nerve growth factor-induced neuronal differentiation of PC12 cells through modulation of the nerve growth factor receptor, TrkA 2004 ·Archives of Biochemistry and Biophysics ·Hideaki Kamata, Shinichi Oka, Yukinao Shibukawa, (unknown), Hajime Hirata	40
13	A PLCδ₁‐binding protein, p122/RhoGAP, is localized in caveolin‐enriched membrane domains and regulates caveolin internalization 2004 ·Genes to Cells ·Masaki Yamaga, Masayuki Sekimata, Makoto Fujii, Katsuhisa Kawai, Hideaki Kamata, Hajime Hirata, Yoshimi Homma, Hitoshi Yagisawa	44
14	Carboxyl‐terminal basic amino acids in the X domain are essential for the nuclear import of phospholipase C δ₁ 2002 ·Genes to Cells ·Masashi Okada, Makoto Fujii, Masaki Yamaga, (unknown), Hiroyuki Sadano, Takashi Osumi, Hideaki Kamata, Hajime Hirata, Hitoshi Yagisawa	21
15	Hydrogen peroxide activates IκB kinases through phosphorylation of serine residues in the activation loops 2002 ·FEBS Letters ·Hideaki Kamata, (unknown), Shinichi Oka, Keiko Kamata, Hajime Hirata	144
16	Epidermal growth factor receptor is modulated by redox through multiple mechanisms 2000 ·European Journal of Biochemistry ·Hideaki Kamata, Yukinao Shibukawa, Shinichi Oka, Hajime Hirata	66
17	Overexpression of the Alanine Carrier Protein Gene from Thermophilic Bacterium PS3 in Escherichia coli 1999 ·The Journal of Biochemistry ·(unknown), Hideaki Kamata, Hitoshi Yagisawa, Hajime Hirata	8
18	Redox Regulation of Cellular Signalling breakdown → 1999 ·Cellular Signalling ·Hideaki Kamata, (unknown)	950
19	Inhibitory effect of NaN3 on the F0F1 ATPase of submitochondrial particles as related to nucleotide binding 1993 ·Biochimica et Biophysica Acta (BBA) - Bioenergetics ·Eiro Muneyuki, (unknown), Hideaki Kamata, Yasuo Kagawa, Masasuke Yoshida, Hajime Hirata	41
20	[A case of diffuse pulmonary hamartoangiomyomatosis with recurrent spontaneous pneumothorax (author's transl)]. 1978 ·Munich Personal RePEc Archive (Ludwig Maximilian University of Munich) ·(unknown), (unknown), Shintaro Arai, Hideaki Kamata, Hideyuki Okano, (unknown), (unknown), Mitsuru Hara, H. Kumada, Atsushi Yamanaka	1

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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