Hiraku Ono

4.8k total citations
79 papers, 3.8k citations indexed

About

Hiraku Ono is a scholar working on Molecular Biology, Surgery and Endocrine and Autonomic Systems. According to data from OpenAlex, Hiraku Ono has authored 79 papers receiving a total of 3.8k indexed citations (citations by other indexed papers that have themselves been cited), including 53 papers in Molecular Biology, 29 papers in Surgery and 15 papers in Endocrine and Autonomic Systems. Recurrent topics in Hiraku Ono's work include Pancreatic function and diabetes (25 papers), Metabolism, Diabetes, and Cancer (22 papers) and Regulation of Appetite and Obesity (14 papers). Hiraku Ono is often cited by papers focused on Pancreatic function and diabetes (25 papers), Metabolism, Diabetes, and Cancer (22 papers) and Regulation of Appetite and Obesity (14 papers). Hiraku Ono collaborates with scholars based in Japan, United States and Denmark. Hiraku Ono's co-authors include Tomoichiro Asano, Hideyuki Sakoda, Motonobu Anai, Yasushi Fukushima, Takehide Ogihara, Yukiko Onishi, Masatoshi Kikuchi, Hideki Katagiri, Midori Fujishiro and Yoshitomo Oka and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Clinical Investigation and Nature Communications.

In The Last Decade

Hiraku Ono

78 papers receiving 3.8k citations

Peers

Hiraku Ono
Hiraku Ono
Citations per year, relative to Hiraku Ono Hiraku Ono (= 1×) peers Frédéric Preitner

Countries citing papers authored by Hiraku Ono

Since Specialization
Citations

This map shows the geographic impact of Hiraku Ono's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hiraku Ono with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hiraku Ono more than expected).

Fields of papers citing papers by Hiraku Ono

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hiraku Ono. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hiraku Ono. The network helps show where Hiraku Ono may publish in the future.

Co-authorship network of co-authors of Hiraku Ono

This figure shows the co-authorship network connecting the top 25 collaborators of Hiraku Ono. A scholar is included among the top collaborators of Hiraku Ono based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hiraku Ono. Hiraku Ono is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Sakai, Shinsuke, Youichi Tanaka, Yusuke Tsukamoto, et al.. (2023). d-Alanine Affects the Circadian Clock to Regulate Glucose Metabolism in the Kidney. Kidney360. 5(2). 237–251. 7 indexed citations
2.
Ono, Hiraku, et al.. (2023). Insulin Sensitivity Initially Worsens but Later Improves With Aging in Male C57BL/6N Mice. The Journals of Gerontology Series A. 78(10). 1785–1792. 1 indexed citations
3.
Kanna, Machi, Yusuke Nakatsu, Takeshi Yamamotoya, et al.. (2023). Hepatic Pin1 Expression, Particularly in Nuclei, Is Increased in NASH Patients in Accordance with Evidence of the Role of Pin1 in Lipid Accumulation Shown in Hepatoma Cell Lines. International Journal of Molecular Sciences. 24(10). 8847–8847. 2 indexed citations
4.
Nakatsu, Yusuke, Takeshi Yamamotoya, Yasuka Matsunaga, et al.. (2020). Prolyl isomerase Pin1 interacts with adipose triglyceride lipase and negatively controls both its expression and lipolysis. Metabolism. 115. 154459–154459. 12 indexed citations
5.
Nakatsu, Yusuke, Takeshi Yamamotoya, Koji Ueda, et al.. (2019). Prolyl isomerase Pin1 in metabolic reprogramming of cancer cells. Cancer Letters. 470. 106–114. 30 indexed citations
6.
Nakatsu, Yusuke, Keiichi Mori, Yasuka Matsunaga, et al.. (2017). The prolyl isomerase Pin1 increases β-cell proliferation and enhances insulin secretion. Journal of Biological Chemistry. 292(28). 11886–11895. 21 indexed citations
7.
Yamamotoya, Takeshi, Yusuke Nakatsu, Akifumi Kushiyama, et al.. (2017). Trk-fused gene (TFG) regulates pancreatic β cell mass and insulin secretory activity. Scientific Reports. 7(1). 13026–13026. 10 indexed citations
8.
Nakano, Takanari, Ikuo Inoue, Yasuhiro Takenaka, et al.. (2016). Ezetimibe Promotes Brush Border Membrane-to-Lumen Cholesterol Efflux in the Small Intestine. PLoS ONE. 11(3). e0152207–e0152207. 34 indexed citations
9.
Uno, Kenji, Tetsuya Yamada, Yasushi Ishigaki, et al.. (2015). A hepatic amino acid/mTOR/S6K-dependent signalling pathway modulates systemic lipid metabolism via neuronal signals. Nature Communications. 6(1). 7940–7940. 50 indexed citations
10.
Lustig, Yaniv, Jorge L. Ruas, Jennifer L. Estall, et al.. (2011). Separation of the gluconeogenic and mitochondrial functions of PGC-1α through S6 kinase. Genes & Development. 25(12). 1232–1244. 87 indexed citations
11.
Nakatsu, Yusuke, Hideyuki Sakoda, Akifumi Kushiyama, et al.. (2010). Pin1 Associates with and Induces Translocation of CRTC2 to the Cytosol, Thereby Suppressing cAMP-responsive Element Transcriptional Activity*. Journal of Biological Chemistry. 285(43). 33018–33027. 27 indexed citations
12.
Kushiyama, Akifumi, Masayasu Yoneda, Yusuke Nakatsu, et al.. (2009). Macrophage foam cell formation is augmented in serum from patients with diabetic angiopathy. Diabetes Research and Clinical Practice. 87(1). 57–63. 12 indexed citations
14.
Blouet, Clémence, Hiraku Ono, & Gary J. Schwartz. (2008). Mediobasal Hypothalamic p70 S6 Kinase 1 Modulates the Control of Energy Homeostasis. Cell Metabolism. 8(6). 459–467. 148 indexed citations
15.
Shojima, Nobuhiro, Takehide Ogihara, Kouichi Inukai, et al.. (2005). Serum concentrations of resistin-like molecules β and γ are elevated in high-fat-fed and obese db/db mice, with increased production in the intestinal tract and bone marrow. Diabetologia. 48(5). 984–992. 37 indexed citations
16.
Onishi, Yukiko, Takehide Ogihara, Hideyuki Sakoda, et al.. (2003). Ethanol feeding induces insulin resistance with enhanced PI 3-kinase activation. Biochemical and Biophysical Research Communications. 303(3). 788–794. 74 indexed citations
17.
Inukai, Kouichi, Makoto Funaki, Motonobu Anai, et al.. (2001). Five isoforms of the phosphatidylinositol 3‐kinase regulatory subunit exhibit different associations with receptor tyrosine kinases and their tyrosine phosphorylations. FEBS Letters. 490(1-2). 32–38. 41 indexed citations
18.
Fukushima, Yasushi, T. Saitoh, Motonobu Anai, et al.. (2001). G649, an allelic variant of the human H2 receptor with low basal activity, is resistant to upregulation upon antagonist exposure. The Pharmacogenomics Journal. 1(1). 78–83. 7 indexed citations
19.
Fukushima, Yasushi, Toshihito Saitoh, Motonobu Anai, et al.. (2001). Palmitoylation of the canine histamine H2 receptor occurs at Cys305 and is important for cell surface targeting. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1539(3). 181–191. 31 indexed citations
20.
Anai, Motonobu, Hiraku Ono, Makoto Funaki, et al.. (1998). Different Subcellular Distribution and Regulation of Expression of Insulin Receptor Substrate (IRS)-3 from Those of IRS-1 and IRS-2. Journal of Biological Chemistry. 273(45). 29686–29692. 60 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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