Yusuke Nakatsu

2.4k total citations
64 papers, 1.9k citations indexed

About

Yusuke Nakatsu is a scholar working on Molecular Biology, Surgery and Epidemiology. According to data from OpenAlex, Yusuke Nakatsu has authored 64 papers receiving a total of 1.9k indexed citations (citations by other indexed papers that have themselves been cited), including 42 papers in Molecular Biology, 16 papers in Surgery and 15 papers in Epidemiology. Recurrent topics in Yusuke Nakatsu's work include Signaling Pathways in Disease (19 papers), Pancreatic function and diabetes (14 papers) and Metabolism, Diabetes, and Cancer (8 papers). Yusuke Nakatsu is often cited by papers focused on Signaling Pathways in Disease (19 papers), Pancreatic function and diabetes (14 papers) and Metabolism, Diabetes, and Cancer (8 papers). Yusuke Nakatsu collaborates with scholars based in Japan, United States and India. Yusuke Nakatsu's co-authors include Tomoichiro Asano, Akifumi Kushiyama, Hideyuki Sakoda, Midori Fujishiro, Takeshi Yamamotoya, Yasuka Matsunaga, Hideaki Kamata, Yaichiro Kotake, Hiraku Ono and Toshiaki Fukushima and has published in prestigious journals such as Journal of Biological Chemistry, Nature Communications and PLoS ONE.

In The Last Decade

Yusuke Nakatsu

62 papers receiving 1.8k citations

Peers

Yusuke Nakatsu

EPIDE

EDM

SURGE

NEPHR

Inhye Park South Korea

Min Zhao China

Stephen C. Benson United States

Shaikh Mizanoor Rahman United States

Masato Tawata Japan

Cheng‐Tien Wu Taiwan

Tariq Hamid United States

Kuo‐Chu Chang Taiwan

Sabbir Khan India

Shiow‐Shih Tang United States

Inhye Park South Korea

Yusuke Nakatsu

553 ×0.6

217 ×0.5

EPIDE

602 ×1.5

EDM

549 ×1.9

SURGE

44 ×0.2

NEPHR

Citations per year, relative to Yusuke Nakatsu Yusuke Nakatsu (= 1×) peers Inhye Park

Countries citing papers authored by Yusuke Nakatsu

Since Specialization

Citations

This map shows the geographic impact of Yusuke Nakatsu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Yusuke Nakatsu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Yusuke Nakatsu more than expected).

Fields of papers citing papers by Yusuke Nakatsu

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Yusuke Nakatsu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Yusuke Nakatsu. The network helps show where Yusuke Nakatsu may publish in the future.

Co-authorship network of co-authors of Yusuke Nakatsu

This figure shows the co-authorship network connecting the top 25 collaborators of Yusuke Nakatsu. A scholar is included among the top collaborators of Yusuke Nakatsu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Yusuke Nakatsu. Yusuke Nakatsu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Sano, Tomomi, et al.. (2025). miR-6402 targets Bmpr2 and negatively regulates mouse adipogenesis. Adipocyte. 14(1). 2474114–2474114.

Kanna, Machi, et al.. (2023). Prolyl isomerase Pin1 promotes extracellular matrix production in hepatic stellate cells through regulating formation of the Smad3-TAZ complex. Experimental Cell Research. 425(2). 113544–113544. 4 indexed citations

Kanna, Machi, Yusuke Nakatsu, Takeshi Yamamotoya, et al.. (2023). Hepatic Pin1 Expression, Particularly in Nuclei, Is Increased in NASH Patients in Accordance with Evidence of the Role of Pin1 in Lipid Accumulation Shown in Hepatoma Cell Lines. International Journal of Molecular Sciences. 24(10). 8847–8847. 2 indexed citations

Yamamotoya, Takeshi, Yusuke Nakatsu, Machi Kanna, et al.. (2021). Prolyl isomerase Pin1 plays an essential role in SARS-CoV-2 proliferation, indicating its possibility as a novel therapeutic target. Scientific Reports. 11(1). 18581–18581. 16 indexed citations

Nakatsu, Yusuke, Takeshi Yamamotoya, Yasuka Matsunaga, et al.. (2020). Prolyl isomerase Pin1 interacts with adipose triglyceride lipase and negatively controls both its expression and lipolysis. Metabolism. 115. 154459–154459. 12 indexed citations

Nakatsu, Yusuke, Takeshi Yamamotoya, Koji Ueda, et al.. (2019). Prolyl isomerase Pin1 in metabolic reprogramming of cancer cells. Cancer Letters. 470. 106–114. 30 indexed citations

Yamazaki, Hiroki, Akifumi Kushiyama, Hideyuki Sakoda, et al.. (2018). Protective Effect of Sex Hormone-Binding Globulin against Metabolic Syndrome:In VitroEvidence Showing Anti-Inflammatory and Lipolytic Effects on Adipocytes and Macrophages. Mediators of Inflammation. 2018. 1–12. 63 indexed citations

Nakatsu, Yusuke, Keiichi Mori, Yasuka Matsunaga, et al.. (2017). The prolyl isomerase Pin1 increases β-cell proliferation and enhances insulin secretion. Journal of Biological Chemistry. 292(28). 11886–11895. 21 indexed citations

Yamamotoya, Takeshi, Yusuke Nakatsu, Akifumi Kushiyama, et al.. (2017). Trk-fused gene (TFG) regulates pancreatic β cell mass and insulin secretory activity. Scientific Reports. 7(1). 13026–13026. 10 indexed citations

10.

Shinjo, Takanori, Yusuke Nakatsu, Misaki Iwashita, et al.. (2015). High-fat diet feeding significantly attenuates anagliptin-induced regeneration of islets of Langerhans in streptozotocin-induced diabetic mice. Diabetology & Metabolic Syndrome. 7(1). 50–50. 9 indexed citations

11.

Yoneda, Masayasu, Hirofumi Okubo, Haruya Ohno, et al.. (2015). Distinct Time Course of the Decrease in Hepatic AMP-Activated Protein Kinase and Akt Phosphorylation in Mice Fed a High Fat Diet. PLoS ONE. 10(8). e0135554–e0135554. 17 indexed citations

12.

Qiang, Shirong, Yusuke Nakatsu, Midori Fujishiro, et al.. (2015). Treatment with the SGLT2 inhibitor luseogliflozin improves nonalcoholic steatohepatitis in a rodent model with diabetes mellitus. Diabetology & Metabolic Syndrome. 7(1). 104–104. 97 indexed citations

13.

Zhang, Jun, Yusuke Nakatsu, Takanori Shinjo, et al.. (2013). Par14 Protein Associates with Insulin Receptor Substrate 1 (IRS-1), Thereby Enhancing Insulin-induced IRS-1 Phosphorylation and Metabolic Actions. Journal of Biological Chemistry. 288(28). 20692–20701. 20 indexed citations

14.

Iwashita, Misaki, Yusuke Nakatsu, Hideyuki Sakoda, et al.. (2012). Valsartan restores inflammatory response by macrophages in adipose and hepatic tissues of LPS-infused mice. Adipocyte. 2(1). 28–32. 11 indexed citations

15.

Nakatsu, Yusuke, et al.. (2009). Long-term exposure to endogenous levels of tributyltin decreases GluR2 expression and increases neuronal vulnerability to glutamate. Toxicology and Applied Pharmacology. 240(2). 292–298. 31 indexed citations

16.

Kushiyama, Akifumi, Masayasu Yoneda, Yusuke Nakatsu, et al.. (2009). Macrophage foam cell formation is augmented in serum from patients with diabetic angiopathy. Diabetes Research and Clinical Practice. 87(1). 57–63. 12 indexed citations

17.

Nakatsu, Yusuke, et al.. (2008). Activation of AMP-activated protein kinase by tributyltin induces neuronal cell death. Toxicology and Applied Pharmacology. 230(3). 358–363. 44 indexed citations

18.

Nakatsu, Yusuke, Yaichiro Kotake, & Satoshi Ohta. (2007). Concentration Dependence of the Mechanisms of Tributyltin-Induced Apoptosis. Toxicological Sciences. 97(2). 438–447. 64 indexed citations

19.

Nakatsu, Yusuke, Yaichiro Kotake, & Shigeru Ohta. (2006). Tributyltin-induced cell death is mediated by calpain in PC12 cells. NeuroToxicology. 27(4). 587–593. 22 indexed citations

20.

Nakatsu, Yusuke, et al.. (2005). Glutamate Excitotoxicity Is Involved in Cell Death Caused by Tributyltin in Cultured Rat Cortical Neurons. Toxicological Sciences. 89(1). 235–242. 54 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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