Edward B. Lee

26.7k total citations · 4 hit papers
208 papers, 10.6k citations indexed

About

Edward B. Lee is a scholar working on Physiology, Neurology and Psychiatry and Mental health. According to data from OpenAlex, Edward B. Lee has authored 208 papers receiving a total of 10.6k indexed citations (citations by other indexed papers that have themselves been cited), including 108 papers in Physiology, 99 papers in Neurology and 58 papers in Psychiatry and Mental health. Recurrent topics in Edward B. Lee's work include Alzheimer's disease research and treatments (98 papers), Parkinson's Disease Mechanisms and Treatments (67 papers) and Dementia and Cognitive Impairment Research (55 papers). Edward B. Lee is often cited by papers focused on Alzheimer's disease research and treatments (98 papers), Parkinson's Disease Mechanisms and Treatments (67 papers) and Dementia and Cognitive Impairment Research (55 papers). Edward B. Lee collaborates with scholars based in United States, United Kingdom and Germany. Edward B. Lee's co-authors include John Q. Trojanowski, Virginia M.‐Y. Lee, David J. Irwin, Vivianna M. Van Deerlin, Murray Grossman, Linda K. Kwong, Corey T. McMillan, John Robinson, Sharon X. Xie and EunRan Suh and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nucleic Acids Research and Journal of Biological Chemistry.

In The Last Decade

Edward B. Lee

199 papers receiving 10.5k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Stages of pTDP‐43 pathology in amyotrophic lateral sclerosis

2013 750 citations John Robinson, David J. Irwin et al. profile →
Gains or losses: molecular mechanisms of TDP43-mediated neurodegeneration

2011 515 citations Edward B. Lee, Virginia M.‐Y. Lee et al. profile →
SpaGCN: Integrating gene expression, spatial location and histology to identify spatial domains and spatially variable genes by graph convolutional network

2021 450 citations Jian Hu, Amelia Schroeder et al. profile →
Inferring super-resolution tissue architecture by integrating spatial transcriptomics with histology

2024 73 citations Daiwei Zhang, Amelia Schroeder et al. Nature Biotechnology profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Edward B. Lee United States	53	4.9k	4.2k	3.7k	1.9k	1.6k	208	10.6k
Thomas Arzberger Germany	46	4.3k 0.9×	3.3k 0.8×	3.6k 1.0×	1.7k 0.9×	1.3k 0.8×	116	9.7k
Tetsuaki Arai Japan	47	6.3k 1.3×	3.8k 0.9×	3.6k 1.0×	2.0k 1.1×	1.8k 1.2×	216	10.0k
Vivianna M. Van Deerlin United States	53	6.0k 1.2×	4.1k 1.0×	2.4k 0.6×	2.1k 1.1×	1.4k 0.9×	148	9.6k
Mark S. Forman United States	47	4.7k 1.0×	4.0k 0.9×	2.9k 0.8×	1.7k 0.9×	1.0k 0.7×	82	9.1k
Tammaryn Lashley United Kingdom	48	4.7k 1.0×	4.4k 1.1×	3.3k 0.9×	2.1k 1.1×	699 0.4×	158	9.1k
Ekaterina Rogaeva Canada	53	3.4k 0.7×	3.4k 0.8×	3.6k 1.0×	1.4k 0.7×	870 0.6×	188	8.3k
Tibor Hortobágyi Hungary	43	3.2k 0.7×	2.2k 0.5×	2.7k 0.7×	1.4k 0.7×	1.5k 1.0×	227	8.3k
David J. Irwin United States	50	4.9k 1.0×	4.1k 1.0×	2.7k 0.7×	1.6k 0.9×	833 0.5×	274	10.1k
Shigeo Murayama Japan	61	6.0k 1.2×	5.8k 1.4×	5.4k 1.5×	2.8k 1.5×	765 0.5×	409	14.2k
Leo McCluskey United States	44	8.0k 1.6×	2.8k 0.7×	3.4k 0.9×	1.9k 1.0×	3.3k 2.1×	90	11.0k

Countries citing papers authored by Edward B. Lee

Since Specialization

Citations

This map shows the geographic impact of Edward B. Lee's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Edward B. Lee with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Edward B. Lee more than expected).

Fields of papers citing papers by Edward B. Lee

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Edward B. Lee. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Edward B. Lee. The network helps show where Edward B. Lee may publish in the future.

Co-authorship network of co-authors of Edward B. Lee

This figure shows the co-authorship network connecting the top 25 collaborators of Edward B. Lee. A scholar is included among the top collaborators of Edward B. Lee based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Edward B. Lee. Edward B. Lee is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Zhang, Hanwen, Daniel Kargilis, Thomas F. Tropea, et al.. (2025). Calcium modulating ligand confers risk for Parkinson's disease and impacts lysosomes. Annals of Clinical and Translational Neurology. 12(5). 925–937. 1 indexed citations

Xu, Hong, Qi Qiu, Peng Hu, et al.. (2024). MSUT2 regulates tau spreading via adenosinergic signaling mediated ASAP1 pathway in neurons. Acta Neuropathologica. 147(1). 55–55. 3 indexed citations

Zhang, Daiwei, Amelia Schroeder, Hanying Yan, et al.. (2024). Inferring super-resolution tissue architecture by integrating spatial transcriptomics with histology. Nature Biotechnology. 42(9). 1372–1377. 73 indexed citations breakdown →

Creekmore, Benjamin C., et al.. (2024). Ultrastructure of human brain tissue vitrified from autopsy revealed by cryo-ET with cryo-plasma FIB milling. Nature Communications. 15(1). 2660–2660. 12 indexed citations

Cousins, Katheryn A Q, Jeffrey S. Phillips, Sandhitsu R. Das, et al.. (2024). Pathologic and cognitive correlates of plasma biomarkers in neurodegenerative disease. Alzheimer s & Dementia. 20(6). 3889–3905. 16 indexed citations

Phan, Jessica M., et al.. (2024). VCP activator reverses nuclear proteostasis defects and enhances TDP-43 aggregate clearance in multisystem proteinopathy models. Journal of Clinical Investigation. 134(14). 4 indexed citations

Das, Sandhitsu R., Christopher A. Olm, Katheryn A Q Cousins, et al.. (2024). Posterior hippocampal sparing in Lewy body disorders with Alzheimer’s copathology: An in vivo MRI study. NeuroImage Clinical. 45. 103714–103714. 1 indexed citations

Creekmore, Benjamin C., et al.. (2024). Bridging structural biology and clinical research through in-tissue cryo-electron tomography. The EMBO Journal. 43(21). 4810–4813. 1 indexed citations

Uemura, Maiko, Eun Ran Suh, John Robinson, et al.. (2022). Abundant copathologies of polyglucosan bodies, frontotemporal lobar degeneration with TDP‐43 inclusions and ageing‐related tau astrogliopathy in a family with a GBE1 mutation. Neuropathology and Applied Neurobiology. 49(1). e12865–e12865. 4 indexed citations

10.

Cousins, Katheryn A Q, Sanjana Shellikeri, Daniel T. Ohm, et al.. (2022). CSF Biomarkers of Alzheimer Disease in Patients With Concomitant α-Synuclein Pathology. Neurology. 99(20). e2303–e2312. 13 indexed citations

11.

Shellikeri, Sanjana, Sunghye Cho, Katheryn A Q Cousins, et al.. (2022). Natural speech markers of Alzheimer's disease co-pathology in Lewy body dementias. Parkinsonism & Related Disorders. 102. 94–100. 7 indexed citations

12.

Coughlin, David G., Annie Hiniker, Claire Peterson, et al.. (2022). Digital Histological Study of Neocortical Grey and White Matter Tau Burden Across Tauopathies. Journal of Neuropathology & Experimental Neurology. 81(12). 953–964. 7 indexed citations

13.

Burke, Sarah, Jeffrey S. Phillips, Christopher A. Olm, et al.. (2022). Phases of volume loss in patients with known frontotemporal lobar degeneration spectrum pathology. Neurobiology of Aging. 113. 95–107. 6 indexed citations

14.

Porta, Sílvia, Yan Xu, Bin Zhang, et al.. (2021). Distinct brain‐derived TDP‐43 strains from FTLD‐TDP subtypes induce diverse morphological TDP‐43 aggregates and spreading patterns in vitro and in vivo. Neuropathology and Applied Neurobiology. 47(7). 1033–1049. 33 indexed citations

15.

Lin, Yen‐Chen, Nandini Ramesh, Eric N. Anderson, et al.. (2021). Interactions between ALS-linked FUS and nucleoporins are associated with defects in the nucleocytoplasmic transport pathway. Nature Neuroscience. 24(8). 1077–1088. 63 indexed citations

16.

Cousins, Katheryn A Q, David J. Irwin, David A. Wolk, et al.. (2020). ATN status in amnestic and non-amnestic Alzheimer’s disease and frontotemporal lobar degeneration. Brain. 143(7). 2295–2311. 28 indexed citations

17.

Webber, Ann, et al.. (2018). Suppression rather than visual acuity loss limits threshold stereoacuity.. Investigative Ophthalmology & Visual Science. 59(9). 4115–4115. 2 indexed citations

18.

Cali, Christopher P., et al.. (2017). RNA metabolism in neurodegenerative disease. Disease Models & Mechanisms. 10(5). 509–518. 103 indexed citations

19.

Phillips, Jeffrey S., Fulvio Da Re, Sharon X. Xie, et al.. (2017). Neocortical origin and progression of gray matter atrophy in nonamnestic Alzheimer's disease. Neurobiology of Aging. 63. 75–87. 52 indexed citations

20.

Baer, George Μ., Felicia Cooper, EunRan Suh, et al.. (2017). Sporadic FTLD TDP type C is a distinct clinicopathological entity from FTLD types A/B (S7.007). Neurology. 88(16_supplement).

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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