Chris J. Vlahos
- Molecular Biology top 1%
- Immunology top 2%
- Cell Biology top 1%
- Physiology top 2%
- Oncology top 5%
- Co-authors
- William F. MatterRaymond F. BrownK. Y. HuiJohn BlenisBentley CheathamLynn CheathamC. Ronald KahnBonnie L. Blazer‐Yost
- Topics
- Protein Kinase Regulation and GTPase Signaling (16 papers)PI3K/AKT/mTOR signaling in cancer (7 papers)Blood Coagulation and Thrombosis Mechanisms (6 papers)
- Partner nations
- United StatesSingaporeCanada
In The Last Decade
Chris J. Vlahos
68 papers receiving 8.4k citations
Hit Papers
Peers
Comparison fields: 5 of 133
- Molecular Biology 5.8k
- Immunology 1.1k
- Cell Biology 1.0k
- Physiology 939
- Oncology 928
Countries citing papers authored by Chris J. Vlahos
This map shows the geographic impact of Chris J. Vlahos's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Chris J. Vlahos with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Chris J. Vlahos more than expected).
Fields of papers citing papers by Chris J. Vlahos
This network shows the impact of papers produced by Chris J. Vlahos. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Chris J. Vlahos. The network helps show where Chris J. Vlahos may publish in the future.
Co-authorship network of co-authors of Chris J. Vlahos
This figure shows the co-authorship network connecting the top 25 collaborators of Chris J. Vlahos. A scholar is included among the top collaborators of Chris J. Vlahos based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Chris J. Vlahos. Chris J. Vlahos is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 22 | |
| 2 | 103 | |
| 3 | 41 | |
| 4 | 87 | |
| 5 | 66 | |
| 6 | 77 | |
| 7 | 43 | |
| 8 | 28 | |
| 9 | 145 | |
| 10 | 52 | |
| 11 | 177 | |
| 12 | 110 | |
| 13 | 19 | |
| 14 | Phosphatidylinositol 3-Kinase Activation Is Required for Insulin Stimulation of pp70 S6 Kinase, DNA Synthesis, and Glucose Transporter Translocationbreakdown → | 913 |
| 15 | 120 | |
| 16 | 65 | |
| 17 | 52 | |
| 18 | 166 | |
| 19 | 13 | |
| 20 | 3 |
About Chris J. Vlahos
Chris J. Vlahos is a scholar working on Virology, Molecular Biology and Hematology, having authored 68 papers that have together received 8.5k indexed citations. Recurring topics across this work include Protein Kinase Regulation and GTPase Signaling (16 papers), PI3K/AKT/mTOR signaling in cancer (7 papers) and Blood Coagulation and Thrombosis Mechanisms (6 papers). The work is most often cited by research in Molecular Biology (5.8k citations), Immunology and Allergy (394 citations) and Cell Biology (1.0k citations). Chris J. Vlahos has collaborated with scholars based in United States, Singapore and Canada. Frequent co-authors include William F. Matter, Raymond F. Brown, K. Y. Hui, John Blenis, Bentley Cheatham, Lynn Cheatham, C. Ronald Kahn, C. Ronald Kahn, Bonnie L. Blazer‐Yost and George L. King. Their work appears in journals such as Journal of Biological Chemistry, Circulation and Blood.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.