Charles Redwood
- Cardiology and Cardiovascular Medicine top 0.5%
- Molecular Biology top 2%
- Cell Biology top 2%
- Epidemiology
- Cellular and Molecular Neuroscience top 10%
- Co-authors
- Hugh WatkinsSteven B. MarstonHouman AshrafianPaul RobinsonEmily FlashmanEdward BlairJohanna C. Moolman‐SmookWilliam J. McKenna
- Topics
- Cardiomyopathy and Myosin Studies (94 papers)Cardiovascular Effects of Exercise (54 papers)Muscle Physiology and Disorders (44 papers)
- Partner nations
- United KingdomRussiaUnited States
In The Last Decade
Charles Redwood
113 papers receiving 4.9k citations
Peers
Comparison fields: 5 of 104
- Cardiology and Cardiovascular Medicine 4.1k
- Molecular Biology 2.9k
- Cell Biology 468
- Epidemiology 247
- Cellular and Molecular Neuroscience 230
Countries citing papers authored by Charles Redwood
This map shows the geographic impact of Charles Redwood's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Charles Redwood with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Charles Redwood more than expected).
Fields of papers citing papers by Charles Redwood
This network shows the impact of papers produced by Charles Redwood. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Charles Redwood. The network helps show where Charles Redwood may publish in the future.
Co-authorship network of co-authors of Charles Redwood
This figure shows the co-authorship network connecting the top 25 collaborators of Charles Redwood. A scholar is included among the top collaborators of Charles Redwood based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Charles Redwood. Charles Redwood is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 10 | |
| 2 | 3 | |
| 3 | MYBPC3 Mutations Cause Hypertrophic Cardiomyopathy by Dysregulating Myosin: Implications for Therapy | 1 |
| 4 | 15 | |
| 5 | 4 | |
| 6 | 38 | |
| 7 | 7 | |
| 8 | Functional investigation of troponin with the homozygous HCM mutation, TNNT2 K280N, obtained from an explanted heart | 1 |
| 9 | Alteration of beta-tropomyosin function by nernaline myopathy-causing mutations E117K and Q147P | 1 |
| 10 | In vitro motility studies of HCM and DCM mutations in cardiac muscle actin | 1 |
| 11 | Mechanical effects of human cardiac troponin C mutation Gly159Asp in exchanged rabbit psoas fibres | 3 |
| 12 | A novel interaction between the C5 and C8 domains of myosin binding protein-C and its modulation by HCM mutations | 1 |
| 13 | Comparison of the functional effects of mutations in troponin T which cause dilated and hypertrophic cardiomyopathies | 1 |
| 14 | 31 | |
| 15 | Hypertrophic cardiomyopathy mutations in the gamma 2 subunit of AMP-activated kinase suggest a central role of energy compromise in disease pathogenesis | 1 |
| 16 | Mutations in the gamma2 subunit of AMP-activated protein kinase cause familial hypertrophic cardiomyopathy: evidence for the central role of energy compromise in disease pathogenesis. | 115 |
| 17 | Interaction analyses elucidate the pathophysiology of HCM-causing missense mutations in cardiac myosin binding protein-C. | 1 |
| 18 | Ca2+regulatory properties of a truncated troponin T that causes FHC depend on the ratio of mutant to wild-type peptide. | 1 |
| 19 | Defective inhibitory properties of human cardiac troponin I mutants that cause familial hypertrophic cardiomyopathy (FHC) | 4 |
| 20 | Functional effects of hypertrophic cardiomyopathy mutations in alpha-tropomyosin on Ca2+ regulation of thm filaments | 1 |
About Charles Redwood
Charles Redwood is a scholar working on Cardiology and Cardiovascular Medicine, Molecular Biology and Aging, having authored 113 papers that have together received 4.9k indexed citations. Recurring topics across this work include Cardiomyopathy and Myosin Studies (94 papers), Cardiovascular Effects of Exercise (54 papers) and Muscle Physiology and Disorders (44 papers). The work is most often cited by research in Cardiology and Cardiovascular Medicine (4.1k citations), Molecular Biology (2.9k citations) and Cell Biology (468 citations). Charles Redwood has collaborated with scholars based in United Kingdom, Russia and United States. Frequent co-authors include Hugh Watkins, Steven B. Marston, Houman Ashrafian, Paul Robinson, Emily Flashman, Edward Blair, Johanna C. Moolman‐Smook, William J. McKenna, Kathryn Elliott and Peter Griffiths. Their work appears in journals such as New England Journal of Medicine, Journal of Biological Chemistry and Circulation.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.