Katja Gehmlich

2.4k total citations · 1 hit paper
55 papers, 1.5k citations indexed

About

Katja Gehmlich is a scholar working on Cardiology and Cardiovascular Medicine, Molecular Biology and Cell Biology. According to data from OpenAlex, Katja Gehmlich has authored 55 papers receiving a total of 1.5k indexed citations (citations by other indexed papers that have themselves been cited), including 41 papers in Cardiology and Cardiovascular Medicine, 18 papers in Molecular Biology and 10 papers in Cell Biology. Recurrent topics in Katja Gehmlich's work include Cardiomyopathy and Myosin Studies (29 papers), Cardiovascular Effects of Exercise (20 papers) and Muscle Physiology and Disorders (8 papers). Katja Gehmlich is often cited by papers focused on Cardiomyopathy and Myosin Studies (29 papers), Cardiovascular Effects of Exercise (20 papers) and Muscle Physiology and Disorders (8 papers). Katja Gehmlich collaborates with scholars based in United Kingdom, Germany and United States. Katja Gehmlich's co-authors include Elisabeth Ehler, Dieter O. Fürst, Petros Syrris, William J. McKenna, Chris Denning, Davor Pavlović, Caitlin Hall, Christian Geier, Peter F. M. van der Ven and Giovanni Quarta and has published in prestigious journals such as Circulation, Nature Communications and Journal of Molecular Biology.

In The Last Decade

Katja Gehmlich

50 papers receiving 1.5k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Complex Relationship Between Cardiac Fibroblasts and Cardiomyocytes in Health and Disease

2021 131 citations Caitlin Hall, Katja Gehmlich et al. profile →

Peers

Countries citing papers authored by Katja Gehmlich

Since Specialization

Citations

This map shows the geographic impact of Katja Gehmlich's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Katja Gehmlich with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Katja Gehmlich more than expected).

Fields of papers citing papers by Katja Gehmlich

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Katja Gehmlich. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Katja Gehmlich. The network helps show where Katja Gehmlich may publish in the future.

Co-authorship network of co-authors of Katja Gehmlich

This figure shows the co-authorship network connecting the top 25 collaborators of Katja Gehmlich. A scholar is included among the top collaborators of Katja Gehmlich based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Katja Gehmlich. Katja Gehmlich is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

#	Work	Indexed citations
1	HCM-Associated MuRF1 Variants Compromise Ubiquitylation and Are Predicted to Alter Protein Structure 2025 ·International Journal of Molecular Sciences ·(unknown), (unknown), (unknown), (unknown), (unknown), (unknown), Katja Gehmlich, Fiyaz Mohammed, Yu‐Chiang Lai	0
2	Mechanosensitive biochemical imprinting of the talin interaction with DLC1 regulates RhoA activity and cardiomyocyte remodeling 2025 ·Science Advances ·(unknown), (unknown), (unknown), (unknown), (unknown), (unknown), (unknown), (unknown), (unknown), Davor Pavlović, Katja Gehmlich, Giuseppe Faggian, Pamela Swiatlowska, Elisabeth Ehler, James A. Levitt, Simon P. Poland, Simon Ameer‐Beg, Benjamin T. Goult, Thomas Iskratsch	0
3	Exploring Syndecan-4 and MLP and Their Interaction in Primary Cardiomyocytes and H9c2 Cells 2024 ·Cells ·(unknown), Marianne Lunde, Katja Gehmlich, Geir Christensen, William E. Louch, Cathrine R. Carlson	1
4	Arrhythmogenesis in Fabry Disease 2024 ·Current Cardiology Reports ·(unknown), (unknown), Christopher O’Shea, Andrew P. Holmes, Manish Kalla, Katja Gehmlich, Tarekegn Geberhiwot, Richard P. Steeds	5
5	Defining the cardiovascular phenotype of adults with Alström syndrome 2024 ·International Journal of Cardiology ·(unknown), Leena Patel, (unknown), Christopher O’Shea, (unknown), (unknown), (unknown), Shanat Baig, Karina V Bunting, Katja Gehmlich, (unknown), Richard P. Steeds	3
6	Characterisation of infantile cardiomyopathy in Alström syndrome using ALMS1 knockout induced pluripotent stem cell derived cardiomyocyte model 2024 ·Molecular Genetics and Metabolism ·(unknown), (unknown), (unknown), Christopher O’Shea, (unknown), (unknown), Caitlin Hall, Benjamin Davies, (unknown), Davor Pavlović, Ashish Chikermane, (unknown), Richard P. Steeds, Katja Gehmlich	2
7	Structural and signaling proteins in the Z-disk and their role in cardiomyopathies 2023 ·Frontiers in Physiology ·(unknown), Katja Gehmlich	17
8	The Utilisation of Hydrogels for iPSC-Cardiomyocyte Research 2023 ·International Journal of Molecular Sciences ·(unknown), Joshua C. Worch, Andrew P. Dove, Katja Gehmlich	7
9	Should young athletes be screened for cardiomyopathies to reduce the burden of sudden cardiac death in athletes? 2023 ·Biophysical Reviews ·(unknown), Mauricio Villarroel, Katja Gehmlich	0
10	Phenoage and longitudinal changes on transthoracic echocardiography in Alström syndrome: a disease of accelerated ageing? 2023 ·GeroScience ·Leena Patel, (unknown), (unknown), (unknown), Shanat Baig, Karina V Bunting, James Hodson, Katja Gehmlich, Janet M. Lord, Tarekegn Geberhiwot, Richard P. Steeds	3
11	Insights into the Role of a Cardiomyopathy-Causing Genetic Variant in ACTN2 2023 ·Cells ·(unknown), (unknown), (unknown), Charlotte Hooper, Gillian Douglas, Violetta Steeples, (unknown), (unknown), Jasmeet S. Reyat, (unknown), Elisabeth Ehler, Pauline M. Bennett, Jacinta I. Kalisch‐Smith, Duncan B. Sparrow, Benjamin Davies, Kristina Djinović‐Carugo, Mathias Gautel, Hugh Watkins, Katja Gehmlich	11
12	Familial atrial fibrillation mutation M1875T-SCN5A increases early sodium current and dampens the effect of flecainide 2022 ·EP Europace ·Molly O’Reilly, (unknown), Christopher O’Shea, (unknown), (unknown), Jasmeet S. Reyat, (unknown), (unknown), (unknown), (unknown), Lisa Fortmueller, Katja Gehmlich, Davor Pavlović, Boris V. Skryabin, Andrew P. Holmes, Paulus Kirchhof, Larissa Fabritz	12
13	Nitric Oxide and Mechano-Electrical Transduction in Cardiomyocytes 2020 ·Frontiers in Physiology ·Hannah E. Boycott, My‐Nhan Nguyen, (unknown), Katja Gehmlich, Paul Robinson	21
14	HspB1 phosphorylation regulates its intramolecular dynamics and mechanosensitive molecular chaperone interaction with filamin C 2019 ·Science Advances ·M Collier, T. Reid Alderson, (unknown), (unknown), (unknown), Timothy M. Allison, Matteo T. Degiacomi, (unknown), Georg Mlynek, Dieter O. Fürst, Peter F. M. van der Ven, Kristina Djinović‐Carugo, Andrew J. Baldwin, Hugh Watkins, Katja Gehmlich, Justin L. P. Benesch	54
15	Mutant Muscle LIM Protein C58G causes cardiomyopathy through protein depletion 2018 ·Journal of Molecular and Cellular Cardiology ·(unknown), Matt Kelly, Charlotte Hooper, Arash Yavari, (unknown), Mohamed Bellahcene, (unknown), Giulia Poloni, Anuj Goel, Theodosios Kyriakou, (unknown), Elisabeth Ehler, Eugene V. Makeyev, Stephan Lange, Houman Ashrafian, Charles Redwood, Benjamin Davies, Hugh Watkins, Katja Gehmlich	15
16	Combination of Whole Genome Sequencing, Linkage, and Functional Studies Implicates a Missense Mutation in Titin as a Cause of Autosomal Dominant Cardiomyopathy With Features of Left Ventricular Noncompaction 2016 ·Circulation Cardiovascular Genetics ·Robert Hastings, (unknown), Charlotte Hooper, Elizabeth Ormondroyd, Alistair T. Pagnamenta, Stefano Lise, Silvia Salatino, Samantha J.L. Knight, Jenny C. Taylor, Kate Thomson, (unknown), S.D. Chatziefthimiou, Petr V. Konarev, Matthias Wilmanns, Elisabeth Ehler, Andrea Ghisleni, Mathias Gautel, Edward Blair, Hugh Watkins, Katja Gehmlich	43
17	Heterozygous plakoglobin deficiency results in increased biventricular beta-catenin expression 2015 ·European Heart Journal ·(unknown), Farisa Syeda, (unknown), (unknown), Katja Gehmlich, Paulus Kirchhof, Larissa Fabritz	1
18	Changes in the cardiac metabolome caused by perhexiline treatment in a mouse model of hypertrophic cardiomyopathy 2014 ·Molecular BioSystems ·Katja Gehmlich, Michael S. Dodd, J. William Allwood, Matt Kelly, Mohamed Bellahcene, Heena V. Lad, Alexander Stockenhuber, Charlotte Hooper, Houman Ashrafian, Charles Redwood, Lucie Carrier, Warwick B. Dunn	38
19	Mechanistic insights into arrhythmogenic right ventricular cardiomyopathy caused by desmocollin-2 mutations 2010 ·Cardiovascular Research ·Katja Gehmlich, Petros Syrris, Emma Peskett, Alison Evans, Elisabeth Ehler, Angeliki Asimaki, Aris Anastasakis, Adalena Tsatsopoulou, (unknown), Christodoulos Stefanadis, Jeffrey E. Saffitz, Nikos Protonotarios, William J. McKenna	41
20	Cyclin B degradation leads to NuMA release from dynein/dynactin and from spindle poles 2004 ·EMBO Reports ·Katja Gehmlich, Laurence Haren, Andreas Merdes	43

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact