K Schwartz

5.8k total citations
79 papers, 4.6k citations indexed

About

K Schwartz is a scholar working on Cardiology and Cardiovascular Medicine, Molecular Biology and Cell Biology. According to data from OpenAlex, K Schwartz has authored 79 papers receiving a total of 4.6k indexed citations (citations by other indexed papers that have themselves been cited), including 52 papers in Cardiology and Cardiovascular Medicine, 47 papers in Molecular Biology and 10 papers in Cell Biology. Recurrent topics in K Schwartz's work include Cardiomyopathy and Myosin Studies (41 papers), Muscle Physiology and Disorders (20 papers) and Cardiovascular Effects of Exercise (16 papers). K Schwartz is often cited by papers focused on Cardiomyopathy and Myosin Studies (41 papers), Muscle Physiology and Disorders (20 papers) and Cardiovascular Effects of Exercise (16 papers). K Schwartz collaborates with scholars based in France, United States and United Kingdom. K Schwartz's co-authors include Anne‐Marie Lompré, P. Bouveret, J.-J. Mercadier, C Wisnewsky, D. de la Bastie, Bernard Swynghedauw, Jane‐Lise Samuel, Stefano Schiaffino, L. Rappaport and J. Bercovici and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Circulation.

In The Last Decade

K Schwartz

78 papers receiving 4.5k citations

Peers

Countries citing papers authored by K Schwartz

Since Specialization

Citations

This map shows the geographic impact of K Schwartz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by K Schwartz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites K Schwartz more than expected).

Fields of papers citing papers by K Schwartz

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by K Schwartz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by K Schwartz. The network helps show where K Schwartz may publish in the future.

Co-authorship network of co-authors of K Schwartz

This figure shows the co-authorship network connecting the top 25 collaborators of K Schwartz. A scholar is included among the top collaborators of K Schwartz based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with K Schwartz. K Schwartz is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Work	Indexed citations
1	A new polymorphism in human calmodulin III gene promoter is a potential modifier gene for familial hypertrophic cardiomyopathy 2009 ·European Heart Journal ·Felix W. Friedrich, Pedro Bausero, Yi Sun, András Treszl, Edgar R. Kramer, (unknown), Pascale Richard, Karl Wegscheider, K Schwartz, Dulce Brito, Eloisa Arbustini, Anders Waldenström, Richard Isnard, Michel Komajda, Thomas Eschenhagen, Lucie Carrier, (unknown)	32
2	Congestive Heart Failure after Myocardial Infarction in the Rat: Cardiac Force and Spontaneous Sarcomere Activity 2004 ·Annals of the New York Academy of Sciences ·A. Davidoff, Penelope A. Boyden, K Schwartz, J.B. Michel, (unknown), Masakazu Obayashi, Deborah L. Crabbe, Henk E.D.J. ter Keurs	25
3	Nuclear envelope alterations in fibroblasts from patients with muscular dystrophy, cardiomyopathy, and partial lipodystrophy carrying lamin A/C gene mutations 2004 ·Muscle & Nerve ·Antoine Muchir, Jacques Médioni, (unknown), Catherine Massart, Takuro Arimura, Anneke J. van der Kooi, Isabelle Desguerre, M. Mayer, Xavier Ferrer, Sylvain Briault, Michio Hirano, Howard J. Worman, Audrey Mallet, Manfred Wehnert, K Schwartz, Gisèle Bonne	139
4	Selective Muscle Involvement on Magnetic Resonance Imaging in Autosomal Dominant Emery-Dreifuss Muscular Dystrophy 2002 ·Neuropediatrics ·Eugenio Mercuri, Serena J. Counsell, Joanna Allsop, Heinz Jungbluth, Maria Kinali, Gisèle Bonne, K Schwartz, Graeme M. Bydder, Victor Dubowitz, Francesco Muntoni	73
5	Electrotransfer of naked DNA in the skeletal muscles of animal models of muscular dystrophies 2001 ·Gene Therapy ·Jean‐Thomas Vilquin, (unknown), Marion Paturneau-Jouas, Pierre Chapdelaine, Nicolas Boissel, P. Delaère, Jacques P. Tremblay, Daniel Scherman, (unknown), K Schwartz	84
6	Double heterozygosity for mutations in the beta-myosin heavy chain and in the cardiac myosin binding protein C genes in a family with hypertrophic cardiomyopathy. 1999 ·PubMed ·Pascale Richard, Richard Isnard, Lucie Carrier, Olivier Dubourg, (unknown), (unknown), Gisèle Bonne, Françoise Gary, Philippe Charron, (unknown), Michel Komajda, K Schwartz, Bernard Hainque	60
7	Myoblast transplantations lead to the expression of the laminin α2 chain in normal and dystrophic (dy/dy) mouse muscles 1999 ·Gene Therapy ·Jean‐Thomas Vilquin, B. Guérette, Jack Puymirat, David Yaffe, F. Tomé, Michel Fardeau, Marc Fiszman, K Schwartz, Jacques P. Tremblay	24
8	The IVth workshop on Duchenne muscular dystrophy gene therapy 1999 ·The Journal of Gene Medicine ·K Schwartz, F Leterrier	1
9	Gene delivery to the myocardium by intrapericardial injection 1999 ·Gene Therapy ·Yves Fromes, (unknown), (unknown), H. Barry Collin, Andrée Rouche, Albert Hagège, K Schwartz, Marc Fiszman	95
10	Calcium transport proteins in the nonfailing and failing heart: gene expression and function 1995 ·Journal of Molecular Medicine ·Michael Wankerl, K Schwartz	46
11	Characterization of a Novel Transcript of Retroviral Origin Expressed in Rat Heart and Liver 1995 ·Journal of Molecular and Cellular Cardiology ·X. Martin, K Schwartz, Kenneth R. Boheler	7
12	Contractile protein gene expression in serum-free cultured adult rat cardiac myocytes 1993 ·Pflügers Archiv - European Journal of Physiology ·Isabelle Dubus, L. Rappaport, A Barrieux, (unknown), K Schwartz, Jane‐Lise Samuel	12
13	Contractile proteins and sarcoplasmic reticulum calcium-ATPase gene expression in the hypertrophied and failing heart 1992 ·Steinkopff eBooks ·K Schwartz, Lucie Carrier, Anne‐Marie Lompré, J.-J. Mercadier, Kenneth R. Boheler	27
14	Skeletal actin mRNA increases in the human heart during ontogenic development and is the major isoform of control and failing adult hearts. 1991 ·Journal of Clinical Investigation ·Kenneth R. Boheler, Lucie Carrier, D. de la Bastie, Paul D. Allen, Michel Komajda, J.-J. Mercadier, K Schwartz	86
15	Regulation of myosin heavy chain and actin isogenes expression during cardiac growth 1991 ·Molecular and Cellular Biochemistry ·Kenneth R. Boheler, Lucie Carrier, C. Chassagne, D. de la Bastie, J.-J. Mercadier, K Schwartz	15
16	Phénoconversion et transduction mécanogénique du cœur de mammifère 1990 ·médecine/sciences ·K Schwartz	1
17	Striated muscle overload 1988 ·European Heart Journal ·Bernard Swynghedauw, K Schwartz, Bernward Lauer, Anne‐Marie Lompré, J.-J. Mercadier, Jane‐Lise Samuel, L. Rappaport	15
18	Myosin heavy chain messenger RNA and protein isoform transitions during cardiac hypertrophy. Interaction between hemodynamic and thyroid hormone-induced signals. 1987 ·Journal of Clinical Investigation ·Seigo Izumo, Anne‐Marie Lompré, Rumiko Matsuoka, Gideon Koren, K Schwartz, B Nadal-Ginard, Vijak Mahdavi	382
19	Isomyosins, microtubules and desmin during the onset of cardiac hypertrophy in the rat 1984 ·European Heart Journal ·L. Rappaport, Jane‐Lise Samuel, (unknown), Lawrence B. Bugaisky, F. Marotte, Anne Mercadier, K Schwartz	16
20	Species- and age-dependent changes in the relative amounts of cardiac myosin isoenzymes in mammals 1981 ·Developmental Biology ·Anne‐Marie Lompré, J.-J. Mercadier, C Wisnewsky, P. Bouveret, C. Pantaloni, Anne d’Albis, K Schwartz	297

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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