Ingegerd Östman‐Smith

2.3k total citations
49 papers, 1.6k citations indexed

About

Ingegerd Östman‐Smith is a scholar working on Cardiology and Cardiovascular Medicine, Epidemiology and Molecular Biology. According to data from OpenAlex, Ingegerd Östman‐Smith has authored 49 papers receiving a total of 1.6k indexed citations (citations by other indexed papers that have themselves been cited), including 40 papers in Cardiology and Cardiovascular Medicine, 14 papers in Epidemiology and 12 papers in Molecular Biology. Recurrent topics in Ingegerd Östman‐Smith's work include Cardiomyopathy and Myosin Studies (33 papers), Cardiovascular Effects of Exercise (17 papers) and Cardiovascular Function and Risk Factors (13 papers). Ingegerd Östman‐Smith is often cited by papers focused on Cardiomyopathy and Myosin Studies (33 papers), Cardiovascular Effects of Exercise (17 papers) and Cardiovascular Function and Risk Factors (13 papers). Ingegerd Östman‐Smith collaborates with scholars based in Sweden, United Kingdom and Czechia. Ingegerd Östman‐Smith's co-authors include Anne de‐Wahl Granelli, Hugh Watkins, Edward Blair, Göran Wettrell, Tomas Riesenfeld, William J. McKenna, Ernest Boehm, Andrew M. Blamire, B. Rajagopalan and Peter Styles and has published in prestigious journals such as New England Journal of Medicine, Circulation and Journal of the American College of Cardiology.

In The Last Decade

Ingegerd Östman‐Smith

46 papers receiving 1.5k citations

Peers

Ingegerd Östman‐Smith
P Cosnay France
Luís R. Lopes United Kingdom
Ralf Seipelt Germany
Tjeerd Germans Netherlands
P Cosnay France
Ingegerd Östman‐Smith
Citations per year, relative to Ingegerd Östman‐Smith Ingegerd Östman‐Smith (= 1×) peers P Cosnay

Countries citing papers authored by Ingegerd Östman‐Smith

Since Specialization
Citations

This map shows the geographic impact of Ingegerd Östman‐Smith's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ingegerd Östman‐Smith with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ingegerd Östman‐Smith more than expected).

Fields of papers citing papers by Ingegerd Östman‐Smith

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ingegerd Östman‐Smith. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ingegerd Östman‐Smith. The network helps show where Ingegerd Östman‐Smith may publish in the future.

Co-authorship network of co-authors of Ingegerd Östman‐Smith

This figure shows the co-authorship network connecting the top 25 collaborators of Ingegerd Östman‐Smith. A scholar is included among the top collaborators of Ingegerd Östman‐Smith based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ingegerd Östman‐Smith. Ingegerd Östman‐Smith is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
2.
Bergman, Gunnar, et al.. (2024). Screening for Critical Congenital Heart Defects in Sweden. Obstetrical & Gynecological Survey. 79(4). 185–187. 1 indexed citations
3.
Östman‐Smith, Ingegerd, Henrik Gréen, Cecilia Gunnarsson, et al.. (2024). Biomarkers and Proteomics in Sarcomeric Hypertrophic Cardiomyopathy in the Young—FGF-21 Highly Associated with Overt Disease. Journal of Cardiovascular Development and Disease. 11(4). 105–105. 1 indexed citations
4.
Jablonowski, Robert, Ingegerd Östman‐Smith, Marcus Carlsson, et al.. (2024). Spatial QRS-T angle can indicate presence of myocardial fibrosis in pediatric and young adult patients with hypertrophic cardiomyopathy. Journal of Electrocardiology. 89. 153859–153859.
5.
Kaski, Juan Pablo, Janneke A.E. Kammeraad, Nico A. Blom, et al.. (2023). Indications and management of implantable cardioverter-defibrillator therapy in childhood hypertrophic cardiomyopathy. Cardiology in the Young. 33(5). 681–698. 8 indexed citations
6.
Andersson, Bert, et al.. (2020). Morbidity and resource usage after myectomy- or pacing-treatment in hypertrophic obstructive cardiomyopathy: A case-control study. International Journal of Cardiology. 322. 197–203. 4 indexed citations
7.
Östman‐Smith, Ingegerd. (2011). Sudden cardiac death in young athletes. Open Access Journal of Sports Medicine. 2. 85–85. 2 indexed citations
8.
Falkenberg, Cecilia, et al.. (2010). A study of the physiological consequences of sympathetic denervation of the heart caused by the arterial switch procedure. Cardiology in the Young. 20(2). 150–158. 10 indexed citations
9.
Östman‐Smith, Ingegerd. (2010). Hypertrophic cardiomyopathy in childhood and adolescence – strategies to prevent sudden death. Fundamental and Clinical Pharmacology. 24(5). 637–652. 36 indexed citations
10.
Bratt, Ewa‐Lena, Ingegerd Östman‐Smith, Carina Sparud‐Lundin, & Bertil Axelsson. (2010). Parents’ experiences of having an asymptomatic child diagnosed with hypertrophic cardiomyopathy through family screening. Cardiology in the Young. 21(1). 8–14. 13 indexed citations
11.
Östman‐Smith, Ingegerd, Aase Wisten, Eva Nylander, et al.. (2009). Electrocardiographic amplitudes: a new risk factor for sudden death in hypertrophic cardiomyopathy. European Heart Journal. 31(4). 439–449. 53 indexed citations
12.
Kollberg, Gittan, M. Tulinius, Thomas Gilljam, et al.. (2007). Cardiomyopathy and Exercise Intolerance in Muscle Glycogen Storage Disease 0. New England Journal of Medicine. 357(15). 1507–1514. 94 indexed citations
13.
Östman‐Smith, Ingegerd, Göran Wettrell, Barry R. Keeton, et al.. (2005). Echocardiographic and electrocardiographic identification of those children with hypertrophic cardiomyopathy who should be considered at high-risk of dying suddenly. Cardiology in the Young. 15(6). 632–642. 55 indexed citations
14.
Crilley, Jenifer, Ernest Boehm, Edward Blair, et al.. (2003). Hypertrophic cardiomyopathy due to sarcomeric gene mutations is characterized by impaired energy metabolism irrespective of the degree of hypertrophy. Journal of the American College of Cardiology. 41(10). 1776–1782. 329 indexed citations
15.
Oliveira, María José, Javed Ehtisham, Charles Redwood, et al.. (2002). Mutation analysis of genes encoding Subunits of AMP-activated protein kinase (AMPK) in inherited cardiomyopathies. Circulation. 106. 140–140. 3 indexed citations
16.
Blair, E, Charles Redwood, Sandra Marisa Oliveira, et al.. (2001). Hypertrophic cardiomyopathy mutations in the gamma 2 subunit of AMP-activated kinase suggest a central role of energy compromise in disease pathogenesis. Circulation. 104. 21–21. 1 indexed citations
17.
Blair, Edward, Charles Redwood, Houman Ashrafian, et al.. (2001). Mutations in the gamma2 subunit of AMP-activated protein kinase cause familial hypertrophic cardiomyopathy: evidence for the central role of energy compromise in disease pathogenesis.. Journal of Medical Genetics. 38. 115 indexed citations
18.
Blair, E, Ingegerd Östman‐Smith, & Hugh Watkins. (2000). Double mutations in CIS can confound genotype-phenotype correlations in hypertrophic cardiomyopathy. Circulation. 102(18). 178–178. 1 indexed citations
19.
Manning, N., et al.. (2000). Prenatal diagnosis and successful preterm delivery of a fetus with long QT syndrome. BJOG An International Journal of Obstetrics & Gynaecology. 107(8). 1049–1051. 6 indexed citations
20.
Östman‐Smith, Ingegerd, Göran Wettrell, & Tomas Riesenfeld. (1999). A cohort study of childhood hypertrophic cardiomyopathy. Journal of the American College of Cardiology. 34(6). 1813–1822. 112 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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