Benjamin Schmid

3.5k citations
60 papers · 1.9k · 1 hit paper · h-index 22

Impact in

Papers in

    • Pluripotent Stem Cells Research 25
    • CRISPR and Genetic Engineering 22
    • Biomedical Ethics and Regulation 6
    • Alzheimer's disease research and treatments 4

Benjamin Schmid

60 papers receiving 1.9k citations

Hit Papers

iPSC-derived neurons from GBA1-associated Parkinson’s disease patients show autophagic defects and impaired calcium homeostasis 2014 · 406 citations
4060+4+8Years since publication100200300400

Peers

Benjamin Schmid
Comparison fields: 5 of 116
  • Aging 70
  • Developmental Neuroscience 133
  • Neurology 412
  • Cellular and Molecular Neuroscience 389
  • Physiology 551
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Jay Penney United States
Javier Cuesta Spain
Brad E. Morrison United States
Jin H. Son United States
Claudia Crosio Italy
Natalija Popović Spain
Edward B. Holson United States
Melissa Nassif Chile
Mathieu Bourdenx France
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Citations per year

Countries citing papers authored by Benjamin Schmid

Since Specialization
Citations

This map shows the geographic impact of Benjamin Schmid's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Benjamin Schmid with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Benjamin Schmid more than expected).

Fields of papers citing papers by Benjamin Schmid

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Benjamin Schmid. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Benjamin Schmid. The network helps show where Benjamin Schmid may publish in the future.

Co-authors

The 25 scholars most cited alongside Benjamin Schmid, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Benjamin Schmid Line = papers co-authored together Benjamin Schmid links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 60 papers — load more, or switch the sort, to bring in the rest.

#Work
1
iPSC-derived neurons from GBA1-associated Parkinson’s disease patients show autophagic defects and impaired calcium homeostasis
Hit paper breakdown →
2014406
2 2013295
3 2001102
4 200195
5 200186
6 200869
7 201364
8 200761
9 199256
10 199150
11 201746
12 201642
13 201033
14 201932
15 201630
16 201227
17 201724
18 200723
19 202122
20 201022

About Benjamin Schmid

Benjamin Schmid is a scholar working on Molecular Biology, Physiology, Cellular and Molecular Neuroscience, Plant Science and Aging, having authored 60 papers that have together received 1.9k indexed citations. Recurring topics across this work include Pluripotent Stem Cells Research (25 papers), CRISPR and Genetic Engineering (22 papers), Genetic Neurodegenerative Diseases (8 papers), Genetics, Aging, and Longevity in Model Organisms (6 papers), Biomedical Ethics and Regulation (6 papers), Parkinson's Disease Mechanisms and Treatments (5 papers), Alzheimer's disease research and treatments (4 papers) and Neuroscience and Neural Engineering (3 papers). The work is most often cited by research in Aging (70 citations), Developmental Neuroscience (133 citations), Neurology (412 citations), Cellular and Molecular Neuroscience (389 citations) and Physiology (551 citations). Benjamin Schmid has collaborated with scholars based in Germany, Denmark and Thailand. Frequent co-authors include Thomas Gasser, Ina Kötter, Lutz Heide, Bjørn Holst, Daniela Berg, Troels T. Nielsen, Kristine Freude, Michela Deleidi, David C. Schöndorf and Florian Mayer. Their work appears in journals such as Stem Cell Research, PLoS ONE, Naunyn-Schmiedeberg s Archives of Pharmacology, Journal of Alzheimer s Disease and European Journal of Clinical Pharmacology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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