Patrizia Marini

1.9k total citations · 1 hit paper
22 papers, 1.6k citations indexed

About

Patrizia Marini is a scholar working on Molecular Biology, Cancer Research and Oncology. According to data from OpenAlex, Patrizia Marini has authored 22 papers receiving a total of 1.6k indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Molecular Biology, 11 papers in Cancer Research and 10 papers in Oncology. Recurrent topics in Patrizia Marini's work include Cell death mechanisms and regulation (16 papers), Cancer-related Molecular Pathways (6 papers) and NF-κB Signaling Pathways (6 papers). Patrizia Marini is often cited by papers focused on Cell death mechanisms and regulation (16 papers), Cancer-related Molecular Pathways (6 papers) and NF-κB Signaling Pathways (6 papers). Patrizia Marini collaborates with scholars based in Germany and United States. Patrizia Marini's co-authors include Claus Belka, Klaus Schulze‐Osthoff, Wilfried Budach, Sebastian Wesselborg, Kirsten Lauber, M. Bamberg, Gernot Stuhler, Stefan Kröber, Yi-jin Xiao and Erwin Bohn and has published in prestigious journals such as Cell, Oncogene and British Journal of Cancer.

In The Last Decade

Patrizia Marini

21 papers receiving 1.6k citations

Hit Papers

Apoptotic Cells Induce Migration of Phagocytes via Caspas... 2003 2026 2010 2018 2003 250 500 750
What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

  1. Apoptotic Cells Induce Migration of Phagocytes via Caspase-3-Mediated Release of a Lipid Attraction Signal
    2003 756 citations Kirsten Lauber, Erwin Bohn et al. Cell profile →

Peers

Patrizia Marini
Amnon Altman United States
Ling-Mei Wang United States
Abdelhadi Rebbaa United States
Francesco Pacifico Italy
David Fernández United States
Paul J. Coffer Netherlands
Céline Charvet France
Sathish Kumar Mungamuri India
Anthony Valentijn United Kingdom
Makoto Katsumata United States
Amnon Altman United States
Patrizia Marini
Citations per year, relative to Patrizia Marini Patrizia Marini (= 1×) peers Amnon Altman

Countries citing papers authored by Patrizia Marini

Since Specialization
Citations

This map shows the geographic impact of Patrizia Marini's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Patrizia Marini with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Patrizia Marini more than expected).

Fields of papers citing papers by Patrizia Marini

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Patrizia Marini. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Patrizia Marini. The network helps show where Patrizia Marini may publish in the future.

Co-authorship network of co-authors of Patrizia Marini

This figure shows the co-authorship network connecting the top 25 collaborators of Patrizia Marini. A scholar is included among the top collaborators of Patrizia Marini based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Patrizia Marini. Patrizia Marini is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Niyazi, Maximilian, Patrizia Marini, Peter T. Daniel, et al.. (2009). Efficacy of a Triple Treatment with Irradiation, Agonistic TRAIL Receptor Antibodies and EGFR Blockade. Strahlentherapie und Onkologie. 185(1). 8–18. 16 indexed citations
2.
Li, Minglun, Ute Ganswindt, Patrizia Marini, et al.. (2009). Aurora kinase inhibitor ZM447439 induces apoptosis via mitochondrial pathways. Biochemical Pharmacology. 79(2). 122–129. 53 indexed citations
3.
4.
Marini, Patrizia, et al.. (2009). Combination of the Pro-Apoptotic TRAIL-Receptor Antibody Mapatumumab With Ionizing Radiation Strongly Increases Long-Term Tumor Control Under Ambient and Hypoxic Conditions. International Journal of Radiation Oncology*Biology*Physics. 75(1). 198–202. 16 indexed citations
5.
6.
Marini, Patrizia, A. Schmid, Verena Jendrossek, et al.. (2005). Irradiation specifically sensitises solid tumour cell lines to TRAIL mediated apoptosis. BMC Cancer. 5(1). 5–5. 76 indexed citations
7.
Weinmann, Martin, et al.. (2004). Influence of hypoxia on TRAIL-induced apoptosis in tumor cells. International Journal of Radiation Oncology*Biology*Physics. 58(2). 386–396. 30 indexed citations
8.
Marini, Patrizia & Claus Belka. (2003). Death Receptor Ligands: New Strategies for Combined Treatment with Ionizing Radiation. PubMed. 3(5). 334–342. 12 indexed citations
9.
Lauber, Kirsten, Erwin Bohn, Stefan Kröber, et al.. (2003). Apoptotic Cells Induce Migration of Phagocytes via Caspase-3-Mediated Release of a Lipid Attraction Signal. Cell. 113(6). 717–730. 756 indexed citations breakdown →
10.
Marini, Patrizia, et al.. (2003). Molecular requirements for the combined effects of TRAIL and ionising radiation. Radiotherapy and Oncology. 68(2). 189–198. 22 indexed citations
11.
Belka, Claus, et al.. (2003). Zelltodliganden in Kombination mit ionisierender Strahlung: Rationale und Kenntnisstand. Strahlentherapie und Onkologie. 179(3). 141–151. 11 indexed citations
12.
13.
Budach, Wilfried, Frank Paulsen, Stefan Welz, et al.. (2002). Mitomycin C in combination with radiotherapy as a potent inhibitor of tumour cell repopulation in a human squamous cell carcinoma. British Journal of Cancer. 86(3). 470–476. 22 indexed citations
14.
Rudner, Justine, Claus Belka, Patrizia Marini, et al.. (2001). Radiation sensitivity and apoptosis in human lymphoma cells. International Journal of Radiation Biology. 77(1). 1–11. 37 indexed citations
15.
Belka, Claus, Benjamin Schmid, Patrizia Marini, et al.. (2001). Sensitization of resistant lymphoma cells to irradiation-induced apoptosis by the death ligand TRAIL. Oncogene. 20(17). 2190–2196. 102 indexed citations
16.
Belka, Claus, Justine Rudner, Sebastian Wesselborg, et al.. (2000). Differential role of caspase-8 and BID activation during radiation- and CD95-induced apoptosis. Oncogene. 19(9). 1181–1190. 115 indexed citations
17.
Marini, Patrizia, Roderick A.F. MacLeod, Waltraud Böhm, et al.. (1999). SiMa, a New Neuroblastoma Cell Line Combining Poor Prognostic Cytogenetic Markers with High Adrenergic Differentiation. Cancer Genetics and Cytogenetics. 112(2). 161–164. 26 indexed citations
18.
Belka, Claus, Patrizia Marini, A. Lepple-Wienhues, et al.. (1999). The tyrosine kinase Lck is required for CD95-independent caspase-8 activation and apoptosis in response to ionizing radiation. Oncogene. 18(35). 4983–4992. 83 indexed citations
19.
Belka, Claus, Patrizia Marini, Wilfried Budach, et al.. (1998). Radiation-Induced Apoptosis in Human Lymphocytes and Lymphoma Cells Critically Relies on the Up-Regulation of CD95/Fas/APO-1 Ligand. Radiation Research. 149(6). 588–588. 80 indexed citations
20.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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