Jin H. Son

8.9k total citations
60 papers, 2.4k citations indexed

About

Jin H. Son is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Neurology. According to data from OpenAlex, Jin H. Son has authored 60 papers receiving a total of 2.4k indexed citations (citations by other indexed papers that have themselves been cited), including 27 papers in Cellular and Molecular Neuroscience, 22 papers in Molecular Biology and 17 papers in Neurology. Recurrent topics in Jin H. Son's work include Parkinson's Disease Mechanisms and Treatments (16 papers), Autophagy in Disease and Therapy (10 papers) and Neurogenesis and neuroplasticity mechanisms (10 papers). Jin H. Son is often cited by papers focused on Parkinson's Disease Mechanisms and Treatments (16 papers), Autophagy in Disease and Therapy (10 papers) and Neurogenesis and neuroplasticity mechanisms (10 papers). Jin H. Son collaborates with scholars based in United States, South Korea and South Sudan. Jin H. Son's co-authors include Hong Sung Chun, Tong H. Joh, Kyung‐Hee Kim, Bruno Conti, Ji‐Young Han, Jung Hee Shim, Lorraine A. DeGiorgio, Jeong Won Jahng, Nan Min and Vincent J. Kidd and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Journal of Neuroscience.

In The Last Decade

Jin H. Son

59 papers receiving 2.3k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Jin H. Son United States 26 1.1k 751 497 469 327 60 2.4k
Santiago Ambrosio Spain 28 1.1k 1.0× 876 1.2× 771 1.6× 320 0.7× 353 1.1× 76 2.6k
Neelam Shahani United States 26 1.5k 1.4× 696 0.9× 385 0.8× 246 0.5× 588 1.8× 39 2.6k
Caty Casas Spain 26 1.1k 1.0× 678 0.9× 277 0.6× 324 0.7× 505 1.5× 51 2.4k
Marta Barrachina Spain 30 1.3k 1.2× 651 0.9× 720 1.4× 221 0.5× 706 2.2× 45 2.8k
Francesca Biagioni Italy 39 1.6k 1.5× 1.5k 2.0× 676 1.4× 564 1.2× 624 1.9× 137 4.0k
Vassiliki Nikoletopoulou Greece 16 1.2k 1.1× 451 0.6× 179 0.4× 854 1.8× 316 1.0× 31 2.7k
Yasuko Kitao Japan 32 1.2k 1.1× 555 0.7× 327 0.7× 565 1.2× 291 0.9× 70 2.7k
Christian Néri France 28 1.8k 1.7× 1.0k 1.3× 345 0.7× 429 0.9× 628 1.9× 71 3.1k
Ko Miyoshi Japan 28 1.3k 1.2× 720 1.0× 267 0.5× 235 0.5× 345 1.1× 47 2.5k
Yi Fan China 28 1.1k 1.0× 630 0.8× 359 0.7× 161 0.3× 260 0.8× 61 2.1k

Countries citing papers authored by Jin H. Son

Since Specialization
Citations

This map shows the geographic impact of Jin H. Son's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jin H. Son with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jin H. Son more than expected).

Fields of papers citing papers by Jin H. Son

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jin H. Son. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jin H. Son. The network helps show where Jin H. Son may publish in the future.

Co-authorship network of co-authors of Jin H. Son

This figure shows the co-authorship network connecting the top 25 collaborators of Jin H. Son. A scholar is included among the top collaborators of Jin H. Son based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jin H. Son. Jin H. Son is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Kim, Ji Soo, et al.. (2017). The Tnfaip8-PE complex is a novel upstream effector in the anti-autophagic action of insulin. Scientific Reports. 7(1). 6248–6248. 18 indexed citations
2.
Han, Ji‐Young, Minji Kang, Kyung‐Hee Kim, et al.. (2015). Nitric Oxide Induction of Parkin Translocation in PTEN-induced Putative Kinase 1 (PINK1) Deficiency. Journal of Biological Chemistry. 290(16). 10325–10335. 28 indexed citations
3.
Kim, Kyung‐Hee, et al.. (2012). Rescue of PINK1 Protein Null-specific Mitochondrial Complex IV Deficits by Ginsenoside Re Activation of Nitric Oxide Signaling. Journal of Biological Chemistry. 287(53). 44109–44120. 39 indexed citations
4.
Kwon, Hyun Joo, Jun Young Heo, Jung Hee Shim, et al.. (2011). DJ-1 mediates paraquat-induced dopaminergic neuronal cell death. Toxicology Letters. 202(2). 85–92. 21 indexed citations
5.
Shim, Jung Hee, Kyung‐Hee Kim, Ji‐Young Han, et al.. (2011). The antioxidant Trolox helps recovery from the familial Parkinson's disease-specific mitochondrial deficits caused by PINK1- and DJ-1-deficiency in dopaminergic neuronal cells. Mitochondrion. 11(5). 707–715. 44 indexed citations
6.
Moon, Hyo-Eun, Hyung Woo Park, Hye Young Shin, et al.. (2010). Genetic Profiling in Human Adipose Tissue-Derived Mesenchymal Stromal Cells from the Idiopathic and Familial Parkin-Deficient Patients of Parkinson`s Disease in Comparison with non-PD patients. Tissue Engineering and Regenerative Medicine. 2 indexed citations
7.
Son, Jin H., Hibiki Kawamata, Dae Joon Kim, et al.. (2005). Neurotoxicity and behavioral deficits associated with Septin 5 accumulation in dopaminergic neurons. Journal of Neurochemistry. 94(4). 1040–1053. 54 indexed citations
8.
Kawamata, Hibiki, et al.. (2004). Experimental Strategy to Identify Genes Susceptible to Oxidative Stress in Nigral Dopaminergic Neurons. Neurochemical Research. 29(6). 1223–1234. 12 indexed citations
9.
Chun, Hong Sung, et al.. (2003). Oxidative stress regulated genes in nigral dopaminergic neuronal cells: correlation with the known pathology in Parkinson’s disease. Molecular Brain Research. 110(1). 76–84. 91 indexed citations
10.
DeGiorgio, Lorraine A., Yoshinori Shimizu, Hong Sung Chun, et al.. (2002). Amyloid precursor protein gene disruption attenuates degeneration of substantia nigra compacta neurons following axotomy. Brain Research. 938(1-2). 38–44. 17 indexed citations
11.
Chun, Hong Sung, Gary E. Gibson, Lorraine A. DeGiorgio, et al.. (2001). Dopaminergic cell death induced by MPP+, oxidant and specific neurotoxicants shares the common molecular mechanism. Journal of Neurochemistry. 76(4). 1010–1021. 220 indexed citations
12.
Lee, Tae Hoon, et al.. (2000). The Effects of Teaching Methods on the Dietary Compliance and Hemoglobin A1c Level in Patients with Diabetes Mellitus. 24(5). 560–573. 4 indexed citations
13.
Chun, Hong Sung, et al.. (2000). Identification of potential compounds promoting BDNF production in nigral dopaminergic neurons. Neuroreport. 11(3). 511–514. 25 indexed citations
14.
Messer, Chad, Jin H. Son, Tong H. Joh, Klaus D. Beck, & Eric J. Nestler. (1999). Regulation of tyrosine hyroxylase gene transcription in ventral midbrain by glial cell line-derived neurotrophic factor. Synapse. 34(3). 241–243. 8 indexed citations
15.
Suh, Byung‐Chang, Jin H. Son, Tong H. Joh, & Kyong‐Tai Kim. (1997). Two Distinct P2 Purinergic Receptors, P2Y and P2U, Are Coupled to Phospholipase C in Mouse Pineal Gland Tumor Cells. Journal of Neurochemistry. 68(4). 1622–1632. 16 indexed citations
16.
Joh, Tong H., et al.. (1997). Unique and Cell-Type-Specific Tyrosine Hydroxylase Gene Expression. Advances in pharmacology. 42. 33–36. 10 indexed citations
17.
Son, Jin H., Nan Min, & Tong H. Joh. (1996). Early ontogeny of catecholaminergic cell lineage in brain and peripheral neurons monitored by tyrosine hydroxylase-lacZ transgene. Molecular Brain Research. 36(2). 300–308. 27 indexed citations
18.
Son, Jin H., et al.. (1996). Immortalization of neuroendocrine pinealocytes from transgenic mice by targeted tumorigenesis using the tryptophan hydroxylase promoter. Molecular Brain Research. 37(1-2). 32–40. 19 indexed citations
19.
Son, Jin H., et al.. (1994). Drastic and selective hyperinnervation of central serotonergic neurons in a lethal neurodevelopmental mouse mutant, Anorexia (anx). Molecular Brain Research. 25(1-2). 129–134. 35 indexed citations
20.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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