Benjamin J. Wilkins

5.6k total citations · 1 hit paper
66 papers, 4.2k citations indexed

About

Benjamin J. Wilkins is a scholar working on Molecular Biology, Surgery and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, Benjamin J. Wilkins has authored 66 papers receiving a total of 4.2k indexed citations (citations by other indexed papers that have themselves been cited), including 27 papers in Molecular Biology, 24 papers in Surgery and 11 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Benjamin J. Wilkins's work include Signaling Pathways in Disease (13 papers), Eosinophilic Esophagitis (12 papers) and Cardiac Fibrosis and Remodeling (8 papers). Benjamin J. Wilkins is often cited by papers focused on Signaling Pathways in Disease (13 papers), Eosinophilic Esophagitis (12 papers) and Cardiac Fibrosis and Remodeling (8 papers). Benjamin J. Wilkins collaborates with scholars based in United States, Canada and New Zealand. Benjamin J. Wilkins's co-authors include Jeffery D. Molkentin, Orlando F. Bueno, Stephanie A. Parsons, Yan‐Shan Dai, Betty J. Glascock, Thomas F. Kimball, Thomas R. Kimball, Julian C. Braz, David M. Plank and Jian Xu and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Circulation.

In The Last Decade

Benjamin J. Wilkins

63 papers receiving 4.2k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Calcineurin/NFAT Coupling Participates in Pathological, but not Physiological, Cardiac Hypertrophy

2003 605 citations Benjamin J. Wilkins, Yan‐Shan Dai et al. profile →

Peers

Countries citing papers authored by Benjamin J. Wilkins

Since Specialization

Citations

This map shows the geographic impact of Benjamin J. Wilkins's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Benjamin J. Wilkins with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Benjamin J. Wilkins more than expected).

Fields of papers citing papers by Benjamin J. Wilkins

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Benjamin J. Wilkins. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Benjamin J. Wilkins. The network helps show where Benjamin J. Wilkins may publish in the future.

Co-authorship network of co-authors of Benjamin J. Wilkins

This figure shows the co-authorship network connecting the top 25 collaborators of Benjamin J. Wilkins. A scholar is included among the top collaborators of Benjamin J. Wilkins based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Benjamin J. Wilkins. Benjamin J. Wilkins is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Title	Journal	Authors	Indexed citations
1	Oral N-acetylcysteine ameliorates liver fibrosis and enhances regenerative responses in Mdr2 knockout mice	Scientific Reports	Adi Har‐Zahav, Ana Tobar et al.	3
2	Granulomatous hyperinflammatory state induced by dupilumab treatment for eosinophilic esophagitis	SHILAP Revista de lepidopterología	Melissa A. Lerman, Jon M. Burnham et al.	0
3	Intestinal transit-amplifying cells require METTL3 for growth factor signaling and cell survival	JCI Insight	Charles Danan, Katharina E. Hayer et al.	4
4	No-Implant Interatrial Shunt for HFpEF	JACC Heart Failure	James E. Udelson, Colin M. Barker et al.	14
5	Novel Development of Magnetic Resonance Imaging to Quantify the Structural Anatomic Growth of Diverse Organs in Adult and Mutant Zebrafish	Zebrafish	Sonal Sharma, Sergey Magnitsky et al.	1
6	Loss of zebrafish pkd1l1 causes biliary defects that have implications for biliary atresia splenic malformation	Disease Models & Mechanisms	Benjamin J. Wilkins, Saul J. Karpen et al.	4
7	E-42 \| Multi-Center Clinical Outcomes of a No-Implant Interatrial Shunt for Heart Failure With Preserved and Reduced Ejection Fraction: Six-Month Outcomes from the ALLEVIATE-HF Feasibility Program	Journal of the Society for Cardiovascular Angiography & Interventions	Molly Szerlip, Gerard Wilkins et al.	1
8	Esophageal remodeling in eosinophilic esophagitis: Relationships to luminal captured biomarkers of inflammation and periostin	Journal of Allergy and Clinical Immunology	Amanda B. Muir, Steven J. Ackerman et al.	25
9	Highly Multiplexed Image Analysis of Intestinal Tissue Sections in Patients With Inflammatory Bowel Disease	Gastroenterology	Ayano Kondo, Michelle Y. Y. Lee et al.	30
10	Robust, 3-Dimensional Visualization of Human Colon Enteric Nervous System Without Tissue Sectioning	Gastroenterology	Rajarshi Sengupta, Archana Shenoy et al.	48
11	Persistent Basal Cell Hyperplasia Is Associated With Clinical and Endoscopic Findings in Patients With Histologically Inactive Eosinophilic Esophagitis	Clinical Gastroenterology and Hepatology	Kelly A. Whelan, Benjamin J. Wilkins et al.	49
12	Efficacy of Epicutaneous Immunotherapy in Children With Milk-Induced Eosinophilic Esophagitis	Clinical Gastroenterology and Hepatology	Jonathan M. Spergel, Okan U. Elci et al.	36
13	Posttranscriptional regulation of colonic epithelial repair by RNA binding protein IMP 1/ IGF 2 BP 1	EMBO Reports	Priya Chatterji, Patrick A. Williams et al.	23
14	Preferential Secretion of Thymic Stromal Lymphopoietin (TSLP) by Terminally Differentiated Esophageal Epithelial Cells: Relevance to Eosinophilic Esophagitis (EoE)	PLoS ONE	Prasanna M. Chandramouleeswaran, Anna J. Lee et al.	36
15	A novel keratin18 promoter that drives reporter gene expression in the intrahepatic and extrahepatic biliary system allows isolation of cell-type specific transcripts from zebrafish liver	Gene Expression Patterns	Benjamin J. Wilkins, Weilong Gong et al.	24
16	Zebrafish Models of Human Liver Development and Disease	Comprehensive physiology	Benjamin J. Wilkins, Michael Pack	75
17	Zebrafish Models of Human Liver Development and Disease	Comprehensive physiology	Benjamin J. Wilkins, Michael Pack	3
18	Non-Functional Parathyroid Carcinoma: A Review of the Literature and Report of a Case Requiring Extensive Surgery	Head and Neck Pathology	Benjamin J. Wilkins, James S. Lewis	64
19	Genetic inhibition of cardiac ERK1/2 promotes stress-induced apoptosis and heart failure but has no effect on hypertrophy in vivo	Proceedings of the National Academy of Sciences	Nicole H. Purcell, Benjamin J. Wilkins et al.	197
20	Targeted inhibition of p38 MAPK promotes hypertrophic cardiomyopathy through upregulation of calcineurin-NFAT signaling	Journal of Clinical Investigation	Julian C. Braz, Orlando F. Bueno et al.	262

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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