Marc K. Saba-El-Leil

3.2k total citations · 1 hit paper
28 papers, 2.4k citations indexed

About

Marc K. Saba-El-Leil is a scholar working on Molecular Biology, Oncology and Pathology and Forensic Medicine. According to data from OpenAlex, Marc K. Saba-El-Leil has authored 28 papers receiving a total of 2.4k indexed citations (citations by other indexed papers that have themselves been cited), including 22 papers in Molecular Biology, 8 papers in Oncology and 4 papers in Pathology and Forensic Medicine. Recurrent topics in Marc K. Saba-El-Leil's work include Melanoma and MAPK Pathways (9 papers), Pluripotent Stem Cells Research (4 papers) and Protein Kinase Regulation and GTPase Signaling (3 papers). Marc K. Saba-El-Leil is often cited by papers focused on Melanoma and MAPK Pathways (9 papers), Pluripotent Stem Cells Research (4 papers) and Protein Kinase Regulation and GTPase Signaling (3 papers). Marc K. Saba-El-Leil collaborates with scholars based in Canada, United States and France. Marc K. Saba-El-Leil's co-authors include Sylvain Meloche, Jason Wray, Tilo Kunath, Austin Smith, Laure Voisin, Jeffery D. Molkentin, Siew‐Lan Ang, Christophe Frémin, Allen J. York and Benjamin J. Wilkins and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Circulation and Nature Communications.

In The Last Decade

Marc K. Saba-El-Leil

27 papers receiving 2.4k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

FGF stimulation of the Erk1/2 signalling cascade triggers transition of pluripotent embryonic stem cells from self-renewal to lineage commitment

2007 606 citations Tilo Kunath, Marc K. Saba-El-Leil et al. Development profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Marc K. Saba-El-Leil Canada	19	1.9k	344	307	225	209	28	2.4k
Mimi Tamamori‐Adachi Japan	21	1.0k 0.6×	306 0.9×	174 0.6×	192 0.9×	171 0.8×	41	1.5k
Günter Daum United States	27	1.7k 0.9×	321 0.9×	215 0.7×	250 1.1×	341 1.6×	55	2.5k
Xiaojun Zhu China	25	1.9k 1.0×	204 0.6×	258 0.8×	292 1.3×	242 1.2×	58	2.5k
Gabriele D’Uva Italy	19	1.0k 0.6×	355 1.0×	351 1.1×	278 1.2×	120 0.6×	31	1.8k
Xuan Cheng China	26	1.5k 0.8×	365 1.1×	189 0.6×	531 2.4×	176 0.8×	89	2.6k
Domenica Mangieri Italy	29	1.1k 0.6×	363 1.1×	126 0.4×	212 0.9×	168 0.8×	51	2.0k
Brad A. Bryan United States	27	1.2k 0.6×	487 1.4×	154 0.5×	291 1.3×	315 1.5×	63	2.1k
Juan A. Bernal Spain	19	1.2k 0.6×	259 0.8×	203 0.7×	223 1.0×	171 0.8×	38	1.7k
Mara Brancaccio Italy	28	1.7k 0.9×	265 0.8×	602 2.0×	210 0.9×	548 2.6×	71	2.7k
Oliver Renner Spain	19	1.8k 1.0×	429 1.2×	120 0.4×	478 2.1×	263 1.3×	26	3.0k

Countries citing papers authored by Marc K. Saba-El-Leil

Since Specialization

Citations

This map shows the geographic impact of Marc K. Saba-El-Leil's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Marc K. Saba-El-Leil with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Marc K. Saba-El-Leil more than expected).

Fields of papers citing papers by Marc K. Saba-El-Leil

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Marc K. Saba-El-Leil. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Marc K. Saba-El-Leil. The network helps show where Marc K. Saba-El-Leil may publish in the future.

Co-authorship network of co-authors of Marc K. Saba-El-Leil

This figure shows the co-authorship network connecting the top 25 collaborators of Marc K. Saba-El-Leil. A scholar is included among the top collaborators of Marc K. Saba-El-Leil based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Marc K. Saba-El-Leil. Marc K. Saba-El-Leil is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Voisin, Laure, et al.. (2024). Syngeneic mouse model of YES-driven metastatic and proliferative hepatocellular carcinoma. Disease Models & Mechanisms. 17(7). 2 indexed citations

Goupil, Eugénie, et al.. (2024). OSGN-1 is a conserved flavin-containing monooxygenase required to stabilize the intercellular bridge in late cytokinesis. Proceedings of the National Academy of Sciences. 121(11). e2308570121–e2308570121. 3 indexed citations

Houlès, Thibault, Geneviève Lavoie, Sami Nourreddine, et al.. (2022). CDK12 is hyperactivated and a synthetic-lethal target in BRAF-mutated melanoma. Nature Communications. 13(1). 6457–6457. 23 indexed citations

Langlois, Marie‐Josée, Sarah Pasquin, Marc K. Saba-El-Leil, et al.. (2020). Loss of interleukin-17 receptor D promotes chronic inflammation-associated tumorigenesis. Oncogene. 40(2). 452–464. 23 indexed citations

Lebraud, Honorine, Rebecca Gilley, Andrew M. Kidger, et al.. (2017). Visualization of Endogenous ERK1/2 in Cells with a Bioorthogonal Covalent Probe. Bioconjugate Chemistry. 28(6). 1677–1683. 8 indexed citations

Pyakurel, Aswin, Marc K. Saba-El-Leil, Caroline Kizilyaprak, et al.. (2017). Loss of Extracellular Signal-Regulated Kinase 1/2 in the Retinal Pigment Epithelium Leads to RPE65 Decrease and Retinal Degeneration. Molecular and Cellular Biology. 37(24). 12 indexed citations

Saba-El-Leil, Marc K., Christophe Frémin, & Sylvain Meloche. (2016). Isolation of Mouse Embryonic Stem Cell Lines in the Study of ERK1/2 MAP Kinase Signaling. Methods in molecular biology. 1487. 243–253. 1 indexed citations

Saba-El-Leil, Marc K., Christophe Frémin, & Sylvain Meloche. (2016). Redundancy in the World of MAP Kinases: All for One. Frontiers in Cell and Developmental Biology. 4. 67–67. 50 indexed citations

Frémin, Christophe, et al.. (2015). Functional Redundancy of ERK1 and ERK2 MAP Kinases during Development. Cell Reports. 12(6). 913–921. 78 indexed citations

10.

Deschênes‐Simard, Xavier, Marie‐France Gaumont‐Leclerc, Véronique Bourdeau, et al.. (2013). Tumor suppressor activity of the ERK/MAPK pathway by promoting selective protein degradation. Genes & Development. 27(8). 900–915. 147 indexed citations

11.

Wang, Jinyong, Guangyao Kong, Yangang Liu, et al.. (2013). NrasG12D/+ promotes leukemogenesis by aberrantly regulating hematopoietic stem cell functions. Blood. 121(26). 5203–5207. 39 indexed citations

12.

Roméo, Yves, J. Moreau, Marc K. Saba-El-Leil, et al.. (2012). RSK regulates activated BRAF signalling to mTORC1 and promotes melanoma growth. Oncogene. 32(24). 2917–2926. 53 indexed citations

13.

Kehat, Izhak, Jennifer Davis, Malte Tiburcy, et al.. (2010). Extracellular Signal-Regulated Kinases 1 and 2 Regulate the Balance Between Eccentric and Concentric Cardiac Growth. Circulation Research. 108(2). 176–183. 197 indexed citations

14.

Voisin, Laure, Catherine Julien, Stéphanie Duhamel, et al.. (2008). Activation of MEK1 or MEK2 isoform is sufficient to fully transform intestinal epithelial cells and induce the formation of metastatic tumors. BMC Cancer. 8(1). 337–337. 58 indexed citations

15.

Purcell, Nicole H., Benjamin J. Wilkins, Allen J. York, et al.. (2007). Genetic inhibition of cardiac ERK1/2 promotes stress-induced apoptosis and heart failure but has no effect on hypertrophy in vivo. Proceedings of the National Academy of Sciences. 104(35). 14074–14079. 197 indexed citations

16.

Kunath, Tilo, et al.. (2007). FGF stimulation of the Erk1/2 signalling cascade triggers transition of pluripotent embryonic stem cells from self-renewal to lineage commitment. Development. 134(16). 2895–2902. 606 indexed citations breakdown →

17.

Saba-El-Leil, Marc K., et al.. (2005). EN2 is a candidate oncogene in human breast cancer. Oncogene. 24(46). 6890–6901. 45 indexed citations

18.

Meloche, Sylvain, et al.. (2004). Erk2 Signaling and Early Embryo Stem Cell Self-Renewal. Cell Cycle. 3(3). 239–241. 20 indexed citations

19.

Lips, Daniel J., Orlando F. Bueno, Benjamin J. Wilkins, et al.. (2004). MEK1-ERK2 Signaling Pathway Protects Myocardium From Ischemic Injury In Vivo. Circulation. 109(16). 1938–1941. 193 indexed citations

20.

Saba-El-Leil, Marc K., Bertrand Vernay, Laure Voisin, et al.. (2003). An essential function of the mitogen‐activated protein kinase Erk2 in mouse trophoblast development. EMBO Reports. 4(10). 964–968. 309 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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