Jonathan Schug

12.4k citations
100 papers · 8.5k indexed · 1 hit paper · h-index 48

Jonathan Schug

100 papers receiving 8.4k citations

Hit Papers

PPARγ and C/EBP factors orchestrate adipocyte biology via...6742008202620142020200400600

Peers

Jonathan Schug
Comparison fields: 5 of 154
  • Molecular Biology 5.3k
  • Cancer Research 1.2k
  • Genetics 2.1k
  • Endocrinology, Diabetes and Metabolism 1.0k
  • Surgery 2.6k
Replace Jianming Xu with:
Jianming Xu United States
Yusuke Nakamura Japan
Xiaoyong Yang United States
Paolo Meda Switzerland
Mogens Kruhøffer Denmark
Katriina Aalto‐Setälä Finland
Johan Björkegren Sweden
Kazuyoshi Yonezawa Japan
Glynn Dennis United States
Douglas A Hosack United States
Jonathan Schug relative to Jianming Xu United States Jianming Xu's profile →
Citations per field
00.5×3.6×
Jianming Xu · 1×
Citations per year

Countries citing papers authored by Jonathan Schug

Since Specialization
Citations

This map shows the geographic impact of Jonathan Schug's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jonathan Schug with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jonathan Schug more than expected).

Fields of papers citing papers by Jonathan Schug

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jonathan Schug. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jonathan Schug. The network helps show where Jonathan Schug may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Jonathan Schug, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Jonathan Schug Line = papers co-authored together Jonathan Schug links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown
#Work
1 20243
2 202312
3 20228
4 202139
5 202130
6 20215
7 202058
8 201911
9 201840
10 201728
11 2016190
12 2014133
13 201361
14 201337
15 2013181
16 201372
17 2012157
18 20052
19 2005192
20 199836

About Jonathan Schug

Jonathan Schug is a scholar working on Genetics, Molecular Biology and Hepatology, having authored 100 papers that have together received 8.5k indexed citations. Recurring topics across this work include Pancreatic function and diabetes (30 papers), Epigenetics and DNA Methylation (17 papers), Diabetes and associated disorders (13 papers), Genomics and Chromatin Dynamics (13 papers), Genetics and Neurodevelopmental Disorders (12 papers), RNA modifications and cancer (11 papers), Genomics and Phylogenetic Studies (9 papers) and Cancer-related gene regulation (7 papers). The work is most often cited by research in Molecular Biology (5.3k citations), Cancer Research (1.2k citations) and Genetics (2.1k citations). Jonathan Schug has collaborated with scholars based in United States, Israel and France. Frequent co-authors include Klaus H. Kaestner, Christian J. Stoeckert, Ali Naji, Geetu Tuteja, Chengyang Liu, Mitchell A. Lazar, David J. Steger, Markus Grompe, Zhaoyu Li and Craig Dorrell. Their work appears in journals such as Genes & Development, Diabetes, Proceedings of the National Academy of Sciences, Cell Metabolism and Scientific Reports.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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