Bénédicte Lemmers
- Oncology top 5%
- Cancer-related Molecular Pathways 10
- Molecular Biology top 5%
- Cell death mechanisms and regulation 8
- DNA Repair Mechanisms 4
- Ubiquitin and proteasome pathways 4
- Hedgehog Signaling Pathway Studies 3
- Cancer Research top 5%
- Cell Biology top 5%
- Microtubule and mitosis dynamics 8
- Immunology top 5%
- Immunotherapy and Immune Responses 3
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- Monoclonal and Polyclonal Antibodies Research 4
- Co-authors
- Razqallah HakemNawal BendrisJean Marie BlanchardElzbieta Matysiak‐ZablockiEva MigonAnne HakemRoger LengYunping Lin
- Partner nations
- FranceUnited StatesCanada
In The Last Decade
Bénédicte Lemmers
30 papers receiving 2.4k citations
Hit Papers
Peers
Comparison fields: 5 of 101
- Oncology 824
- Molecular Biology 1.8k
- Cancer Research 384
- Cell Biology 392
- Immunology 493
Countries citing papers authored by Bénédicte Lemmers
This map shows the geographic impact of Bénédicte Lemmers's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Bénédicte Lemmers with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Bénédicte Lemmers more than expected).
Fields of papers citing papers by Bénédicte Lemmers
This network shows the impact of papers produced by Bénédicte Lemmers. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Bénédicte Lemmers. The network helps show where Bénédicte Lemmers may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Bénédicte Lemmers, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2021 | 13 | |
| 2 | 2015 | 25 | |
| 3 | 2014 | 2 | |
| 4 | 2014 | 86 | |
| 5 | 2014 | 22 | |
| 6 | 2012 | 23 | |
| 7 | 2012 | 53 | |
| 8 | 2011 | 54 | |
| 9 | 2007 | 147 | |
| 10 | 2006 | 59 | |
| 11 | 2005 | 72 | |
| 12 | 2004 | 168 | |
| 13 | 2004 | 54 | |
| 14 | 2004 | 159 | |
| 15 | Pirh2, a p53-Induced Ubiquitin-Protein Ligase, Promotes p53 Degradationbreakdown → | 2003 | 584 |
| 16 | 2003 | 8 | |
| 17 | 2003 | 396 | |
| 18 | 2000 | 13 | |
| 19 | 2000 | 12 | |
| 20 | 1999 | 20 |
About Bénédicte Lemmers
Bénédicte Lemmers is a scholar working on Aging, Cell Biology and Immunology, having authored 30 papers that have together received 2.4k indexed citations. Recurring topics across this work include Cancer-related Molecular Pathways (10 papers), Microtubule and mitosis dynamics (8 papers), Cell death mechanisms and regulation (8 papers), DNA Repair Mechanisms (4 papers), Ubiquitin and proteasome pathways (4 papers), Monoclonal and Polyclonal Antibodies Research (4 papers), Hedgehog Signaling Pathway Studies (3 papers) and Immunotherapy and Immune Responses (3 papers). The work is most often cited by research in Oncology (824 citations), Molecular Biology (1.8k citations) and Cancer Research (384 citations). Bénédicte Lemmers has collaborated with scholars based in France, United States and Canada. Frequent co-authors include Razqallah Hakem, Nawal Bendris, Jean Marie Blanchard, Elzbieta Matysiak‐Zablocki, Eva Migon, Anne Hakem, Roger Leng, Yunping Lin, John M. Parant and Stephen W. Chung. Their work appears in journals such as Science, Cell and Journal of Biological Chemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.