Yunping Lin

2.4k total citations · 2 hit papers
11 papers, 2.0k citations indexed

About

Yunping Lin is a scholar working on Molecular Biology, Oncology and Pathology and Forensic Medicine. According to data from OpenAlex, Yunping Lin has authored 11 papers receiving a total of 2.0k indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Molecular Biology, 9 papers in Oncology and 2 papers in Pathology and Forensic Medicine. Recurrent topics in Yunping Lin's work include Cancer-related Molecular Pathways (9 papers), Cell death mechanisms and regulation (3 papers) and RNA modifications and cancer (2 papers). Yunping Lin is often cited by papers focused on Cancer-related Molecular Pathways (9 papers), Cell death mechanisms and regulation (3 papers) and RNA modifications and cancer (2 papers). Yunping Lin collaborates with scholars based in Canada and United States. Yunping Lin's co-authors include Samuel Benchimol, Weili Ma, David MacPherson, Tak W. Mak, Brian J. Snow, Vuk Stambolic, Roger Leng, Bénédicte Lemmers, Hong Wu and John M. Parant and has published in prestigious journals such as Cell, Nature Genetics and Blood.

In The Last Decade

Yunping Lin

11 papers receiving 2.0k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Regulation of PTEN Transcription by p53

2001 753 citations Vuk Stambolic, David MacPherson et al. Molecular Cell profile →
Pirh2, a p53-Induced Ubiquitin-Protein Ligase, Promotes p53 Degradation

2003 584 citations Roger Leng, Yunping Lin et al. Cell profile →

Peers

Countries citing papers authored by Yunping Lin

Since Specialization

Citations

This map shows the geographic impact of Yunping Lin's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Yunping Lin with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Yunping Lin more than expected).

Fields of papers citing papers by Yunping Lin

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Yunping Lin. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Yunping Lin. The network helps show where Yunping Lin may publish in the future.

Co-authorship network of co-authors of Yunping Lin

This figure shows the co-authorship network connecting the top 25 collaborators of Yunping Lin. A scholar is included among the top collaborators of Yunping Lin based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Yunping Lin. Yunping Lin is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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11 of 11 papers shown

#	Work	Indexed citations
1	Inhibition of p53 expression by peptide-conjugated phosphorodiamidate morpholino oligomers sensitizes human cancer cells to chemotherapeutic drugs 2011 ·Oncogene ·Weili Ma, Yunping Lin, Wanli Xuan, Patrick L. Iversen, Lachlan J. Smith, Samuel Benchimol	12
2	ROS-Mediated p53 Induction of Lpin1 Regulates Fatty Acid Oxidation in Response to Nutritional Stress 2011 ·Molecular Cell ·(unknown), (unknown), Keith Wheaton, Yunping Lin, Weili Ma, Wanli Xuan, (unknown), Katsuya Tsuchihara, Tak W. Mak, Samuel Benchimol	158
3	Identification of p53-Regulated Genes by the Method of Differential Display 2005 ·Humana Press eBooks ·Yunping Lin, Roger Leng, Samuel Benchimol	1
4	Pirh2, a p53-Induced Ubiquitin-Protein Ligase, Promotes p53 Degradation breakdown → 2003 ·Cell ·Roger Leng, Yunping Lin, Weili Ma, Hong Wu, Bénédicte Lemmers, Stephen W. Chung, John M. Parant, Guillermina Lozano, Razqallah Hakem, Samuel Benchimol	584
5	The death-promoting activity of p53 can be inhibited by distinct signaling pathways 2002 ·Blood ·Yunping Lin, (unknown), David W. Hedley, Dwayne L. Barber, Samuel Benchimol	16
6	Regulation of PTEN Transcription by p53 breakdown → 2001 ·Molecular Cell ·Vuk Stambolic, David MacPherson, (unknown), Yunping Lin, Brian J. Snow, (unknown), Samuel Benchimol, Tak W. Mak	753
7	Structure and functionality of a designed p53 dimer11Edited by P. E. Wright 2001 ·Journal of Molecular Biology ·Timothy S. Davison, (unknown), Weili Ma, Yunping Lin, C.M. Kay, Samuel Benchimol, C.H. Arrowsmith	66
8	Pidd, a new death-domain–containing protein, is induced by p53 and promotes apoptosis 2000 ·Nature Genetics ·Yunping Lin, Weili Ma, Samuel Benchimol	274
9	A Role for Perforin in Activation-Induced Cell Death 1998 ·The Journal of Immunology ·David Spaner, Kaliannan Raju, Laszlo Radvanyi, Yunping Lin, Richard G. Miller	53
10	p53-mediated cell cycle arrest and apoptosis. 1997 ·PubMed ·Yunping Lin, Samuel Benchimol	9
11	Cytokines Inhibit p53-Mediated Apoptosis but Not p53-Mediated G₁ Arrest 1995 ·Molecular and Cellular Biology ·Yunping Lin, Samuel Benchimol	67

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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