Antonio Sanz-Clemente
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- Neuroscience and Neuropharmacology Research 14
- Physiology top 2%
- Alzheimer's disease research and treatments 3
- Biological Psychiatry top 5%
- Developmental Neuroscience top 5%
- Neurology top 5%
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- Ion channel regulation and function 5
- Retinal Development and Disorders 3
- Mitochondrial Function and Pathology 3
- Receptor Mechanisms and Signaling 3
- ATP Synthase and ATPases Research 2
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- Genetics and Neurodevelopmental Disorders 3
Antonio Sanz-Clemente
17 papers receiving 2.1k citations
Hit Papers
Peers
Comparison fields: 5 of 92
- Cellular and Molecular Neuroscience 1.4k
- Physiology 1.0k
- Biological Psychiatry 91
- Developmental Neuroscience 150
- Neurology 297
Countries citing papers authored by Antonio Sanz-Clemente
This map shows the geographic impact of Antonio Sanz-Clemente's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Antonio Sanz-Clemente with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Antonio Sanz-Clemente more than expected).
Fields of papers citing papers by Antonio Sanz-Clemente
This network shows the impact of papers produced by Antonio Sanz-Clemente. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Antonio Sanz-Clemente. The network helps show where Antonio Sanz-Clemente may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Antonio Sanz-Clemente, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2025 | 1 | |
| 2 | 2019 | 4 | |
| 3 | 2019 | 28 | |
| 4 | 2019 | 1 | |
| 5 | 2017 | 47 | |
| 6 | 2017 | 63 | |
| 7 | 2015 | 126 | |
| 8 | 2013 | 84 | |
| 9 | 2012 | 65 | |
| 10 | 2012 | 311 | |
| 11 | 2011 | 46 | |
| 12 | 2011 | 154 | |
| 13 | 2010 | 164 | |
| 14 | 2008 | 16 | |
| 15 | Aβ Oligomer-Induced Aberrations in Synapse Composition, Shape, and Density Provide a Molecular Basis for Loss of Connectivity in Alzheimer's Diseasebreakdown → | 2007 | 994 |
| 16 | 2007 | 26 | |
| 17 | 2005 | 29 |
About Antonio Sanz-Clemente
Antonio Sanz-Clemente is a scholar working on Cellular and Molecular Neuroscience, Neurology and Molecular Biology, having authored 17 papers that have together received 2.2k indexed citations. Recurring topics across this work include Neuroscience and Neuropharmacology Research (14 papers), Ion channel regulation and function (5 papers), Retinal Development and Disorders (3 papers), Mitochondrial Function and Pathology (3 papers), Genetics and Neurodevelopmental Disorders (3 papers), Alzheimer's disease research and treatments (3 papers), Receptor Mechanisms and Signaling (3 papers) and ATP Synthase and ATPases Research (2 papers). The work is most often cited by research in Cellular and Molecular Neuroscience (1.4k citations), Physiology (1.0k citations) and Biological Psychiatry (91 citations). Antonio Sanz-Clemente has collaborated with scholars based in United States, Czechia and Spain. Frequent co-authors include Katherine W. Roche, Roger A. Nicoll, Maria C. Buniel, Margaret Wood, Kirsten L. Viola, Pauline T. Velasco, William L. Klein, Paul W. Furlow, Pascale N. Lacor and José A. Matta. Their work appears in journals such as Journal of Biological Chemistry, Neuron and Journal of Neuroscience.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.