Jannic Boehm

2.5k total citations · 1 hit paper
15 papers, 2.0k citations indexed

About

Jannic Boehm is a scholar working on Cellular and Molecular Neuroscience, Physiology and Molecular Biology. According to data from OpenAlex, Jannic Boehm has authored 15 papers receiving a total of 2.0k indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Cellular and Molecular Neuroscience, 8 papers in Physiology and 6 papers in Molecular Biology. Recurrent topics in Jannic Boehm's work include Neuroscience and Neuropharmacology Research (13 papers), Alzheimer's disease research and treatments (8 papers) and Cholinesterase and Neurodegenerative Diseases (3 papers). Jannic Boehm is often cited by papers focused on Neuroscience and Neuropharmacology Research (13 papers), Alzheimer's disease research and treatments (8 papers) and Cholinesterase and Neurodegenerative Diseases (3 papers). Jannic Boehm collaborates with scholars based in Canada, United States and Japan. Jannic Boehm's co-authors include Roberto Malinow, Helen Hsieh, Sangram S. Sisodia, Chihiro Sato, Takeshi Iwatsubo, Taisuke Tomita, Wei Wei, Charles D. Kopec, Bo Li and Myoung‐Goo Kang and has published in prestigious journals such as Journal of Biological Chemistry, Neuron and Journal of Neuroscience.

In The Last Decade

Jannic Boehm

15 papers receiving 2.0k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

AMPAR Removal Underlies Aβ-Induced Synaptic Depression and Dendritic Spine Loss

2006 865 citations Helen Hsieh, Jannic Boehm et al. Neuron profile →

Peers

Jannic Boehm

CMN

PHYSI

NEURO

Antonio Sanz-Clemente United States

Graham L. Collingridge United Kingdom

Li-Lian Yuan United States

Volker Nimmrich Germany

Yi Nong United States

Brian Halabisky United States

Kaiwen He China

Shan‐Xue Jin United States

Timothy D. Moran Canada

Robert C. Malenka United States

Antonio Sanz-Clemente United States

Jannic Boehm

1.4k ×0.9

CMN

1.0k ×1.0

PHYSI

947 ×1.2

313 ×0.9

297 ×1.0

NEURO

Citations per year, relative to Jannic Boehm Jannic Boehm (= 1×) peers Antonio Sanz-Clemente

Countries citing papers authored by Jannic Boehm

Since Specialization

Citations

This map shows the geographic impact of Jannic Boehm's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jannic Boehm with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jannic Boehm more than expected).

Fields of papers citing papers by Jannic Boehm

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jannic Boehm. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jannic Boehm. The network helps show where Jannic Boehm may publish in the future.

Co-authorship network of co-authors of Jannic Boehm

This figure shows the co-authorship network connecting the top 25 collaborators of Jannic Boehm. A scholar is included among the top collaborators of Jannic Boehm based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jannic Boehm. Jannic Boehm is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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15 of 15 papers shown

Trillaud-Doppia, Émilie, et al.. (2020). RCAN1 Regulates Bidirectional Synaptic Plasticity. Current Biology. 30(7). 1167–1176.e2. 12 indexed citations

Boehm, Jannic, et al.. (2018). NMDA receptor–dependent dephosphorylation of serine 387 in Argonaute 2 increases its degradation and affects dendritic spine density and maturation. Journal of Biological Chemistry. 293(24). 9311–9325. 11 indexed citations

Trillaud-Doppia, Émilie, et al.. (2016). A single amino acid difference between the intracellular domains of amyloid precursor protein and amyloid-like precursor protein 2 enables induction of synaptic depression and block of long-term potentiation. Neurobiology of Disease. 91. 94–104. 8 indexed citations

Trillaud-Doppia, Émilie & Jannic Boehm. (2016). The Amyloid Precursor Protein Intracellular Domain Is an Effector Molecule of Metaplasticity. Biological Psychiatry. 83(5). 406–415. 9 indexed citations

Bourgeois, Catherine, et al.. (2013). NMDA-Receptor Activation but Not Ion Flux Is Required for Amyloid-Beta Induced Synaptic Depression. PLoS ONE. 8(6). e65350–e65350. 57 indexed citations

Boehm, Jannic. (2013). A ‘danse macabre’: tau and Fyn in STEP with amyloid beta to facilitate induction of synaptic depression and excitotoxicity. European Journal of Neuroscience. 37(12). 1925–1930. 29 indexed citations

Mondragón‐Rodríguez, Siddhartha, Émilie Trillaud-Doppia, Catherine Bourgeois, et al.. (2012). Interaction of Endogenous Tau Protein with Synaptic Proteins Is Regulated by N-Methyl-d-aspartate Receptor-dependent Tau Phosphorylation. Journal of Biological Chemistry. 287(38). 32040–32053. 200 indexed citations

Mondragón‐Rodríguez, Siddhartha, George Perry, Xiongwei Zhu, & Jannic Boehm. (2012). Amyloid Beta and Tau Proteins as Therapeutic Targets for Alzheimer’s Disease Treatment: Rethinking the Current Strategy. International Journal of Alzheimer s Disease. 2012. 1–7. 51 indexed citations

Mondragón‐Rodríguez, Siddhartha, et al.. (2011). P4‐323: Tau phosphorylation links Amyloid beta and NMDA receptor activation, implications for Alzheimer's disease. Alzheimer s & Dementia. 7(4S_Part_24). 1 indexed citations

10.

Hsieh, Helen, Jannic Boehm, Chihiro Sato, et al.. (2006). AMPAR Removal Underlies Aβ-Induced Synaptic Depression and Dendritic Spine Loss. Neuron. 52(5). 831–843. 865 indexed citations breakdown →

11.

Kopec, Charles D., Bo Li, Wei Wei, Jannic Boehm, & Roberto Malinow. (2006). Glutamate Receptor Exocytosis and Spine Enlargement during Chemically Induced Long-Term Potentiation. Journal of Neuroscience. 26(7). 2000–2009. 377 indexed citations

12.

Boehm, Jannic, Myoung‐Goo Kang, Richard C. Johnson, et al.. (2006). Synaptic Incorporation of AMPA Receptors during LTP Is Controlled by a PKC Phosphorylation Site on GluR1. Neuron. 51(2). 213–225. 298 indexed citations

13.

Boehm, Jannic, Ingrid Ehrlich, Helen Hsieh, & Roberto Malinow. (2006). Two mutations preventing PDZ–protein interactions of GluR1 have opposite effects on synaptic plasticity. Learning & Memory. 13(5). 562–565. 18 indexed citations

14.

Boehm, Jannic & Roberto Malinow. (2005). AMPA receptor phosphorylation during synaptic plasticity. Biochemical Society Transactions. 33(6). 1354–1356. 36 indexed citations

15.

Malinow, Roberto & Jannic Boehm. (2005). AMPA receptor phosphorylation during synaptic plasticity. Biochemical Society Transactions. 33(6). 1354–1354. 55 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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