Annalisa Cavallini
- Neurology top 2%
- Neuroinflammation and Neurodegeneration Mechanisms 2
- Physiology top 5%
- Alzheimer's disease research and treatments 9
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- Neuroscience and Neuropharmacology Research 3
- Neurology top 10%
- Neuroinflammation and Neurodegeneration Mechanisms 2
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- Cholinesterase and Neurodegenerative Diseases 3
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- Prion Diseases and Protein Misfolding 2
- 14-3-3 protein interactions 2
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- Memory and Neural Mechanisms 2
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- Medicinal Plants and Neuroprotection 1
- Co-authors
- Suchira BoseMichael HuttonTracey K. MurrayMichel GoedertSamuel J. JacksonMichael J. O’NeillZeshan AhmedPhilip Szekeres
- Journals
- Journal of Biological Chemistry (4 papers)Journal of Neuroscience (1 paper)Scientific Reports (1 paper)
- Partner nations
- United KingdomUnited StatesSwitzerland
In The Last Decade
Annalisa Cavallini
11 papers receiving 898 citations
Peers
Comparison fields: 5 of 66
- Neurology 314
- Physiology 711
- Cellular and Molecular Neuroscience 268
- Biological Psychiatry 26
- Neurology 132
Countries citing papers authored by Annalisa Cavallini
This map shows the geographic impact of Annalisa Cavallini's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Annalisa Cavallini with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Annalisa Cavallini more than expected).
Fields of papers citing papers by Annalisa Cavallini
This network shows the impact of papers produced by Annalisa Cavallini. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Annalisa Cavallini. The network helps show where Annalisa Cavallini may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Annalisa Cavallini, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2024 | 3 | |
| 2 | 2021 | 3 | |
| 3 | 2020 | 21 | |
| 4 | 2019 | 42 | |
| 5 | 2018 | 62 | |
| 6 | 2017 | 3 | |
| 7 | 2016 | 110 | |
| 8 | 2014 | 364 | |
| 9 | 2014 | 199 | |
| 10 | 2013 | 95 | |
| 11 | 2012 | 1 | |
| 12 | 2010 | 0 |
About Annalisa Cavallini
Annalisa Cavallini is a scholar working on Neurology, Physiology and Cellular and Molecular Neuroscience, having authored 12 papers that have together received 903 indexed citations. Recurring topics across this work include Alzheimer's disease research and treatments (9 papers), Neuroscience and Neuropharmacology Research (3 papers), Cholinesterase and Neurodegenerative Diseases (3 papers), Prion Diseases and Protein Misfolding (2 papers), 14-3-3 protein interactions (2 papers), Memory and Neural Mechanisms (2 papers), Neuroinflammation and Neurodegeneration Mechanisms (2 papers) and Medicinal Plants and Neuroprotection (1 paper). The work is most often cited by research in Neurology (314 citations), Physiology (711 citations) and Cellular and Molecular Neuroscience (268 citations). Annalisa Cavallini has collaborated with scholars based in United Kingdom, United States and Switzerland. Frequent co-authors include Suchira Bose, Michael Hutton, Tracey K. Murray, Michel Goedert, Samuel J. Jackson, Michael J. O’Neill, Zeshan Ahmed, Philip Szekeres, Jane Cooper and Benjamin Falcon. Their work appears in journals such as Journal of Biological Chemistry, Journal of Neuroscience and Scientific Reports.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.