Yuichiro Mita

1.7k total citations · 1 hit paper
24 papers, 1.2k citations indexed

About

Yuichiro Mita is a scholar working on Nutrition and Dietetics, Molecular Biology and Neurology. According to data from OpenAlex, Yuichiro Mita has authored 24 papers receiving a total of 1.2k indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Nutrition and Dietetics, 8 papers in Molecular Biology and 5 papers in Neurology. Recurrent topics in Yuichiro Mita's work include Selenium in Biological Systems (5 papers), Parkinson's Disease Mechanisms and Treatments (5 papers) and Autophagy in Disease and Therapy (5 papers). Yuichiro Mita is often cited by papers focused on Selenium in Biological Systems (5 papers), Parkinson's Disease Mechanisms and Treatments (5 papers) and Autophagy in Disease and Therapy (5 papers). Yuichiro Mita collaborates with scholars based in Japan, United States and France. Yuichiro Mita's co-authors include Satoaki Matoba, Eri Iwai‐Kanai, Yoshifumi Okawa, Atsushi Hoshino, Koji Ikeda, Makoto Ariyoshi, Tomomi Ueyama, Takehiro Ogata, Noriko Noguchi and Yoshiro Saito and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nucleic Acids Research and Journal of Biological Chemistry.

In The Last Decade

Yuichiro Mita

24 papers receiving 1.2k citations

Hit Papers

Cytosolic p53 inhibits Parkin-mediated mitophagy and prom... 2013 2026 2017 2021 2013 100 200 300 400

Peers

Yuichiro Mita
Comparison fields: 5 of 94
  • Molecular Biology 563
  • Epidemiology 390
  • Physiology 215
  • Nutrition and Dietetics 193
  • Cardiology and Cardiovascular Medicine 152
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Citations per field, relative to Yuichiro Mita
Yuichiro Mita · 1×
Citations per year, relative to Yuichiro Mita
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Countries citing papers authored by Yuichiro Mita

Since Specialization
Citations

This map shows the geographic impact of Yuichiro Mita's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Yuichiro Mita with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Yuichiro Mita more than expected).

Fields of papers citing papers by Yuichiro Mita

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Yuichiro Mita. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Yuichiro Mita. The network helps show where Yuichiro Mita may publish in the future.

Co-authorship network of co-authors of Yuichiro Mita

This figure shows the co-authorship network connecting the top 25 collaborators of Yuichiro Mita. A scholar is included among the top collaborators of Yuichiro Mita based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Yuichiro Mita. Yuichiro Mita is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
# Work Indexed citations
1 1
2 2
3 1
4 2
5 12
6 15
7 20
8 13
9 14
10 34
11 138
12 33
13
Genetic polymorphism at codon 546 of the human RAD17 contributes to the risk for esophageal squamous cell carcinoma.
2
14 30
15 38
16
Cytosolic p53 inhibits Parkin-mediated mitophagy and promotes mitochondrial dysfunction in the mouse heart breakdown →
443
17 139
18 61
19 35
20
[A case of successful management of nonresectable pancreas cancer with liver metastasis by intra-arterial infusion chemotherapy with angiotensin-II and administration of tegafur/uracil].
2

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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