Yuki Ashida

551 total citations
31 papers, 414 citations indexed

About

Yuki Ashida is a scholar working on Molecular Biology, Physiology and Rehabilitation. According to data from OpenAlex, Yuki Ashida has authored 31 papers receiving a total of 414 indexed citations (citations by other indexed papers that have themselves been cited), including 19 papers in Molecular Biology, 9 papers in Physiology and 8 papers in Rehabilitation. Recurrent topics in Yuki Ashida's work include Muscle Physiology and Disorders (14 papers), Exercise and Physiological Responses (8 papers) and Muscle activation and electromyography studies (4 papers). Yuki Ashida is often cited by papers focused on Muscle Physiology and Disorders (14 papers), Exercise and Physiological Responses (8 papers) and Muscle activation and electromyography studies (4 papers). Yuki Ashida collaborates with scholars based in Japan, United States and Sweden. Yuki Ashida's co-authors include Takashi Yamada, Tetsuya Tamatani, Katsumi Motegi, Mitsunobu Sato, Masayuki Azuma, Masayuki Azuma, Girish Barot, Michael R. Roner, Ryotaro Yamada and Charles E. Carraher and has published in prestigious journals such as Proceedings of the National Academy of Sciences, PLoS ONE and The Journal of Physiology.

In The Last Decade

Yuki Ashida

29 papers receiving 409 citations

Peers

Yuki Ashida

PHYSI

ONCOL

REHAB

Bárbara Maximino Rezende Brazil

Toshimasa Jindo Japan

Changyue Wu China

Chiara Fania Italy

Ching‐Wan Lam Hong Kong

Andrew Powlson United Kingdom

Mohammed Al‐Hassani United States

Kevin E. McElhanon United States

Yuehua Li China

Bárbara Maximino Rezende Brazil

Yuki Ashida

188 ×0.9

77 ×0.7

PHYSI

55 ×0.8

ONCOL

44 ×0.7

REHAB

31 ×0.6

Citations per year, relative to Yuki Ashida Yuki Ashida (= 1×) peers Bárbara Maximino Rezende

Countries citing papers authored by Yuki Ashida

Since Specialization

Citations

This map shows the geographic impact of Yuki Ashida's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Yuki Ashida with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Yuki Ashida more than expected).

Fields of papers citing papers by Yuki Ashida

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Yuki Ashida. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Yuki Ashida. The network helps show where Yuki Ashida may publish in the future.

Co-authorship network of co-authors of Yuki Ashida

This figure shows the co-authorship network connecting the top 25 collaborators of Yuki Ashida. A scholar is included among the top collaborators of Yuki Ashida based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Yuki Ashida. Yuki Ashida is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Urano, Yasuomi, et al.. (2025). Unconventional secretion of PARK7 requires lysosomal delivery via chaperone-mediated autophagy and specialized SNARE complex. Proceedings of the National Academy of Sciences. 122(19). e2414790122–e2414790122. 1 indexed citations

Yamada, Takashi, Tatsuya Sato, Yuki Ashida, et al.. (2024). Platelet‐rich plasma does not accelerate the healing of damaged muscle following muscle strain. Journal of Orthopaedic Research®. 42(6). 1190–1199. 1 indexed citations

Sato, Tatsuya, Takashi Yamada, Yuki Ashida, et al.. (2023). Skeletal muscle endurance declines with impaired mitochondrial respiration and inadequate supply of acetyl-CoA during muscle fatigue in 5/6 nephrectomized rats. Journal of Applied Physiology. 135(4). 731–746. 5 indexed citations

Tamai, Katsuyuki, et al.. (2023). High‐intensity interval training in the form of isometric contraction improves fatigue resistance in dystrophin‐deficient muscle. The Journal of Physiology. 601(14). 2917–2933. 11 indexed citations

Yamada, Takashi, et al.. (2022). Improved skeletal muscle fatigue resistance in experimental autoimmune myositis mice following high-intensity interval training. Arthritis Research & Therapy. 24(1). 156–156. 15 indexed citations

Ashida, Yuki, et al.. (2022). Dissociation of SH3 and cysteine-rich domain 3 and junctophilin 1 from dihydropyridine receptor in dystrophin-deficient muscles. American Journal of Physiology-Cell Physiology. 323(3). C885–C895. 5 indexed citations

Sato, Tatsuya, Takashi Yamada, Yuki Ashida, et al.. (2022). Different metabolic changes between fast- and slow-twitch skeletal muscle underlie impaired muscle fatigue resistance in chronic kidney disease. Biophysical Journal. 121(3). 514a–514a.

Yamada, Takashi, Yuki Ashida, Katsuyuki Tamai, et al.. (2021). Larger improvements in fatigue resistance and mitochondrial function with high‐ than with low‐intensity contractions during interval training of mouse skeletal muscle. The FASEB Journal. 35(11). e21988–e21988. 13 indexed citations

Ashida, Yuki, Masami Abe, Yuki Saito, et al.. (2020). Eccentric Resistance Training Ameliorates Muscle Weakness in a Mouse Model of Idiopathic Inflammatory Myopathies. Arthritis & Rheumatology. 73(5). 848–857. 12 indexed citations

10.

Yamada, Takashi, et al.. (2020). Cancer Cachexia Induces Preferential Skeletal Muscle Myosin Loss When Combined With Denervation. Frontiers in Physiology. 11. 445–445. 15 indexed citations

11.

Yamada, Ryotaro, et al.. (2018). High-intensity eccentric training ameliorates muscle wasting in colon 26 tumor-bearing mice. PLoS ONE. 13(6). e0199050–e0199050. 14 indexed citations

12.

Yamada, Takashi, Ryotaro Yamada, Yuki Ashida, et al.. (2018). Electrical Stimulation Prevents Preferential Skeletal Muscle Myosin Loss in Steroid-Denervation Rats. Frontiers in Physiology. 9. 1111–1111. 9 indexed citations

13.

Roner, Michael R., et al.. (2009). Ability of Group IVB metallocene polyethers containing dienestrol to arrest the growth of selected cancer cell lines. BMC Cancer. 9(1). 358–358. 17 indexed citations

14.

Carraher, Charles E., et al.. (2007). Synthesis, structural characterization, and anti-cancer evaluation of group IVB-metallocene polyethers containing the synthetic estrogen diethylstilbestrol. 24(4). 357–369. 9 indexed citations

15.

Azuma, Masayuki, Yuki Ashida, Tetsuya Tamatani, et al.. (2006). Cepharanthin, a biscoclaurine alkaloid, prevents destruction of acinar tissues in murine Sjögren's syndrome.. PubMed. 33(5). 912–20. 12 indexed citations

16.

Ashida, Yuki, et al.. (2005). Anesthetic management of a neonate with vein of Galen aneurysmal malformations and severe pulmonary hypertension. Pediatric Anesthesia. 15(6). 525–528. 5 indexed citations

17.

Motegi, Katsumi, Masayuki Azuma, Tetsuya Tamatani, Yuki Ashida, & Mitsunobu Sato. (2005). Expression of aquaporin-5 in and fluid secretion from immortalized human salivary gland ductal cells by treatment with 5-aza-2′-deoxycytidine: a possibility for improvement of xerostomia in patients with Sjögren's syndrome. Laboratory Investigation. 85(3). 342–353. 37 indexed citations

18.

Tamatani, Tetsuya, Masayuki Azuma, Yuki Ashida, et al.. (2004). Enhanced radiosensitization and chemosensitization in NF‐κB‐suppressed human oral cancer cells via the inhibition of γ‐irradiation‐ and 5‐FU‐induced production of IL‐6 and IL‐8. International Journal of Cancer. 108(6). 912–921. 66 indexed citations

19.

Aota, Keiko, Masayuki Azuma, Tetsuya Tamatani, et al.. (2002). Stable Inhibition of NF-κB in Salivary Gland Cells Does Not Enhance Sensitivity to TNF-α-Induced Apoptosis Due to Upregulation of TRAF-1 Expression. Experimental Cell Research. 276(1). 111–119. 7 indexed citations

20.

Azuma, Masayuki, Keiko Aota, Tetsuya Tamatani, et al.. (2002). Suppression of tumor necrosis factor α–induced matrix metalloproteinase 9 production in human salivary gland acinar cells by cepharanthine occurs via down‐regulation of nuclear factor κB: A possible therapeutic agent for preventing the destruction of the acinar structure in the salivary glands of Sjögren's syndrome patients. Arthritis & Rheumatism. 46(6). 1585–1594. 41 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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