Masaki Kimata

436 total citations
7 papers, 351 citations indexed

About

Masaki Kimata is a scholar working on Surgery, Molecular Biology and Epidemiology. According to data from OpenAlex, Masaki Kimata has authored 7 papers receiving a total of 351 indexed citations (citations by other indexed papers that have themselves been cited), including 3 papers in Surgery, 3 papers in Molecular Biology and 3 papers in Epidemiology. Recurrent topics in Masaki Kimata's work include Mitochondrial Function and Pathology (3 papers), Autophagy in Disease and Therapy (2 papers) and Cardiovascular, Neuropeptides, and Oxidative Stress Research (1 paper). Masaki Kimata is often cited by papers focused on Mitochondrial Function and Pathology (3 papers), Autophagy in Disease and Therapy (2 papers) and Cardiovascular, Neuropeptides, and Oxidative Stress Research (1 paper). Masaki Kimata collaborates with scholars based in Japan. Masaki Kimata's co-authors include Hiroaki Matsubara, Satoaki Matoba, Maki Katamura, Koji Ikeda, Hideo Nakamura, Yuichiro Mita, Mitsuhiko Okigaki, Eri Iwai‐Kanai, Atsushi Hoshino and Yoshifumi Okawa and has published in prestigious journals such as American Journal of Physiology-Heart and Circulatory Physiology, Journal of Molecular and Cellular Cardiology and Circulation Heart Failure.

In The Last Decade

Masaki Kimata

7 papers receiving 346 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Masaki Kimata Japan	7	169	103	76	54	46	7	351
Jiangtao Yan China	12	159 0.9×	62 0.6×	90 1.2×	64 1.2×	36 0.8×	23	460
Gui-Ping Luo China	6	153 0.9×	109 1.1×	52 0.7×	36 0.7×	28 0.6×	6	334
Shunkui Luo China	9	129 0.8×	144 1.4×	36 0.5×	39 0.7×	37 0.8×	15	325
Ferhan Siddiqi Canada	11	280 1.7×	59 0.6×	46 0.6×	65 1.2×	37 0.8×	22	560
Ahmed Elwakiel Germany	9	188 1.1×	67 0.7×	52 0.7×	30 0.6×	28 0.6×	14	424
Jason M. Tanner United States	9	155 0.9×	54 0.5×	100 1.3×	39 0.7×	26 0.6×	11	451
Na Zhou China	13	277 1.6×	87 0.8×	124 1.6×	104 1.9×	59 1.3×	35	537
John Pirault France	12	133 0.8×	71 0.7×	75 1.0×	58 1.1×	17 0.4×	15	568
Alba Bosquet Spain	6	225 1.3×	75 0.7×	41 0.5×	95 1.8×	17 0.4×	7	329
Dina Xie United States	11	203 1.2×	59 0.6×	133 1.8×	76 1.4×	49 1.1×	16	379

Countries citing papers authored by Masaki Kimata

Since Specialization

Citations

This map shows the geographic impact of Masaki Kimata's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Masaki Kimata with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Masaki Kimata more than expected).

Fields of papers citing papers by Masaki Kimata

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Masaki Kimata. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Masaki Kimata. The network helps show where Masaki Kimata may publish in the future.

Co-authorship network of co-authors of Masaki Kimata

This figure shows the co-authorship network connecting the top 25 collaborators of Masaki Kimata. A scholar is included among the top collaborators of Masaki Kimata based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Masaki Kimata. Masaki Kimata is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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7 of 7 papers shown

1.

Ishii, Michiyo, et al.. (2021). The Value of Interleukin-6 among Several Inflammatory Markers as a Predictor of Respiratory Failure in COVID-19 Patients. Diagnostics. 11(8). 1327–1327. 9 indexed citations

2.

Hoshino, Atsushi, Satoaki Matoba, Eri Iwai‐Kanai, et al.. (2011). p53-TIGAR axis attenuates mitophagy to exacerbate cardiac damage after ischemia. Journal of Molecular and Cellular Cardiology. 52(1). 175–184. 139 indexed citations

3.

Nakamura, Hideo, Satoaki Matoba, Eri Iwai‐Kanai, et al.. (2011). p53 Promotes Cardiac Dysfunction in Diabetic Mellitus Caused by Excessive Mitochondrial Respiration-Mediated Reactive Oxygen Species Generation and Lipid Accumulation. Circulation Heart Failure. 5(1). 106–115. 79 indexed citations

4.

Kimata, Masaki, Satoaki Matoba, Eri Iwai‐Kanai, et al.. (2010). p53 and TIGAR regulate cardiac myocyte energy homeostasis under hypoxic stress. American Journal of Physiology-Heart and Circulatory Physiology. 299(6). H1908–H1916. 61 indexed citations

5.

Takeda, Mitsuo, Tetsuya Tatsumi, Shinsaku Matsunaga, et al.. (2007). Spironolactone Modulates Expressions of Cardiac Mineralocorticoid Receptor and 11.BETA.-Hydroxysteroid Dehydrogenase 2 and Prevents Ventricular Remodeling in Post-Infarct Rat Hearts. Hypertension Research. 30(5). 427–437. 37 indexed citations

6.

Katsume, Asako, Masaki Kimata, Satoshi Yamanaka, et al.. (2005). Placement of pacemaker leads via the extrathoracic subclavian vein guided by fluoroscopy and venography in the oblique projection. Heart and Vessels. 20(1). 19–22. 15 indexed citations

7.

Shiraishi, Jun, Tetsuya Tatsumi, Masaki Kimata, et al.. (2003). Echocardiographic Diagnosis of Pulmonary Vein Varix. Circulation Journal. 67(9). 796–798. 11 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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