Thomas Briston
Impact in
- Clinical Biochemistry top 10%
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- Cancer, Hypoxia, and Metabolism
Papers in
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- Mitochondrial Function and Pathology 5
- ATP Synthase and ATPases Research 3
- Signaling Pathways in Disease 1
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- Alzheimer's disease research and treatments 3
- Lysosomal Storage Disorders Research 1
- Co-authors
- György Szabadkai (4 shared papers)Michael R. Duchen (3 shared papers)David L. Selwood (1 shared paper)Emily H. Clark (2 shared papers)Aurelio Vázquez de la Torre (1 shared paper)Tamaki Hoshikawa (3 shared papers)James M. Staddon (4 shared papers)Siân Lewis (3 shared papers)
- Journals
- Scientific Reports (2 papers)SLAS DISCOVERY (1 paper)Frontiers in Oncology (1 paper)Journal of Biological Chemistry (1 paper)Cardiovascular Research (1 paper)
- Partner nations
- United KingdomItalyUnited States
In The Last Decade
Thomas Briston
10 papers receiving 715 citations
Peers
Comparison fields: 5 of 78
- Clinical Biochemistry 55
- Cancer Research 117
- Molecular Biology 460
- Physiology 169
- Aging 11
Countries citing papers authored by Thomas Briston
This map shows the geographic impact of Thomas Briston's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas Briston with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas Briston more than expected).
Fields of papers citing papers by Thomas Briston
This network shows the impact of papers produced by Thomas Briston. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas Briston. The network helps show where Thomas Briston may publish in the future.
Co-authors
The 25 scholars most cited alongside Thomas Briston, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2018 | 156 | |
| 2 | 2014 | 137 | |
| 3 | 2017 | 119 | |
| 4 | 2018 | 92 | |
| 5 | 2020 | 90 | |
| 6 | 2012 | 77 | |
| 7 | 2016 | 23 | |
| 8 | 2018 | 21 | |
| 9 | 2023 | 7 | |
| 10 | 2023 | 1 |
About Thomas Briston
Thomas Briston is a scholar working on Molecular Biology, Physiology, Neurology, Cancer Research and Epidemiology, having authored 10 papers that have together received 723 indexed citations. Recurring topics across this work include Mitochondrial Function and Pathology (5 papers), Alzheimer's disease research and treatments (3 papers), Cancer, Hypoxia, and Metabolism (3 papers), ATP Synthase and ATPases Research (3 papers), Parkinson's Disease Mechanisms and Treatments (3 papers), Autophagy in Disease and Therapy (2 papers), Signaling Pathways in Disease (1 paper) and Lysosomal Storage Disorders Research (1 paper). The work is most often cited by research in Clinical Biochemistry (55 citations), Cancer Research (117 citations), Molecular Biology (460 citations), Physiology (169 citations) and Aging (11 citations). Thomas Briston has collaborated with scholars based in United Kingdom, Italy and United States. Frequent co-authors include György Szabadkai, Michael R. Duchen, David L. Selwood, Emily H. Clark, Aurelio Vázquez de la Torre, Tamaki Hoshikawa, James M. Staddon, Siân Lewis, Malcolm C. Roberts and Patrick H. Maxwell. Their work appears in journals such as Scientific Reports, SLAS DISCOVERY, Frontiers in Oncology, Journal of Biological Chemistry and Cardiovascular Research.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.