Steven W. Plouffe
- Cell Biology top 0.2%
- Molecular Biology top 5%
- Oncology top 10%
- Cancer Research top 10%
- Immunology top 10%
- Co-authors
- Kun‐Liang GuanZhipeng MengFa‐Xing YuHyun Woo ParkToshiro MoroishiYoung Chul KimKimberly C. LinJung-Soon Mo
- Topics
- Hippo pathway signaling and YAP/TAZ (16 papers)Wnt/β-catenin signaling in development and cancer (3 papers)PI3K/AKT/mTOR signaling in cancer (3 papers)
- Partner nations
- United StatesChinaSouth Korea
In The Last Decade
Steven W. Plouffe
21 papers receiving 3.6k citations
Hit Papers
Peers
Comparison fields: 5 of 107
- Cell Biology 2.4k
- Molecular Biology 2.2k
- Oncology 360
- Cancer Research 303
- Immunology 286
Countries citing papers authored by Steven W. Plouffe
This map shows the geographic impact of Steven W. Plouffe's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Steven W. Plouffe with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Steven W. Plouffe more than expected).
Fields of papers citing papers by Steven W. Plouffe
This network shows the impact of papers produced by Steven W. Plouffe. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Steven W. Plouffe. The network helps show where Steven W. Plouffe may publish in the future.
Co-authorship network of co-authors of Steven W. Plouffe
This figure shows the co-authorship network connecting the top 25 collaborators of Steven W. Plouffe. A scholar is included among the top collaborators of Steven W. Plouffe based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Steven W. Plouffe. Steven W. Plouffe is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 18 | |
| 2 | 49 | |
| 3 | 88 | |
| 4 | 10 | |
| 5 | 50 | |
| 6 | 174 | |
| 7 | 156 | |
| 8 | 169 | |
| 9 | 158 | |
| 10 | 212 | |
| 11 | 68 | |
| 12 | 119 | |
| 13 | Alternative Wnt Signaling Activates YAP/TAZbreakdown → | 554 |
| 14 | 165 | |
| 15 | 283 | |
| 16 | MAP4K family kinases act in parallel to MST1/2 to activate LATS1/2 in the Hippo pathwaybreakdown → | 406 |
| 17 | 190 | |
| 18 | 25 | |
| 19 | 108 | |
| 20 | 12 |
About Steven W. Plouffe
Steven W. Plouffe is a scholar working on Cell Biology, Molecular Biology and Biochemistry, having authored 21 papers that have together received 3.6k indexed citations. Recurring topics across this work include Hippo pathway signaling and YAP/TAZ (16 papers), Wnt/β-catenin signaling in development and cancer (3 papers) and PI3K/AKT/mTOR signaling in cancer (3 papers). The work is most often cited by research in Cell Biology (2.4k citations), Biochemistry (246 citations) and Molecular Biology (2.2k citations). Steven W. Plouffe has collaborated with scholars based in United States, China and South Korea. Frequent co-authors include Kun‐Liang Guan, Zhipeng Meng, Fa‐Xing Yu, Hyun Woo Park, Toshiro Moroishi, Young Chul Kim, Kimberly C. Lin, Jung-Soon Mo, Jenna L. Jewell and Vincent S. Tagliabracci. Their work appears in journals such as Science, Cell and Journal of Biological Chemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.