Kimberly C. Lin

3.4k total citations · 1 hit paper
19 papers, 2.1k citations indexed

About

Kimberly C. Lin is a scholar working on Cell Biology, Genetics and Hematology. According to data from OpenAlex, Kimberly C. Lin has authored 19 papers receiving a total of 2.1k indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Cell Biology, 8 papers in Genetics and 8 papers in Hematology. Recurrent topics in Kimberly C. Lin's work include Hippo pathway signaling and YAP/TAZ (9 papers), Chronic Lymphocytic Leukemia Research (8 papers) and Acute Myeloid Leukemia Research (6 papers). Kimberly C. Lin is often cited by papers focused on Hippo pathway signaling and YAP/TAZ (9 papers), Chronic Lymphocytic Leukemia Research (8 papers) and Acute Myeloid Leukemia Research (6 papers). Kimberly C. Lin collaborates with scholars based in United States, China and South Korea. Kimberly C. Lin's co-authors include Kun‐Liang Guan, Hyun Woo Park, Steven W. Plouffe, Zhipeng Meng, Toshiro Moroishi, Caroline M. Alexander, Cun-Yu Wang, Jung-Soon Mo, Fa‐Xing Yu and Young Chul Kim and has published in prestigious journals such as Cell, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Kimberly C. Lin

19 papers receiving 2.1k citations

Hit Papers

Alternative Wnt Signaling Activates YAP/TAZ 2015 2026 2018 2022 2015 100 200 300 400 500

Peers

Kimberly C. Lin
Morvarid Mohseni United States
Karen Keeshan United Kingdom
Abdullah Mahmood Ali United States
W G King United States
Lorraine Lipfert United States
Anna V. Roschke United States
Frank Eckerdt United States
Ting-Lei Gu United States
Morvarid Mohseni United States
Kimberly C. Lin
Citations per year, relative to Kimberly C. Lin Kimberly C. Lin (= 1×) peers Morvarid Mohseni

Countries citing papers authored by Kimberly C. Lin

Since Specialization
Citations

This map shows the geographic impact of Kimberly C. Lin's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kimberly C. Lin with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kimberly C. Lin more than expected).

Fields of papers citing papers by Kimberly C. Lin

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kimberly C. Lin. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kimberly C. Lin. The network helps show where Kimberly C. Lin may publish in the future.

Co-authorship network of co-authors of Kimberly C. Lin

This figure shows the co-authorship network connecting the top 25 collaborators of Kimberly C. Lin. A scholar is included among the top collaborators of Kimberly C. Lin based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kimberly C. Lin. Kimberly C. Lin is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

19 of 19 papers shown
1.
Meng, Zhipeng, Cao Fang, Yunjiang Qiu, et al.. (2022). The Hippo pathway mediates Semaphorin signaling. Science Advances. 8(21). eabl9806–eabl9806. 12 indexed citations
2.
Luo, Min, Zhipeng Meng, Toshiro Moroishi, et al.. (2020). Publisher Correction: Heat stress activates YAP/TAZ to induce the heat shock transcriptome. Nature Cell Biology. 23(2). 209–209. 2 indexed citations
3.
Luo, Min, Zhipeng Meng, Toshiro Moroishi, et al.. (2020). Heat stress activates YAP/TAZ to induce the heat shock transcriptome. Nature Cell Biology. 22(12). 1447–1459. 69 indexed citations
4.
Lin, Kimberly C., Lingjun Zhang, Michael M. C. Kong, et al.. (2020). Development of an anti-human complement C6 monoclonal antibody that inhibits the assembly of membrane attack complexes. Blood Advances. 4(9). 2049–2057. 13 indexed citations
5.
Plouffe, Steven W., Kimberly C. Lin, Frederick E. Tan, et al.. (2018). The Hippo pathway effector proteins YAP and TAZ have both distinct and overlapping functions in the cell. Journal of Biological Chemistry. 293(28). 11230–11240. 174 indexed citations
6.
Smith, Catherine C., Amy L. Paguirigan, Grace R. Jeschke, et al.. (2017). Heterogeneous resistance to quizartinib in acute myeloid leukemia revealed by single-cell analysis. Blood. 130(1). 48–58. 120 indexed citations
7.
Diao, Yarui, Rongxin Fang, Bin Li, et al.. (2017). A tiling-deletion-based genetic screen for cis-regulatory element identification in mammalian cells. Nature Methods. 14(6). 629–635. 169 indexed citations
8.
Lin, Kimberly C., Hyun Woo Park, & Kun‐Liang Guan. (2017). Regulation of the Hippo Pathway Transcription Factor TEAD. Trends in Biochemical Sciences. 42(11). 862–872. 229 indexed citations
9.
Lin, Kimberly C., Toshiro Moroishi, Zhipeng Meng, et al.. (2017). Regulation of Hippo pathway transcription factor TEAD by p38 MAPK-induced cytoplasmic translocation. Nature Cell Biology. 19(8). 996–1002. 156 indexed citations
10.
Lin, Kimberly C., Hyun Woo Park, & Kun‐Liang Guan. (2017). Deregulation and Therapeutic Potential of the Hippo Pathway in Cancer. 2(1). 59–79. 15 indexed citations
11.
Plouffe, Steven W., Zhipeng Meng, Kimberly C. Lin, et al.. (2016). Characterization of Hippo Pathway Components by Gene Inactivation. Molecular Cell. 64(5). 993–1008. 212 indexed citations
12.
Park, Hyun Woo, Young Chul Kim, Bo Yu, et al.. (2015). Alternative Wnt Signaling Activates YAP/TAZ. Cell. 162(4). 780–794. 554 indexed citations breakdown →
13.
Lopez, Michael S., Elisabeth A. Lasater, Kimberly C. Lin, et al.. (2014). Overcoming myelosuppression due to synthetic lethal toxicity for FLT3-targeted acute myeloid leukemia therapy. eLife. 3. 44 indexed citations
14.
Smith, Catherine C., Elisabeth A. Lasater, Kimberly C. Lin, et al.. (2014). Crenolanib is a selective type I pan-FLT3 inhibitor. Proceedings of the National Academy of Sciences. 111(14). 5319–5324. 172 indexed citations
15.
Smith, Catherine C., Kimberly C. Lin, Elisabeth A. Lasater, et al.. (2013). Preclinical and Clinical Resistance Mechanisms To The Investigational Selective FLT3 Inhibitor PLX3397 In FLT3-ITD+ Acute Myeloid Leukemia (AML). Blood. 122(21). 3938–3938. 6 indexed citations
16.
Smith, Catherine C., Elisabeth A. Lasater, Xiaotian Zhu, et al.. (2013). Activity of ponatinib against clinically-relevant AC220-resistant kinase domain mutants of FLT3-ITD. Blood. 121(16). 3165–3171. 101 indexed citations
17.
Smith, Catherine C., Elisabeth A. Lasater, Amy L. Paguirigan, et al.. (2012). Constitutively Activating Mutations At the FLT3 Activation Loop Residue D835 Are Associated with Clinical Resistance to AC220. Blood. 120(21). 674–674. 4 indexed citations
18.
Smith, Catherine C., Elisabeth A. Lasater, Melissa Q. McCreery, et al.. (2012). Crenolanib (CP-868,596) Is a Potent and Selective Type I FLT3 Inhibitor That Retains Activity Against AC220 Resistance-Causing FLT3 Kinase Domain Mutants. Blood. 120(21). 141–141. 14 indexed citations
19.
Smith, Catherine C., Michael P. Brown, Wendy T Parker, et al.. (2012). Single Molecule Real Time (SMRT™) Sequencing Sensitively Detects the Evolution of Polyclonal and Compound BCR-ABL Mutations in Patients Who Relapse On Kinase Inhibitor Therapy. Blood. 120(21). 917–917. 3 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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