Simon Sjödin

667 citations
15 papers · 415 · h-index 10

Impact in

  • Neurology top 10%
    • Parkinson's Disease Mechanisms and Treatments
    • Neurological disorders and treatments
  • Physiology top 10%
    • Alzheimer's disease research and treatments
    • Lysosomal Storage Disorders Research

Papers in

    • Alzheimer's disease research and treatments 8
    • Lysosomal Storage Disorders Research 4
    • Parkinson's Disease Mechanisms and Treatments 6

Simon Sjödin

15 papers receiving 413 citations

Peers

Simon Sjödin
Comparison fields: 5 of 58
  • Neurology 144
  • Physiology 233
  • Neurology 53
  • Cellular and Molecular Neuroscience 70
  • Psychiatry and Mental health 53
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Citations per field
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Citations per year

Countries citing papers authored by Simon Sjödin

Since Specialization
Citations

This map shows the geographic impact of Simon Sjödin's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Simon Sjödin with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Simon Sjödin more than expected).

Fields of papers citing papers by Simon Sjödin

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Simon Sjödin. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Simon Sjödin. The network helps show where Simon Sjödin may publish in the future.

Co-authors

The 25 scholars most cited alongside Simon Sjödin, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Simon Sjödin Line = papers co-authored together Simon Sjödin links everyone, so they are left out of the graph.

All Works

15 of 15 papers shown
#Work
1 201776
2 202172
3 201964
4 202153
5 202030
6 201330
7 201725
8 201622
9 202116
10 20209
11 20158
12 20224
13 20233
14 20232
15 20251

About Simon Sjödin

Simon Sjödin is a scholar working on Physiology, Neurology, Molecular Biology, Cellular and Molecular Neuroscience and Epidemiology, having authored 15 papers that have together received 415 indexed citations. Recurring topics across this work include Alzheimer's disease research and treatments (8 papers), Parkinson's Disease Mechanisms and Treatments (6 papers), Lysosomal Storage Disorders Research (4 papers), Mitochondrial Function and Pathology (2 papers), Autophagy in Disease and Therapy (2 papers), Hereditary Neurological Disorders (1 paper), Machine Learning in Bioinformatics (1 paper) and Ginkgo biloba and Cashew Applications (1 paper). The work is most often cited by research in Neurology (144 citations), Physiology (233 citations), Neurology (53 citations), Cellular and Molecular Neuroscience (70 citations) and Psychiatry and Mental health (53 citations). Simon Sjödin has collaborated with scholars based in Sweden, United Kingdom and Italy. Frequent co-authors include Henrik Zetterberg, Kaj Blennow, Ann Brinkmalm, Gunnar Brinkmalm, Annika Öhrfelt, Oskar Hansson, Anja Hviid Simonsen, Steen Gregers Hasselbalch, Erik Portelius and Lucilla Parnetti. Their work appears in journals such as Alzheimer s Research & Therapy, Alzheimer s & Dementia Diagnosis Assessment & Disease Monitoring, PROTEOMICS - CLINICAL APPLICATIONS, Multiple Sclerosis and Related Disorders and Clinical Epigenetics.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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