Sandra O. Tomé

904 total citations
22 papers, 464 citations indexed

About

Sandra O. Tomé is a scholar working on Physiology, Cellular and Molecular Neuroscience and Neurology. According to data from OpenAlex, Sandra O. Tomé has authored 22 papers receiving a total of 464 indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Physiology, 9 papers in Cellular and Molecular Neuroscience and 8 papers in Neurology. Recurrent topics in Sandra O. Tomé's work include Alzheimer's disease research and treatments (13 papers), Amyotrophic Lateral Sclerosis Research (6 papers) and Dementia and Cognitive Impairment Research (5 papers). Sandra O. Tomé is often cited by papers focused on Alzheimer's disease research and treatments (13 papers), Amyotrophic Lateral Sclerosis Research (6 papers) and Dementia and Cognitive Impairment Research (5 papers). Sandra O. Tomé collaborates with scholars based in Belgium, United States and Germany. Sandra O. Tomé's co-authors include Dietmar Rudolf Thal, Rik Vandenberghe, Thomas Tousseyn, Christine A. F. Von Arnim, Simona Ospitalieri, Evelien Van Schoor, Markus Otto, Philip Van Damme, Clévio Nóbrega and Liliana Mendonça and has published in prestigious journals such as Brain, Human Molecular Genetics and Acta Neuropathologica.

In The Last Decade

Sandra O. Tomé

19 papers receiving 462 citations

Peers

Sandra O. Tomé
Sandra O. Tomé
Citations per year, relative to Sandra O. Tomé Sandra O. Tomé (= 1×) peers Simona Ospitalieri

Countries citing papers authored by Sandra O. Tomé

Since Specialization
Citations

This map shows the geographic impact of Sandra O. Tomé's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sandra O. Tomé with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sandra O. Tomé more than expected).

Fields of papers citing papers by Sandra O. Tomé

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Sandra O. Tomé. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sandra O. Tomé. The network helps show where Sandra O. Tomé may publish in the future.

Co-authorship network of co-authors of Sandra O. Tomé

This figure shows the co-authorship network connecting the top 25 collaborators of Sandra O. Tomé. A scholar is included among the top collaborators of Sandra O. Tomé based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Sandra O. Tomé. Sandra O. Tomé is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
2.
Thal, Dietmar Rudolf, Alicja Ronisz, Simona Ospitalieri, et al.. (2025). Tau pathology in the retina and its role in neurodegeneration. Alzheimer s & Dementia. 21(S1). e102036–e102036. 1 indexed citations
3.
Tomé, Sandra O., Caroline Huart, Karelle Leroy, et al.. (2025). The olfactory epithelium: a critical gateway for pathological tau propagation and a target for mitigating tauopathy in the central nervous system. Acta Neuropathologica. 149(1). 64–64.
4.
Quenon, Lisa, et al.. (2025). Specific atrophy patterns distinguish tau and TDP-43 pathology: a longitudinal MRI ante-mortem study. Acta Neuropathologica Communications. 14(1). 17–17.
5.
Katsumata, Yuriko, David W. Fardo, Davis C. Woodworth, et al.. (2024). Pure LATE-NC: Frequency, clinical impact, and the importance of considering APOE genotype when assessing this and other subtypes of non-Alzheimer’s pathologies. Acta Neuropathologica. 148(1). 66–66. 5 indexed citations
6.
Ronisz, Alicja, Simona Ospitalieri, Sandra O. Tomé, et al.. (2024). pTau pathology in the retina of TAU58 mice: association with ganglion cell degeneration and implications on seeding and propagation of pTau from human brain lysates. Acta Neuropathologica Communications. 12(1). 194–194. 2 indexed citations
7.
Gawor, Klara, Sandra O. Tomé, Rik Vandenberghe, et al.. (2024). Amygdala-predominant α-synuclein pathology is associated with exacerbated hippocampal neuron loss in Alzheimer’s disease. Brain Communications. 6(6). fcae442–fcae442. 6 indexed citations
8.
Küçükali, Fahri, Jasper Van Dongen, Klara Gawor, et al.. (2024). Hypothesis-based investigation of known AD risk variants reveals the genetic underpinnings of neuropathological lesions observed in Alzheimer’s-type dementia. Acta Neuropathologica. 148(1). 55–55.
9.
Tomé, Sandra O., Klara Gawor, & Dietmar Rudolf Thal. (2023). LATE‐NC in Alzheimer's disease: Molecular aspects and synergies. Brain Pathology. 34(4). e13213–e13213. 10 indexed citations
10.
Tomé, Sandra O., Alicja Ronisz, Simona Ospitalieri, et al.. (2023). TDP-43 pathology is associated with increased tau burdens and seeding. Molecular Neurodegeneration. 18(1). 71–71. 32 indexed citations
11.
Schoor, Evelien Van, Simona Ospitalieri, Sandra O. Tomé, et al.. (2022). Increased pyroptosis activation in white matter microglia is associated with neuronal loss in ALS motor cortex. Acta Neuropathologica. 144(3). 393–411. 42 indexed citations
12.
Koper, Marta J., Sandra O. Tomé, Klara Gawor, et al.. (2022). LATE-NC aggravates GVD-mediated necroptosis in Alzheimer’s disease. Acta Neuropathologica Communications. 10(1). 128–128. 24 indexed citations
13.
Thal, Dietmar Rudolf & Sandra O. Tomé. (2022). The central role of tau in Alzheimer’s disease: From neurofibrillary tangle maturation to the induction of cell death. Brain Research Bulletin. 190. 204–217. 54 indexed citations
14.
Tomé, Sandra O., Diogo Teixeira, David V.C. Brito, et al.. (2022). The stress granule protein G3BP1 alleviates spinocerebellar ataxia-associated deficits. Brain. 146(6). 2346–2363. 18 indexed citations
15.
Tomé, Sandra O., et al.. (2021). TDP-43 interacts with pathological τ protein in Alzheimer’s disease. Acta Neuropathologica. 141(5). 795–799. 33 indexed citations
16.
Uytterhoeven, Valerie, Sandra O. Tomé, Thomas Tousseyn, et al.. (2021). Maturation of neuronal AD-tau pathology involves site-specific phosphorylation of cytoplasmic and synaptic tau preceding conformational change and fibril formation. Acta Neuropathologica. 141(2). 173–192. 45 indexed citations
17.
Tomé, Sandra O., Rik Vandenberghe, Simona Ospitalieri, et al.. (2020). Distinct molecular patterns of TDP-43 pathology in Alzheimer’s disease: relationship with clinical phenotypes. Acta Neuropathologica Communications. 8(1). 61–61. 69 indexed citations
18.
Weerasekera, Akila, Melissa Crabbé, Sandra O. Tomé, et al.. (2020). Non-invasive characterization of amyotrophic lateral sclerosis in a hTDP-43A315T mouse model: A PET-MR study. NeuroImage Clinical. 27. 102327–102327. 14 indexed citations
19.
Nóbrega, Clévio, Liliana Mendonça, Adriana Marcelo, et al.. (2019). Restoring brain cholesterol turnover improves autophagy and has therapeutic potential in mouse models of spinocerebellar ataxia. Acta Neuropathologica. 138(5). 837–858. 57 indexed citations
20.
Mendonça, Liliana, Clévio Nóbrega, Maximilian Brinkhaus, et al.. (2019). Ibuprofen enhances synaptic function and neural progenitors proliferation markers and improves neuropathology and motor coordination in Machado–Joseph disease models. Human Molecular Genetics. 28(22). 3691–3703. 26 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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