Shin’ichi Takeda

23.6k citations

427 papers · 17.5k indexed · 3 hit papers · h-index 70

Genetics top 0.2%
- Virus-based gene therapy research 42
- Neurogenetic and Muscular Disorders Research 28
Molecular Biology top 0.2%
- Muscle Physiology and Disorders 233
- RNA Interference and Gene Delivery 29
Aging top 0.5%
Rehabilitation top 0.2%
Physiology top 0.5%
- Adipose Tissue and Metabolism 31
Surgery
- Tissue Engineering and Regenerative Medicine 53
Cardiology and Cardiovascular Medicine
- Cardiomyopathy and Myosin Studies 24
Cellular and Molecular Neuroscience
- Genetic Neurodegenerative Diseases 23

Co-authors: Yuko Miyagoe‐Suzuki Akiyoshi Uezumi So‐ichiro Fukada Akinori Nakamura Kunihiro Tsuchida Yoshitsugu Aoki Naoki Yamamoto Katsutoshi Yuasa
Cited by: Genetics Molecular Biology Aging
Partner nations: Japan United States Canada

In The Last Decade

Shin’ichi Takeda

417 papers receiving 17.3k citations

Hit Papers

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What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

2021 Duchenne muscular dystrophy
2011 Fibrosis and adipogenesis originate from a common mesenchymal progenitor in skeletal muscle
2010 Mesenchymal progenitors distinct from satellite cells contribute to ectopic fat cell formation in skeletal muscle

Peers

Countries citing papers authored by Shin’ichi Takeda

Since Specialization

Citations

This map shows the geographic impact of Shin’ichi Takeda's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Shin’ichi Takeda with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Shin’ichi Takeda more than expected).

Fields of papers citing papers by Shin’ichi Takeda

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Shin’ichi Takeda. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Shin’ichi Takeda. The network helps show where Shin’ichi Takeda may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Shin’ichi Takeda, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Shin’ichi Takeda Line = papers co-authored together Shin’ichi Takeda links everyone, so they are left out of the graph.

All Works

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20 of 20 papers shown

#	Work
1	A historical perspective on the development of antisense oligonucleotide treatments for Duchenne muscular dystrophy and spinal muscular atrophy Annemieke Aartsma‐Rus,Shin’ichi Takeda	2025	1
2	The R436Q missense mutation in WWP1 disrupts autoinhibition of its E3 ubiquitin ligase activity, leading to self-degradation and loss of function Michihiro Imamura,Hirokazu Matsumoto,Hideyuki Mannen,Shin’ichi Takeda,Yoshitsugu Aoki	2024	0
3	Increasing numbers of CD19 + CD24highCD38high regulatory B cells and pre-germinal center B cells reflect activated autoimmunity and predict future treatment response in patients with untreated immune thrombocytopenia Tetsuya Hayashi,Hirohisa Nakamae,Shin’ichi Takeda,Yasuhiro Nakashima,Hideo Koh,Mitsutaka Nishimoto,Hiroshi Okamura,Satoru Nanno,Yosuke Makuuchi,Masatomo Kuno,Mika Nakamae,Asao Hirose,Masayuki Hino	2021	4
4	Duchenne muscular dystrophybreakdown → Dongsheng Duan,Nathalie Goemans,Shin’ichi Takeda,Eugenio Mercuri,Annemieke Aartsma‐Rus	2021	708
5	Psychiatric and neurodevelopmental aspects of Becker muscular dystrophy Madoka Mori‐Yoshimura,Y Mizuno,Sumiko Yoshida,Naoko Ishihara,Narihiro Minami,Emiko Morimoto,Kazushi Maruo,Ikuya Nonaka,Hirofumi Komaki,Ichizo Nishino,Masayuki Sekiguchi,Noriko Sato,Shin’ichi Takeda,Yuji Takahashi	2019	28
6	Induction of Pluripotent Stem Cells from a Manifesting Carrier of Duchenne Muscular Dystrophy and Characterization of Their X-Inactivation Status Yuko Miyagoe‐Suzuki,Takashi Nishiyama,Miho Nakamura,Asako Narita,Fusako Sakai‐Takemura,S. Masuda,Narihiro Minami,Kumiko Murayama,Hirofumi Komaki,Yu‐ichi Goto,Shin’ichi Takeda	2017	6
7	Endogenous Multiple Exon Skipping and Back-Splicing at the DMD Mutation Hotspot Hitoshi Suzuki,Yoshitsugu Aoki,T. Kameyama,Takashi Saito,S. Masuda,Jun Tanihata,Tetsuya Nagata,Akila Mayeda,Shin’ichi Takeda,Toshifumi Tsukahara	2016	36
8	Recent advances in innovative therapeutic approaches for Duchenne muscular dystrophy: from discovery to clinical trials. Yuko Shimizu‐Motohashi,Shouta Miyatake,Hirofumi Komaki,Shin’ichi Takeda,Yoshitsugu Aoki	2016	70
9	DOK7 gene therapy benefits mouse models of diseases characterized by defects in the neuromuscular junction Sumimasa Arimura,Takashi Okada,Tohru Tezuka,Tomoko Chiyo,Yuko Kasahara,Toshiro Yoshimura,Masakatsu Motomura,Nobuaki Yoshida,David Beeson,Shin’ichi Takeda,Yuji Yamanashi	2014	69
10	Ubiquitin Ligase Cbl-b Is a Negative Regulator for Insulin-Like Growth Factor 1 Signaling during Muscle Atrophy Caused by Unloading Reiko Nakao,Katsuya Hirasaka,Jumpei Goto,Kazumi Ishidoh,Chiharu Yamada,Ayako Ohno,Yuushi Okumura,Ikuya Nonaka,Koji Yasutomo,Kenneth M. Baldwin,Eiki Kominami,Akira Higashibata,Keisuke Nagano,Keiji Tanaka,Natsuo Yasui,Edward Mills,Shin’ichi Takeda,Takeshi Nikawa	2009	151
11	A case of heparin-induced thrombocytopenia associated with respiratory failure due to pseudopulmonary embolism Atsushi Kotoda,Maki Kato,Tetsu Akimoto,Hideaki Takahashi,Chiharu Ito,Shin’ichi Takeda,Yukio Ando,Shigeaki Muto,Wako Yumura,Eiji Kusano	2009	1
12	Residual laminin-binding activity and enhanced dystroglycan glycosylation by LARGE in novel model mice to dystroglycanopathy Motoi Kanagawa,Akemi Nishimoto,Tomohiro Chiyonobu,Satoshi Takeda,Yuko Miyagoe‐Suzuki,Fan Wang,Nobuhiro Fujikake,Mariko Taniguchi,Zhongpeng Lu,Masaji Tachikawa,Yoshitaka Nagai,Fumi Tashiro,Jun‐ichi Miyazaki,Youichi Tajima,Shin’ichi Takeda,Tamao Endo,Kazuhiro Kobayashi,Kevin P. Campbell,Tatsushi Toda	2008	61
13	Clinical spectrum of pulmonary hamartoma diagnosed by surgical resection Toshiteru Tokunaga,Shin’ichi Takeda,Masaru Koma,Noriyoshi Sawabata,Hajime Maeda	2008	1
14	Cerebral blood flow, oxygen consumption and glucose utilization in humans:a stereotaxic correlation studied with PET and SPM Kazuhiko Hayashi,Shin’ichi Takeda,Keisuke Miyake,Kentaroh Fujii,Taku Fujimoto,Yasuhiro Ohsaki,Ashik B. Ansar,Katsufumi Kajimoto,Yasuyuki Kimura,Hiroki Katoh,Makiko Tanaka,Eku Shimosegawa,Jun Hatazawa	2007	2
15	NO production results in suspension-induced muscle atrophy through dislocation of neuronal NOS Naoki Suzuki,Norio Motohashi,Akiyoshi Uezumi,So‐ichiro Fukada,Tetsuhiko Yoshimura,Yasuto Itoyama,Masashi Aoki,Yuko Miyagoe‐Suzuki,Shin’ichi Takeda	2007	152
16	The utrophin promoter A drives high expression of the transgenicLacZ gene in liver, testis, colon, submandibular gland, and small intestine Joji Takahashi,Yuka Itoh,Keita Fujimori,Michihiro Imamura,Yoshihiro Wakayama,Yuko Miyagoe‐Suzuki,Shin’ichi Takeda	2004	11
17	Endothelial PAS Domain Protein 1 (EPAS1) Induces Adrenomedullin Gene Expression in Cardiac Myocytes: Role of EPAS1 in an Inflammatory Response in Cardiac Myocytes Toru Tanaka,Hideo Akiyama,Hiroyoshi Kanai,Mahito Sato,Shin’ichi Takeda,Kenichi Sekiguchi,Tomoyuki Yokoyama,Masahiko Kurabayashi	2002	19
18	Efficient modification of a human chromosome by telomere-directed truncation in high homologous recombination-proficient chicken DT40 cells Yoshimi Kuroiwa,Tamao Shinohara,Tomomi Notsu,Kazuma Tomizuka,Hitoshi Yoshida,Shin’ichi Takeda,Mitsuo Oshimura,I. Ishida	1998	50
19	Laminin α2 chain‐null mutant mice by targeted disruption of the Lama2 gene: a new model of merosin (laminin 2)‐deficient congenital muscular dystrophy Yuko Miyagoe,Kazunori Hanaoka,Ikuya Nonaka,Michiko Hayasaka,Yoko Nabeshima,Kiichi Arahata,Yo-ichi Nabeshima,Shin’ichi Takeda	1997	222
20	Two distinct types of crescentic glomerulonephritis. Shin’ichi Takeda,Hiroshi Kida,H. Yokoyama,N Tomosugi,K Kobayashi	1992	10

About Shin’ichi Takeda

Shin’ichi Takeda is a scholar working on Genetics, Molecular Biology and Nephrology, having authored 427 papers that have together received 17.5k indexed citations. Recurring topics across this work include Muscle Physiology and Disorders (233 papers), Tissue Engineering and Regenerative Medicine (53 papers), Virus-based gene therapy research (42 papers), Adipose Tissue and Metabolism (31 papers), RNA Interference and Gene Delivery (29 papers), Neurogenetic and Muscular Disorders Research (28 papers), Cardiomyopathy and Myosin Studies (24 papers) and Genetic Neurodegenerative Diseases (23 papers). The work is most often cited by research in Genetics (2.5k citations), Molecular Biology (12.6k citations) and Aging (315 citations). Shin’ichi Takeda has collaborated with scholars based in Japan, United States and Canada. Frequent co-authors include Yuko Miyagoe‐Suzuki, Akiyoshi Uezumi, So‐ichiro Fukada, Akinori Nakamura, Kunihiro Tsuchida, Yoshitsugu Aoki, Naoki Yamamoto, Katsutoshi Yuasa, Hiroshi Yamamoto and Toshifumi Yokota.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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