Shin’ichi Takeda

23.6k citations

427 papers · 17.5k indexed · 3 hit papers · h-index 70

Molecular Biology top 0.2%
Surgery top 0.5%
Physiology top 0.5%
Genetics top 0.2%
Genetics top 0.5%

Co-authors: Yuko Miyagoe‐Suzuki Akiyoshi Uezumi So‐ichiro Fukada Akinori Nakamura Kunihiro Tsuchida Yoshitsugu Aoki Naoki Yamamoto Katsutoshi Yuasa
Topics: Muscle Physiology and Disorders (233 papers)Tissue Engineering and Regenerative Medicine (53 papers)Virus-based gene therapy research (42 papers)
Cited by: Genetics Molecular Biology Aging
Journals: Nature Science Proceedings of the National Academy of Sciences
Partner nations: Japan United States Canada

In The Last Decade

Shin’ichi Takeda

417 papers receiving 17.3k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Mesenchymal progenitors distinct from satellite cells contribute to ectopic fat cell formation in skeletal muscle

2010 1.0k citations Akiyoshi Uezumi, So‐ichiro Fukada et al. profile →
Duchenne muscular dystrophy

2021 708 citations Dongsheng Duan, Nathalie Goemans et al. Nature Reviews Disease Primers profile →
Fibrosis and adipogenesis originate from a common mesenchymal progenitor in skeletal muscle

2011 499 citations Akiyoshi Uezumi, Yuko Miyagoe‐Suzuki et al. profile →

Peers

Countries citing papers authored by Shin’ichi Takeda

Since Specialization

Citations

This map shows the geographic impact of Shin’ichi Takeda's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Shin’ichi Takeda with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Shin’ichi Takeda more than expected).

Fields of papers citing papers by Shin’ichi Takeda

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Shin’ichi Takeda. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Shin’ichi Takeda. The network helps show where Shin’ichi Takeda may publish in the future.

Co-authorship network of co-authors of Shin’ichi Takeda

This figure shows the co-authorship network connecting the top 25 collaborators of Shin’ichi Takeda. A scholar is included among the top collaborators of Shin’ichi Takeda based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Shin’ichi Takeda. Shin’ichi Takeda is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Work	Indexed citations
1	A historical perspective on the development of antisense oligonucleotide treatments for Duchenne muscular dystrophy and spinal muscular atrophy 2025 ·Journal of Neuromuscular Diseases ·Annemieke Aartsma‐Rus,Shin’ichi Takeda	1
2	The R436Q missense mutation in WWP1 disrupts autoinhibition of its E3 ubiquitin ligase activity, leading to self-degradation and loss of function 2024 ·In Vitro Cellular & Developmental Biology - Animal ·Michihiro Imamura,Hirokazu Matsumoto,Hideyuki Mannen,Shin’ichi Takeda,Yoshitsugu Aoki	0
3	Increasing numbers of CD19 + CD24highCD38high regulatory B cells and pre-germinal center B cells reflect activated autoimmunity and predict future treatment response in patients with untreated immune thrombocytopenia 2021 ·International Journal of Hematology ·Tetsuya Hayashi,Hirohisa Nakamae,Shin’ichi Takeda,Yasuhiro Nakashima,Hideo Koh,Mitsutaka Nishimoto,Hiroshi Okamura,(unknown),(unknown),(unknown),Mika Nakamae,Asao Hirose,Masayuki Hino	4
4	Duchenne muscular dystrophybreakdown → 2021 ·Nature Reviews Disease Primers ·Dongsheng Duan,Nathalie Goemans,Shin’ichi Takeda,Eugenio Mercuri,Annemieke Aartsma‐Rus	708
5	Psychiatric and neurodevelopmental aspects of Becker muscular dystrophy 2019 ·Neuromuscular Disorders ·Madoka Mori‐Yoshimura,(unknown),Sumiko Yoshida,(unknown),Narihiro Minami,Emiko Morimoto,Kazushi Maruo,Ikuya Nonaka,Hirofumi Komaki,Ichizo Nishino,Masayuki Sekiguchi,Noriko Sato,Shin’ichi Takeda,Yuji Takahashi	28
6	Induction of Pluripotent Stem Cells from a Manifesting Carrier of Duchenne Muscular Dystrophy and Characterization of Their X-Inactivation Status 2017 ·Stem Cells International ·Yuko Miyagoe‐Suzuki,Takashi Nishiyama,(unknown),Asako Narita,(unknown),S. Masuda,Narihiro Minami,Kumiko Murayama,Hirofumi Komaki,Yu‐ichi Goto,Shin’ichi Takeda	6
7	Endogenous Multiple Exon Skipping and Back-Splicing at the DMD Mutation Hotspot 2016 ·International Journal of Molecular Sciences ·Hitoshi Suzuki,Yoshitsugu Aoki,T. Kameyama,Takashi Saito,S. Masuda,Jun Tanihata,Tetsuya Nagata,Akila Mayeda,Shin’ichi Takeda,Toshifumi Tsukahara	36
8	Recent advances in innovative therapeutic approaches for Duchenne muscular dystrophy: from discovery to clinical trials. 2016 ·PubMed Central ·Yuko Shimizu‐Motohashi,Shouta Miyatake,Hirofumi Komaki,Shin’ichi Takeda,Yoshitsugu Aoki	70
9	DOK7 gene therapy benefits mouse models of diseases characterized by defects in the neuromuscular junction 2014 ·Science ·(unknown),Takashi Okada,Tohru Tezuka,Tomoko Chiyo,(unknown),Toshiro Yoshimura,Masakatsu Motomura,Nobuaki Yoshida,David Beeson,Shin’ichi Takeda,Yuji Yamanashi	69
10	Ubiquitin Ligase Cbl-b Is a Negative Regulator for Insulin-Like Growth Factor 1 Signaling during Muscle Atrophy Caused by Unloading 2009 ·Molecular and Cellular Biology ·Reiko Nakao,Katsuya Hirasaka,(unknown),Kazumi Ishidoh,Chiharu Yamada,Ayako Ohno,Yuushi Okumura,Ikuya Nonaka,Koji Yasutomo,Kenneth M. Baldwin,Eiki Kominami,Akira Higashibata,Keisuke Nagano,Keiji Tanaka,Natsuo Yasui,Edward Mills,Shin’ichi Takeda,Takeshi Nikawa	151
11	A case of heparin-induced thrombocytopenia associated with respiratory failure due to pseudopulmonary embolism 2009 ·Nihon Toseki Igakkai Zasshi ·(unknown),(unknown),Tetsu Akimoto,Hideaki Takahashi,Chiharu Ito,Shin’ichi Takeda,Yukio Ando,Shigeaki Muto,Wako Yumura,Eiji Kusano	1
12	Residual laminin-binding activity and enhanced dystroglycan glycosylation by LARGE in novel model mice to dystroglycanopathy 2008 ·Human Molecular Genetics ·Motoi Kanagawa,(unknown),Tomohiro Chiyonobu,Satoshi Takeda,Yuko Miyagoe‐Suzuki,Fan Wang,Nobuhiro Fujikake,Mariko Taniguchi,(unknown),Masaji Tachikawa,Yoshitaka Nagai,Fumi Tashiro,Jun‐ichi Miyazaki,Youichi Tajima,Shin’ichi Takeda,Tamao Endo,Kazuhiro Kobayashi,Kevin P. Campbell,Tatsushi Toda	61
13	Clinical spectrum of pulmonary hamartoma diagnosed by surgical resection 2008 ·The Journal of the Japanese Association for Chest Surgery ·Toshiteru Tokunaga,Shin’ichi Takeda,Masaru Koma,Noriyoshi Sawabata,Hajime Maeda	1
14	Cerebral blood flow, oxygen consumption and glucose utilization in humans:a stereotaxic correlation studied with PET and SPM 2007 ·Journal of Cerebral Blood Flow & Metabolism ·Kazuhiko Hayashi,Shin’ichi Takeda,Keisuke Miyake,(unknown),Taku Fujimoto,(unknown),(unknown),Katsufumi Kajimoto,Yasuyuki Kimura,Hiroki Katoh,Makiko Tanaka,Eku Shimosegawa,Jun Hatazawa	2
15	NO production results in suspension-induced muscle atrophy through dislocation of neuronal NOS 2007 ·Journal of Clinical Investigation ·Naoki Suzuki,Norio Motohashi,Akiyoshi Uezumi,So‐ichiro Fukada,Tetsuhiko Yoshimura,Yasuto Itoyama,Masashi Aoki,Yuko Miyagoe‐Suzuki,Shin’ichi Takeda	152
16	The utrophin promoter A drives high expression of the transgenicLacZ gene in liver, testis, colon, submandibular gland, and small intestine 2004 ·The Journal of Gene Medicine ·Joji Takahashi,(unknown),(unknown),Michihiro Imamura,Yoshihiro Wakayama,Yuko Miyagoe‐Suzuki,Shin’ichi Takeda	11
17	Endothelial PAS Domain Protein 1 (EPAS1) Induces Adrenomedullin Gene Expression in Cardiac Myocytes: Role of EPAS1 in an Inflammatory Response in Cardiac Myocytes 2002 ·Journal of Molecular and Cellular Cardiology ·Toru Tanaka,Hideo Akiyama,Hiroyoshi Kanai,Mahito Sato,Shin’ichi Takeda,Kenichi Sekiguchi,Tomoyuki Yokoyama,Masahiko Kurabayashi	19
18	Efficient modification of a human chromosome by telomere-directed truncation in high homologous recombination-proficient chicken DT40 cells 1998 ·Nucleic Acids Research ·Yoshimi Kuroiwa,(unknown),(unknown),Kazuma Tomizuka,Hitoshi Yoshida,Shin’ichi Takeda,Mitsuo Oshimura,I. Ishida	50
19	Laminin α2 chain‐null mutant mice by targeted disruption of the Lama2 gene: a new model of merosin (laminin 2)‐deficient congenital muscular dystrophy 1997 ·FEBS Letters ·Yuko Miyagoe,Kazunori Hanaoka,Ikuya Nonaka,Michiko Hayasaka,Yoko Nabeshima,Kiichi Arahata,Yo-ichi Nabeshima,Shin’ichi Takeda	222
20	Two distinct types of crescentic glomerulonephritis. 1992 ·PubMed ·Shin’ichi Takeda,Hiroshi Kida,H. Yokoyama,(unknown),K Kobayashi	10

About Shin’ichi Takeda

Shin’ichi Takeda is a scholar working on Genetics, Molecular Biology and Nephrology, having authored 427 papers that have together received 17.5k indexed citations. Recurring topics across this work include Muscle Physiology and Disorders (233 papers), Tissue Engineering and Regenerative Medicine (53 papers) and Virus-based gene therapy research (42 papers). The work is most often cited by research in Genetics (2.5k citations), Molecular Biology (12.6k citations) and Aging (315 citations). Shin’ichi Takeda has collaborated with scholars based in Japan, United States and Canada. Frequent co-authors include Yuko Miyagoe‐Suzuki, Akiyoshi Uezumi, So‐ichiro Fukada, Akinori Nakamura, Kunihiro Tsuchida, Yoshitsugu Aoki, Naoki Yamamoto, Katsutoshi Yuasa, Hiroshi Yamamoto and Toshifumi Yokota. Their work appears in journals such as Nature, Science and Proceedings of the National Academy of Sciences.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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