Motoi Kanagawa

5.0k total citations · 1 hit paper
73 papers, 3.6k citations indexed

About

Motoi Kanagawa is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Cell Biology. According to data from OpenAlex, Motoi Kanagawa has authored 73 papers receiving a total of 3.6k indexed citations (citations by other indexed papers that have themselves been cited), including 60 papers in Molecular Biology, 13 papers in Cellular and Molecular Neuroscience and 13 papers in Cell Biology. Recurrent topics in Motoi Kanagawa's work include Muscle Physiology and Disorders (41 papers), Ubiquitin and proteasome pathways (11 papers) and Sirtuins and Resveratrol in Medicine (8 papers). Motoi Kanagawa is often cited by papers focused on Muscle Physiology and Disorders (41 papers), Ubiquitin and proteasome pathways (11 papers) and Sirtuins and Resveratrol in Medicine (8 papers). Motoi Kanagawa collaborates with scholars based in Japan, United States and United Kingdom. Motoi Kanagawa's co-authors include Tatsushi Toda, Kevin P. Campbell, Daniel E. Michele, Kazuhiro Kobayashi, Jakob S. Satz, Steven A. Moore, Fumiaki Saito, Michael B. A. Oldstone, Stefan Kunz and Ichizo Nishino and has published in prestigious journals such as Nature, New England Journal of Medicine and Cell.

In The Last Decade

Motoi Kanagawa

69 papers receiving 3.6k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Post-translational disruption of dystroglycan–ligand interactions in congenital muscular dystrophies

2002 632 citations Daniel E. Michele, Motoi Kanagawa et al. Nature profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Motoi Kanagawa Japan	28	2.9k	794	693	493	446	73	3.6k
Louise V.B. Anderson United Kingdom	31	2.7k 0.9×	844 1.1×	773 1.1×	467 0.9×	233 0.5×	53	3.0k
Stephen J. Crocker United States	34	2.0k 0.7×	903 1.1×	470 0.7×	648 1.3×	220 0.5×	72	4.1k
Julien Fauré France	28	2.5k 0.9×	260 0.3×	1.1k 1.5×	457 0.9×	254 0.6×	82	3.6k
Stefano Biffo Italy	34	3.9k 1.3×	644 0.8×	316 0.5×	259 0.5×	556 1.2×	105	5.4k
Bruce Carter United States	35	2.3k 0.8×	2.3k 2.9×	375 0.5×	345 0.7×	301 0.7×	63	4.5k
Elke Lütjen-Drecoll Germany	48	2.7k 0.9×	503 0.6×	935 1.3×	454 0.9×	319 0.7×	150	6.6k
Toshifumi Tsukahara Japan	32	3.5k 1.2×	429 0.5×	608 0.9×	480 1.0×	276 0.6×	104	4.1k
Isabelle Richard France	38	4.4k 1.5×	1.3k 1.6×	1.6k 2.3×	596 1.2×	646 1.4×	148	5.4k
Atsuro Chiba Japan	20	1.0k 0.4×	1.9k 2.3×	455 0.7×	247 0.5×	113 0.3×	55	3.2k
Paola Podini Italy	30	1.9k 0.7×	630 0.8×	780 1.1×	291 0.6×	128 0.3×	58	3.0k

Countries citing papers authored by Motoi Kanagawa

Since Specialization

Citations

This map shows the geographic impact of Motoi Kanagawa's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Motoi Kanagawa with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Motoi Kanagawa more than expected).

Fields of papers citing papers by Motoi Kanagawa

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Motoi Kanagawa. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Motoi Kanagawa. The network helps show where Motoi Kanagawa may publish in the future.

Co-authorship network of co-authors of Motoi Kanagawa

This figure shows the co-authorship network connecting the top 25 collaborators of Motoi Kanagawa. A scholar is included among the top collaborators of Motoi Kanagawa based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Motoi Kanagawa. Motoi Kanagawa is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Nakano, Hiroko, Pei-Chieng Cha, Yukio Ando, et al.. (2025). Regulation of MCCC1 expression by a Parkinson’s disease-associated intronic variant: implications for pathogenesis. Journal of Human Genetics. 70(7). 371–374. 1 indexed citations

Manya, Hiroshi & Motoi Kanagawa. (2025). Defective O-mannosyl glycan synthesis in dystroglycanopathies: pathogenesis and therapeutic frontiers. Journal of Human Genetics.

Yamada, Yuji, et al.. (2023). Chain-specificity of laminin α1-5 LG45 modules in the recognition of carbohydrate-linked receptors and intramolecular binding. Scientific Reports. 13(1). 10430–10430. 2 indexed citations

Imae, Rieko, Hiroshi Manya, Kazuhiro Kobayashi, et al.. (2022). CDP-ribitol prodrug treatment ameliorates ISPD-deficient muscular dystrophy mouse model. Nature Communications. 13(1). 1847–1847. 15 indexed citations

Morioka, Shigefumi, Hirofumi Sakaguchi, Mariko Taniguchi‐Ikeda, et al.. (2020). Congenital hearing impairment associated with peripheral cochlear nerve dysmyelination in glycosylation-deficient muscular dystrophy. PLoS Genetics. 16(5). e1008826–e1008826. 8 indexed citations

Kuwabara, N., Rieko Imae, Hiroshi Manya, et al.. (2020). Crystal structures of fukutin-related protein (FKRP), a ribitol-phosphate transferase related to muscular dystrophy. Nature Communications. 11(1). 303–303. 23 indexed citations

Kanagawa, Motoi. (2019). Myo-Glyco disease Biology: Genetic Myopathies Caused by Abnormal Glycan Synthesis and Degradation. Journal of Neuromuscular Diseases. 6(2). 175–187. 4 indexed citations

Orlandi, Cesare, Yoshihiro Omori, Yuchen Wang, et al.. (2018). Transsynaptic Binding of Orphan Receptor GPR179 to Dystroglycan-Pikachurin Complex Is Essential for the Synaptic Organization of Photoreceptors. Cell Reports. 25(1). 130–145.e5. 56 indexed citations

Kamizaki, Koki, Ryosuke Doi, Makoto Hayashi, et al.. (2017). The Ror1 receptor tyrosine kinase plays a critical role in regulating satellite cell proliferation during regeneration of injured muscle. Journal of Biological Chemistry. 292(38). 15939–15951. 23 indexed citations

10.

Kanagawa, Motoi, Kazuhiro Kobayashi, Michiko Tajiri, et al.. (2016). Identification of a Post-translational Modification with Ribitol-Phosphate and Its Defect in Muscular Dystrophy. Cell Reports. 14(9). 2209–2223. 163 indexed citations

11.

Kanagawa, Motoi. (2014). Dystroglycan Glycosylation and Its Involvement in Muscular Dystrophy. Trends in Glycoscience and Glycotechnology. 26(149). 41–57. 4 indexed citations

12.

Ogawa, Mitsutaka, Yoshiaki Nakayama, Akira Kurosaka, et al.. (2013). GTDC2 modifies O-mannosylated α-dystroglycan in the endoplasmic reticulum to generate N-acetyl glucosamine epitopes reactive with CTD110.6 antibody. Biochemical and Biophysical Research Communications. 440(1). 88–93. 34 indexed citations

13.

Hara, Yuko, Burcu Balcı-Hayta, Takako Yoshida‐Moriguchi, et al.. (2011). BRIEF REPORT A Dystroglycan Mutation Associated with Limb-Girdle Muscular Dystrophy. UCL Discovery (University College London).

14.

Hara, Yuji, Burcu Balcı-Hayta, Takako Yoshida‐Moriguchi, et al.. (2011). A Dystroglycan Mutation Associated with Limb-Girdle Muscular Dystrophy. New England Journal of Medicine. 364(10). 939–946. 178 indexed citations

15.

Taniguchi‐Ikeda, Mariko, Kazuhiro Kobayashi, Motoi Kanagawa, et al.. (2011). Pathogenic exon-trapping by SVA retrotransposon and rescue in Fukuyama muscular dystrophy. Nature. 478(7367). 127–131. 132 indexed citations

16.

Han, Renzhi, Motoi Kanagawa, Takako Yoshida‐Moriguchi, et al.. (2009). Basal lamina strengthens cell membrane integrity via the laminin G domain-binding motif of α-dystroglycan. Proceedings of the National Academy of Sciences. 106(31). 12573–12579. 122 indexed citations

17.

Kanagawa, Motoi, Tomohiro Chiyonobu, Satoshi Takeda, et al.. (2008). Residual laminin-binding activity and enhanced dystroglycan glycosylation by LARGE in novel model mice to dystroglycanopathy. Human Molecular Genetics. 18(4). 621–631. 61 indexed citations

18.

Nishimura, Hidekazu, et al.. (2008). RESPONSE CONTROL PERFORMANCE OF SEMI-ACTIVE ISOLATION SYSTEM USING THE GS CONTROLE FOR A MULTI-DEGREE-OF-FREEDOM STRUCTURE WITH MAGNETO-RHEOLOGICAL FLUID DAMPER. Journal of Structural and Construction Engineering (Transactions of AIJ). 73(628). 875–882. 2 indexed citations

19.

Kanagawa, Motoi & Tatsushi Toda. (2006). The genetic and molecular basis of muscular dystrophy: roles of cell–matrix linkage in the pathogenesis. Journal of Human Genetics. 51(11). 915–926. 102 indexed citations

20.

Fujitani, Naoki, Motoi Kanagawa, Tomoyasu Aizawa, et al.. (2003). Structure determination and conformational change induced by tyrosine phosphorylation of the N-terminal domain of the α-chain of pig gastric H+/K+-ATPase. Biochemical and Biophysical Research Communications. 300(1). 223–229. 15 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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