Michihiro Imamura

3.0k total citations
51 papers, 2.4k citations indexed

About

Michihiro Imamura is a scholar working on Molecular Biology, Cell Biology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Michihiro Imamura has authored 51 papers receiving a total of 2.4k indexed citations (citations by other indexed papers that have themselves been cited), including 43 papers in Molecular Biology, 18 papers in Cell Biology and 11 papers in Cellular and Molecular Neuroscience. Recurrent topics in Michihiro Imamura's work include Muscle Physiology and Disorders (33 papers), Muscle metabolism and nutrition (7 papers) and Cardiomyopathy and Myosin Studies (7 papers). Michihiro Imamura is often cited by papers focused on Muscle Physiology and Disorders (33 papers), Muscle metabolism and nutrition (7 papers) and Cardiomyopathy and Myosin Studies (7 papers). Michihiro Imamura collaborates with scholars based in Japan, United States and United Kingdom. Michihiro Imamura's co-authors include Shin’ichi Takeda, Takeshi Endo, Eijiro Ozawa, Kiyoko Fukami, Τοmoh Masaki, T Takenawa, Takao Masaki, S. Noguchi, Masashi Yanagisawa and Y. Hamada and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and The Journal of Cell Biology.

In The Last Decade

Michihiro Imamura

49 papers receiving 2.4k citations

Peers

Michihiro Imamura
Chie Matsuda Japan
Dimple Bansal United States
Rita Barresi United Kingdom
Abigail McElhinny United States
Rita Barresi Italy
Aikaterini Kontrogianni‐Konstantopoulos United States
Louise V.B. Anderson United Kingdom
Nathalie Bourg France
Michael Hesse Germany
Eduard Gallardo Spain
Chie Matsuda Japan
Michihiro Imamura
Citations per year, relative to Michihiro Imamura Michihiro Imamura (= 1×) peers Chie Matsuda

Countries citing papers authored by Michihiro Imamura

Since Specialization
Citations

This map shows the geographic impact of Michihiro Imamura's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michihiro Imamura with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michihiro Imamura more than expected).

Fields of papers citing papers by Michihiro Imamura

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Michihiro Imamura. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michihiro Imamura. The network helps show where Michihiro Imamura may publish in the future.

Co-authorship network of co-authors of Michihiro Imamura

This figure shows the co-authorship network connecting the top 25 collaborators of Michihiro Imamura. A scholar is included among the top collaborators of Michihiro Imamura based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Michihiro Imamura. Michihiro Imamura is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Imamura, Michihiro, et al.. (2024). Severe cardiac and skeletal manifestations in DMD-edited microminipigs: an advanced surrogate for Duchenne muscular dystrophy. Communications Biology. 7(1). 523–523. 5 indexed citations
2.
Imamura, Michihiro, Hirokazu Matsumoto, Hideyuki Mannen, Shin’ichi Takeda, & Yoshitsugu Aoki. (2024). The R436Q missense mutation in WWP1 disrupts autoinhibition of its E3 ubiquitin ligase activity, leading to self-degradation and loss of function. In Vitro Cellular & Developmental Biology - Animal. 60(7). 771–780.
3.
Kuniishi, Hiroshi, Kazuhisa Sakai, Yuta Fukushima, et al.. (2022). Brain Dp140 alters glutamatergic transmission and social behaviour in the mdx52 mouse model of Duchenne muscular dystrophy. Progress in Neurobiology. 216. 102288–102288. 24 indexed citations
4.
Mamchaoui, Kamel, M.K. Tsoumpra, Rika Maruyama, et al.. (2021). Immortalized Canine Dystrophic Myoblast Cell Lines for Development of Peptide-Conjugated Splice-Switching Oligonucleotides. Nucleic Acid Therapeutics. 31(2). 172–181. 10 indexed citations
5.
Shiba, Naoko, Daigo Miyazaki, Takahiro Yoshizawa, et al.. (2015). Differential roles of MMP-9 in early and late stages of dystrophic muscles in a mouse model of Duchenne muscular dystrophy. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1852(10). 2170–2182. 22 indexed citations
6.
Imamura, Michihiro, Akinori Nakamura, Hideyuki Mannen, & Shin’ichi Takeda. (2015). Characterization of WWP1 protein expression in skeletal muscle of muscular dystrophy chickens. The Journal of Biochemistry. 159(2). 171–179. 15 indexed citations
7.
Yoshioka, Hiroki, et al.. (2006). ζ-Sarcoglycan is a functional homologue of γ-sarcoglycan in the formation of the sarcoglycan complex. Experimental Cell Research. 312(11). 2083–2092. 27 indexed citations
8.
Takahashi, Joji, et al.. (2004). The utrophin promoter A drives high expression of the transgenicLacZ gene in liver, testis, colon, submandibular gland, and small intestine. The Journal of Gene Medicine. 7(2). 237–248. 11 indexed citations
9.
Fukada, So‐ichiro, Saito Higuchi, Masashi Segawa, et al.. (2004). Purification and cell-surface marker characterization of quiescent satellite cells from murine skeletal muscle by a novel monoclonal antibody. Experimental Cell Research. 296(2). 245–255. 149 indexed citations
10.
Mizuno, Yuji, Jeffrey R. Guyon, Simon C. Watkins, et al.. (2004). β‐synemin localizes to regions of high stress in human skeletal myofibers. Muscle & Nerve. 30(3). 337–346. 25 indexed citations
11.
Furuya, Akiko, Kenya Shitara, Michihiro Imamura, et al.. (2004). α1-Syntrophin Modulates Turnover of ABCA1. Journal of Biological Chemistry. 279(15). 15091–15095. 94 indexed citations
12.
Sasaoka, Toshikuni, Michihiro Imamura, Kenji Araishi, et al.. (2003). Pathological analysis of muscle hypertrophy and degeneration in muscular dystrophy in γ-sarcoglycan-deficient mice. Neuromuscular Disorders. 13(3). 193–206. 30 indexed citations
13.
Kishi, Hiroko, Takashi Mikawa, Minoru Seto, et al.. (2000). Stable Transfectants of Smooth Muscle Cell Line Lacking the Expression of Myosin Light Chain Kinase and Their Characterization with Respect to the Actomyosin System. Journal of Biological Chemistry. 275(2). 1414–1420. 33 indexed citations
14.
Imamura, Michihiro. (2000). A sarcoglycan-dystroglycan complex anchors Dp116 and utrophin in the peripheral nervous system. Human Molecular Genetics. 9(20). 3091–3100. 71 indexed citations
15.
Noguchi, S., et al.. (1999). Developmental Expression of Sarcoglycan Gene Products in Cultured Myocytes. Biochemical and Biophysical Research Communications. 262(1). 88–93. 34 indexed citations
16.
Imamura, Michihiro. (1996). Biological Basis of Angiogenesis and Vascular Remodeling. Extracellular matrix components and angiogenesis.. Folia Pharmacologica Japonica. 107(3). 153–160. 1 indexed citations
17.
Kato, Masaki, Takuya Sasaki, Takeshi Ohya, et al.. (1996). Physical and Functional Interaction of Rabphilin-3A with α-Actinin. Journal of Biological Chemistry. 271(50). 31775–31778. 101 indexed citations
18.
Imamura, Michihiro & Τοmoh Masaki. (1994). Identification of a 115-kDa Protein from Muscle Tissues: Expression of a Novel Nonmuscle α-Actinin in Vascular Endothelial Cells. Experimental Cell Research. 211(2). 380–390. 11 indexed citations
19.
Imamura, Michihiro, Takeshi Sakurai, Yoshikatsu Ogawa, et al.. (1994). Molecular cloning of low‐Ca2+‐sensitive‐type non‐muscle α‐actinin. European Journal of Biochemistry. 223(2). 395–401. 19 indexed citations
20.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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