Shigeo Murata
- Molecular Biology top 0.2%
- Epidemiology top 0.2%
- Cell Biology top 0.1%
- Immunology top 0.5%
- Oncology top 1%
- Co-authors
- Keiji TanakaTomoki ChibaM. TachiyaYasuo UchiyamaMasaaki KomatsuJunichi IwataTakashi UenoEiki Kominami
- Topics
- Ubiquitin and proteasome pathways (93 papers)Endoplasmic Reticulum Stress and Disease (35 papers)Photochemistry and Electron Transfer Studies (26 papers)
- Cited by
- Cell BiologyAgingEpidemiology
- Partner nations
- JapanUnited StatesGermany
In The Last Decade
Shigeo Murata
211 papers receiving 18.9k citations
Hit Papers
Peers
Comparison fields: 5 of 161
- Molecular Biology 10.2k
- Epidemiology 5.7k
- Cell Biology 3.5k
- Immunology 2.9k
- Oncology 2.4k
Countries citing papers authored by Shigeo Murata
This map shows the geographic impact of Shigeo Murata's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Shigeo Murata with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Shigeo Murata more than expected).
Fields of papers citing papers by Shigeo Murata
This network shows the impact of papers produced by Shigeo Murata. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Shigeo Murata. The network helps show where Shigeo Murata may publish in the future.
Co-authorship network of co-authors of Shigeo Murata
This figure shows the co-authorship network connecting the top 25 collaborators of Shigeo Murata. A scholar is included among the top collaborators of Shigeo Murata based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Shigeo Murata. Shigeo Murata is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 1 | |
| 2 | 1 | |
| 3 | 5 | |
| 4 | 1 | |
| 5 | 25 | |
| 6 | 10 | |
| 7 | 9 | |
| 8 | 45 | |
| 9 | 74 | |
| 10 | 154 | |
| 11 | 70 | |
| 12 | 219 | |
| 13 | 68 | |
| 14 | 23 | |
| 15 | 456 | |
| 16 | 17 | |
| 17 | Impairment of starvation-induced and constitutive autophagy in Atg7 -deficient micebreakdown → | 1941 |
| 18 | 63 | |
| 19 | 7 | |
| 20 | Iodine excretion and prevalence of thyroid dysfunction in the Western Part of Germany: results of the Heinz Nixdorf Recall study | 1 |
About Shigeo Murata
Shigeo Murata is a scholar working on Physical and Theoretical Chemistry, Cell Biology and Immunology, having authored 215 papers that have together received 19.1k indexed citations. Recurring topics across this work include Ubiquitin and proteasome pathways (93 papers), Endoplasmic Reticulum Stress and Disease (35 papers) and Photochemistry and Electron Transfer Studies (26 papers). The work is most often cited by research in Cell Biology (3.5k citations), Aging (355 citations) and Epidemiology (5.7k citations). Shigeo Murata has collaborated with scholars based in Japan, United States and Germany. Frequent co-authors include Keiji Tanaka, Tomoki Chiba, M. Tachiya, Yasuo Uchiyama, Masaaki Komatsu, Junichi Iwata, Takashi Ueno, Eiki Kominami, Isei Tanida and Satoshi Waguri. Their work appears in journals such as Nature, Science and Cell.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.