Jun Hamazaki
- Molecular Biology top 5%
- Cell Biology top 2%
- Epidemiology top 10%
- Oncology top 10%
- Genetics top 10%
- Co-authors
- Shigeo MurataKeiji TanakaHideki YashirodaTohru NatsumeShun‐ichiro IemuraShoshiro HirayamaKatsuhiro SasakiMasayuki Miura
- Topics
- Ubiquitin and proteasome pathways (27 papers)Endoplasmic Reticulum Stress and Disease (12 papers)Genetics and Neurodevelopmental Disorders (6 papers)
- Cited by
- AgingCell BiologyMolecular Biology
- Partner nations
- JapanUnited StatesCanada
In The Last Decade
Jun Hamazaki
32 papers receiving 1.7k citations
Peers
Comparison fields: 5 of 90
- Molecular Biology 1.5k
- Cell Biology 541
- Epidemiology 407
- Oncology 286
- Genetics 207
Countries citing papers authored by Jun Hamazaki
This map shows the geographic impact of Jun Hamazaki's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jun Hamazaki with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jun Hamazaki more than expected).
Fields of papers citing papers by Jun Hamazaki
This network shows the impact of papers produced by Jun Hamazaki. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jun Hamazaki. The network helps show where Jun Hamazaki may publish in the future.
Co-authorship network of co-authors of Jun Hamazaki
This figure shows the co-authorship network connecting the top 25 collaborators of Jun Hamazaki. A scholar is included among the top collaborators of Jun Hamazaki based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jun Hamazaki. Jun Hamazaki is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 1 | |
| 2 | 10 | |
| 3 | 4 | |
| 4 | 11 | |
| 5 | 29 | |
| 6 | 6 | |
| 7 | 1 | |
| 8 | 24 | |
| 9 | 10 | |
| 10 | 61 | |
| 11 | 13 | |
| 12 | 41 | |
| 13 | 24 | |
| 14 | 68 | |
| 15 | 45 | |
| 16 | 212 | |
| 17 | 37 | |
| 18 | 219 | |
| 19 | 98 | |
| 20 | 209 |
About Jun Hamazaki
Jun Hamazaki is a scholar working on Cell Biology, Molecular Biology and Immunology, having authored 32 papers that have together received 1.7k indexed citations. Recurring topics across this work include Ubiquitin and proteasome pathways (27 papers), Endoplasmic Reticulum Stress and Disease (12 papers) and Genetics and Neurodevelopmental Disorders (6 papers). The work is most often cited by research in Aging (94 citations), Cell Biology (541 citations) and Molecular Biology (1.5k citations). Jun Hamazaki has collaborated with scholars based in Japan, United States and Canada. Frequent co-authors include Shigeo Murata, Keiji Tanaka, Hideki Yashiroda, Tohru Natsume, Shun‐ichiro Iemura, Shoshiro Hirayama, Katsuhiro Sasaki, Masayuki Miura, Erina Kuranaga and Takeyasu Tomioka. Their work appears in journals such as Cell, Journal of Biological Chemistry and Journal of Clinical Investigation.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.