Sarah R. Weiler
- Aging top 2%
- Physiology top 5%
- Telomeres, Telomerase, and Senescence 3
- Biotechnology top 10%
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- Immune cells in cancer 3
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- Retinoids in leukemia and cellular processes 4
- DNA Repair Mechanisms 1
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- Acute Myeloid Leukemia Research 3
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- Cytokine Signaling Pathways and Interactions 3
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- Genetic Syndromes and Imprinting 1
- Genomics and Rare Diseases 1
- Co-authors
- Ronald A. DePinhoK. Lenhard RudolphLynda ChinRoger A. GreenbergJonathan R. KellerMaja Katrin TietzeShridar GanesanMaja Artandi
- Cited by
- AgingPhysiologyBiotechnology
- Journals
- Proceedings of the National Academy of Sciences (2 papers)Journal of Biological Chemistry (2 papers)Nature Genetics (1 paper)
- Partner nations
- United StatesNetherlandsGermany
In The Last Decade
Sarah R. Weiler
16 papers receiving 914 citations
Peers
Comparison fields: 5 of 93
- Aging 110
- Physiology 504
- Biotechnology 81
- Immunology 177
- Molecular Biology 512
Countries citing papers authored by Sarah R. Weiler
This map shows the geographic impact of Sarah R. Weiler's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sarah R. Weiler with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sarah R. Weiler more than expected).
Fields of papers citing papers by Sarah R. Weiler
This network shows the impact of papers produced by Sarah R. Weiler. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sarah R. Weiler. The network helps show where Sarah R. Weiler may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Sarah R. Weiler, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2021 | 16 | |
| 2 | 2019 | 14 | |
| 3 | 2015 | 15 | |
| 4 | The Scalable Collaborative Infrastructure for a Learning Health System. | 2015 | 1 |
| 5 | 2015 | 2 | |
| 6 | 2004 | 17 | |
| 7 | 2002 | 240 | |
| 8 | 2001 | 101 | |
| 9 | 2000 | 261 | |
| 10 | 2000 | 21 | |
| 11 | 1999 | 12 | |
| 12 | 1999 | 30 | |
| 13 | 1998 | 62 | |
| 14 | 1997 | 41 | |
| 15 | 1996 | 22 | |
| 16 | 1995 | 66 |
About Sarah R. Weiler
Sarah R. Weiler is a scholar working on Aging, Hematology and Genetics, having authored 16 papers that have together received 921 indexed citations. Recurring topics across this work include Retinoids in leukemia and cellular processes (4 papers), Acute Myeloid Leukemia Research (3 papers), Immune cells in cancer (3 papers), Telomeres, Telomerase, and Senescence (3 papers), Cytokine Signaling Pathways and Interactions (3 papers), Genetic Syndromes and Imprinting (1 paper), Genomics and Rare Diseases (1 paper) and DNA Repair Mechanisms (1 paper). The work is most often cited by research in Aging (110 citations), Physiology (504 citations) and Biotechnology (81 citations). Sarah R. Weiler has collaborated with scholars based in United States, Netherlands and Germany. Frequent co-authors include Ronald A. DePinho, K. Lenhard Rudolph, Lynda Chin, Roger A. Greenberg, Jonathan R. Keller, Maja Katrin Tietze, Shridar Ganesan, Maja Artandi, Sandy Chang and James W. Horner. Their work appears in journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Nature Genetics.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.