Sonja Schaetzlein

1.5k total citations
15 papers, 1.1k citations indexed

About

Sonja Schaetzlein is a scholar working on Molecular Biology, Physiology and Pathology and Forensic Medicine. According to data from OpenAlex, Sonja Schaetzlein has authored 15 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 8 papers in Physiology and 4 papers in Pathology and Forensic Medicine. Recurrent topics in Sonja Schaetzlein's work include Telomeres, Telomerase, and Senescence (8 papers), DNA Repair Mechanisms (7 papers) and Genetic factors in colorectal cancer (4 papers). Sonja Schaetzlein is often cited by papers focused on Telomeres, Telomerase, and Senescence (8 papers), DNA Repair Mechanisms (7 papers) and Genetic factors in colorectal cancer (4 papers). Sonja Schaetzlein collaborates with scholars based in United States, Germany and Cuba. Sonja Schaetzlein's co-authors include K. Lenhard Rudolph, Michael P. Manns, Meta W. Djojosubroto, André Lechel, Zhenyu Ju, Jan Buer, Peter Schirmacher, Anna Stępczyńska, Winfried Edelmann and Martina Dorsch and has published in prestigious journals such as Cell, Proceedings of the National Academy of Sciences and Nature Medicine.

In The Last Decade

Sonja Schaetzlein

14 papers receiving 1.1k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Sonja Schaetzlein United States 11 706 564 180 173 115 15 1.1k
Miguel Foronda Spain 15 804 1.1× 459 0.8× 139 0.8× 182 1.1× 155 1.3× 20 1.3k
Sarah R. Weiler United States 13 512 0.7× 504 0.9× 155 0.9× 110 0.6× 72 0.6× 16 921
Meta W. Djojosubroto Germany 7 402 0.6× 357 0.6× 123 0.7× 94 0.5× 58 0.5× 8 680
Marta Garcı́a-Cao Spain 9 1.3k 1.8× 829 1.5× 395 2.2× 181 1.0× 176 1.5× 11 1.7k
Lorel M. Colgin Australia 11 797 1.1× 774 1.4× 104 0.6× 128 0.7× 92 0.8× 13 1.1k
Giulia Celli United States 13 1.4k 2.0× 777 1.4× 246 1.4× 113 0.7× 93 0.8× 16 1.8k
Michele F. Haughton United Kingdom 16 762 1.1× 706 1.3× 289 1.6× 170 1.0× 93 0.8× 18 1.2k
H W Sharma United States 8 805 1.1× 432 0.8× 350 1.9× 50 0.3× 212 1.8× 9 1.3k
Rachel Litman Flynn United States 12 1.3k 1.8× 540 1.0× 271 1.5× 54 0.3× 162 1.4× 18 1.5k
Nina S. Heiss Germany 17 1.1k 1.6× 736 1.3× 86 0.5× 48 0.3× 97 0.8× 21 1.5k

Countries citing papers authored by Sonja Schaetzlein

Since Specialization
Citations

This map shows the geographic impact of Sonja Schaetzlein's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sonja Schaetzlein with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sonja Schaetzlein more than expected).

Fields of papers citing papers by Sonja Schaetzlein

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Sonja Schaetzlein. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sonja Schaetzlein. The network helps show where Sonja Schaetzlein may publish in the future.

Co-authorship network of co-authors of Sonja Schaetzlein

This figure shows the co-authorship network connecting the top 25 collaborators of Sonja Schaetzlein. A scholar is included among the top collaborators of Sonja Schaetzlein based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Sonja Schaetzlein. Sonja Schaetzlein is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

15 of 15 papers shown
1.
Will, Britta, Thomas O. Vogler, Boris Bartholdy, et al.. (2015). Minimal PU.1 reduction induces a preleukemic state and promotes development of acute myeloid leukemia. Nature Medicine. 21(10). 1172–1181. 81 indexed citations
2.
Will, Britta, Thomas O. Vogler, Boris Bartholdy, et al.. (2015). Minimal Reduction of PU.1 Is Sufficient to Induce a Preleukemic State and Promote Development of Acute Myeloid Leukemia. Blood. 126(23). 305–305.
3.
Tosti, Elena, Sonja Schaetzlein, Hyun Soo Kim, et al.. (2014). Evolutionarily conserved genetic interactions with budding and fission yeast MutS identify orthologous relationships in mismatch repair-deficient cancer cells. Genome Medicine. 6(9). 68–68. 10 indexed citations
4.
Oers, Johanna M.M. van, Richard Chahwan, Cameron Smith, et al.. (2013). The MutSβ complex is a modulator of p53-driven tumorigenesis through its functions in both DNA double-strand break repair and mismatch repair. Oncogene. 33(30). 3939–3946. 36 indexed citations
5.
Schaetzlein, Sonja, Richard Chahwan, Elena Avdievich, et al.. (2013). MammalianExo1encodes both structural and catalytic functions that play distinct roles in essential biological processes. Proceedings of the National Academy of Sciences. 110(27). E2470–9. 55 indexed citations
6.
Bothmer, Anne, Philipp C. Rommel, Anna Gazumyan, et al.. (2012). Mechanism of DNA resection during intrachromosomal recombination and immunoglobulin class switching. The Journal of Cell Biology. 199(7). i11–i11. 1 indexed citations
7.
Bothmer, Anne, Philipp C. Rommel, Anna Gazumyan, et al.. (2012). Mechanism of DNA resection during intrachromosomal recombination and immunoglobulin class switching. The Journal of Experimental Medicine. 210(1). 115–123. 46 indexed citations
8.
Schaetzlein, Sonja, Zhenyu Ju, André Lechel, et al.. (2007). Exonuclease-1 Deletion Impairs DNA Damage Signaling and Prolongs Lifespan of Telomere-Dysfunctional Mice. Cell. 131(1). 190–190. 2 indexed citations
9.
Schaetzlein, Sonja, Zhenyu Ju, André Lechel, et al.. (2007). Exonuclease-1 Deletion Impairs DNA Damage Signaling and Prolongs Lifespan of Telomere-Dysfunctional Mice. Cell. 130(5). 863–877. 115 indexed citations
10.
Choudhury, Aaheli Roy, Zhenyu Ju, Meta W. Djojosubroto, et al.. (2006). Cdkn1a deletion improves stem cell function and lifespan of mice with dysfunctional telomeres without accelerating cancer formation. Nature Genetics. 39(1). 99–105. 337 indexed citations
11.
Djojosubroto, Meta W., Allison C. Chin, Ning Go, et al.. (2005). Telomerase Antagonists GRN163 and GRN163L Inhibit Tumor Growth and Increase Chemosensitivity of Human Hepatoma *. Hepatology. 42(5). 1127–1136. 111 indexed citations
12.
Lechel, André, Satyanarayana Ande, Zhenyu Ju, et al.. (2005). The cellular level of telomere dysfunction determines induction of senescence or apoptosis in vivo. EMBO Reports. 6(3). 275–281. 71 indexed citations
13.
Schaetzlein, Sonja, Andrea Lucas‐Hahn, Erika Lemme, et al.. (2004). Telomere length is reset during early mammalian embryogenesis. Proceedings of the National Academy of Sciences. 101(21). 8034–8038. 170 indexed citations
14.
Schaetzlein, Sonja & K. Lenhard Rudolph. (2004). Telomere length regulation during cloning, embryogenesis and ageing. Reproduction Fertility and Development. 17(2). 85–96. 24 indexed citations
15.
Ande, Satyanarayana, Roger A. Greenberg, Sonja Schaetzlein, et al.. (2004). Mitogen Stimulation Cooperates with Telomere Shortening To Activate DNA Damage Responses and Senescence Signaling. Molecular and Cellular Biology. 24(12). 5459–5474. 58 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact

Rankless by CCL
2026