Saba Aïd

2.8k total citations
28 papers, 2.2k citations indexed

About

Saba Aïd is a scholar working on Physiology, Neurology and Molecular Biology. According to data from OpenAlex, Saba Aïd has authored 28 papers receiving a total of 2.2k indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Physiology, 10 papers in Neurology and 8 papers in Molecular Biology. Recurrent topics in Saba Aïd's work include Neuroinflammation and Neurodegeneration Mechanisms (10 papers), Alzheimer's disease research and treatments (8 papers) and Inflammatory mediators and NSAID effects (7 papers). Saba Aïd is often cited by papers focused on Neuroinflammation and Neurodegeneration Mechanisms (10 papers), Alzheimer's disease research and treatments (8 papers) and Inflammatory mediators and NSAID effects (7 papers). Saba Aïd collaborates with scholars based in France, United States and Canada. Saba Aïd's co-authors include Francesca Bosetti, Sangho Choi, Martin Holzenberger, Sylvie Vancassel, Monique Lavialle, Philippe Guesnet, Zayna Chaker, Robert Langenbach, Hyung‐Wook Kim and Sharon H. Jackson and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Neuroscience and SHILAP Revista de lepidopterología.

In The Last Decade

Saba Aïd

27 papers receiving 2.2k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Saba Aïd France 20 759 669 658 344 340 28 2.2k
Maria Antonietta Ajmone‐Cat Italy 33 1.1k 1.5× 883 1.3× 437 0.7× 167 0.5× 241 0.7× 55 2.7k
Leon M. Tai United States 30 818 1.1× 908 1.4× 1.6k 2.5× 263 0.8× 185 0.5× 63 2.9k
Juliet M. Taylor Australia 23 1.0k 1.4× 1.3k 2.0× 600 0.9× 129 0.4× 237 0.7× 40 2.9k
Elisa Mitiko Kawamoto Brazil 29 465 0.6× 922 1.4× 661 1.0× 120 0.3× 248 0.7× 67 2.6k
Mercedes Lasaga Argentina 29 431 0.6× 585 0.9× 490 0.7× 276 0.8× 174 0.5× 96 2.3k
Antonietta Bernardo Italy 28 677 0.9× 1.1k 1.7× 519 0.8× 122 0.4× 106 0.3× 50 2.2k
Carlos Beas‐Zárate Mexico 34 561 0.7× 1.2k 1.8× 1.1k 1.7× 348 1.0× 268 0.8× 125 3.6k
Sofie Lautrup Norway 15 562 0.7× 1.7k 2.6× 1.3k 2.0× 139 0.4× 363 1.1× 25 3.7k
Sebastián Jiménez Spain 25 869 1.1× 612 0.9× 1.2k 1.8× 197 0.6× 178 0.5× 37 2.3k
Małgorzata Chalimoniuk Poland 28 573 0.8× 850 1.3× 871 1.3× 115 0.3× 135 0.4× 100 2.9k

Countries citing papers authored by Saba Aïd

Since Specialization
Citations

This map shows the geographic impact of Saba Aïd's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Saba Aïd with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Saba Aïd more than expected).

Fields of papers citing papers by Saba Aïd

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Saba Aïd. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Saba Aïd. The network helps show where Saba Aïd may publish in the future.

Co-authorship network of co-authors of Saba Aïd

This figure shows the co-authorship network connecting the top 25 collaborators of Saba Aïd. A scholar is included among the top collaborators of Saba Aïd based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Saba Aïd. Saba Aïd is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
2.
Shapira, Ilana, Eman M. Abbas, Boaz Styr, et al.. (2022). IGF-1 receptor regulates upward firing rate homeostasis via the mitochondrial calcium uniporter. Proceedings of the National Academy of Sciences. 119(33). e2121040119–e2121040119. 13 indexed citations
3.
Sánchez-Alavez, Manuel, William Nguyen, Simone Mori, et al.. (2017). Insulin-like growth factor 1 receptor regulates hypothermia during calorie restriction. Proceedings of the National Academy of Sciences. 114(36). 9731–9736. 27 indexed citations
4.
Gontier, Géraldine, et al.. (2017). The Alzheimer’s disease transcriptome mimics the neuroprotective signature of IGF-1 receptor-deficient neurons. Brain. 140(7). 2012–2027. 59 indexed citations
5.
Dansokho, Cira, Saba Aïd, Cécile Toly‐Ndour, et al.. (2016). Regulatory T cells delay disease progression in Alzheimer-like pathology. Brain. 139(4). 1237–1251. 270 indexed citations
6.
Gontier, Géraldine, et al.. (2015). Blocking IGF Signaling in Adult Neurons Alleviates Alzheimer's Disease Pathology through Amyloid-β Clearance. Journal of Neuroscience. 35(33). 11500–11513. 121 indexed citations
7.
Dansokho, Cira, Cécile Toly‐Ndour, Saba Aïd, et al.. (2014). Beneficial role of regulatory T cells in a mouse model of Alzheimer's disease. Journal of Neuroimmunology. 275(1-2). 124–124. 2 indexed citations
8.
Choi, Sangho, Saba Aïd, Hyung‐Wook Kim, Sharon H. Jackson, & Francesca Bosetti. (2011). Inhibition of NADPH oxidase promotes alternative and anti‐inflammatory microglial activation during neuroinflammation. Journal of Neurochemistry. 120(2). 292–301. 191 indexed citations
9.
Minami, Sakura, Elkhansa Sidahmed, Saba Aïd, et al.. (2010). Therapeutic versus neuroinflammatory effects of passive immunization is dependent on Aβ/amyloid burden in a transgenic mouse model of Alzheimer's disease. Journal of Neuroinflammation. 7(1). 57–57. 19 indexed citations
10.
Aïd, Saba, et al.. (2010). Neuronal overexpression of cyclooxygenase-2 does not alter the neuroinflammatory response during brain innate immune activation. Neuroscience Letters. 478(3). 113–118. 7 indexed citations
11.
Aïd, Saba & Francesca Bosetti. (2010). Targeting cyclooxygenases-1 and -2 in neuroinflammation: Therapeutic implications. Biochimie. 93(1). 46–51. 190 indexed citations
12.
Choi, Sung‐Hyuk, Saba Aïd, Uimook Choi, & Francesca Bosetti. (2009). Cyclooxygenases-1 and -2 differentially modulate leukocyte recruitment into the inflamed brain. The Pharmacogenomics Journal. 10(5). 448–457. 50 indexed citations
13.
Choi, Sangho, Saba Aïd, & Francesca Bosetti. (2009). The distinct roles of cyclooxygenase-1 and -2 in neuroinflammation: implications for translational research. Trends in Pharmacological Sciences. 30(4). 174–181. 318 indexed citations
14.
Aïd, Saba, Robert Langenbach, & Francesca Bosetti. (2008). Neuroinflammatory response to lipopolysaccharide is exacerbated in mice genetically deficient in cyclooxygenase-2. Journal of Neuroinflammation. 5(1). 17–17. 122 indexed citations
15.
16.
Aïd, Saba & Francesca Bosetti. (2007). Gene expression of cyclooxygenase-1 and Ca2+-independent phospholipase A2 is altered in rat hippocampus during normal aging. Brain Research Bulletin. 73(1-3). 108–113. 34 indexed citations
17.
Vancassel, Sylvie, Saba Aïd, Fabien Pifferi, et al.. (2005). Cerebral Asymmetry and Behavioral Lateralization in Rats Chronically Lacking n-3 Polyunsaturated Fatty Acids. Biological Psychiatry. 58(10). 805–811. 25 indexed citations
18.
19.
Alessandri, Jean‐Marc, Philippe Guesnet, Sylvie Vancassel, et al.. (2004). Polyunsaturated fatty acids in the central nervous system: evolution of concepts and nutritional implications throughout life. annales de biologie animale biochimie biophysique. 44(6). 509–538. 233 indexed citations
20.
Aïd, Saba, et al.. (2003). Effect of a diet-induced n-3 PUFA depletion on cholinergic parameters in the rat hippocampus. Journal of Lipid Research. 44(8). 1545–1551. 127 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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