Martin Holzenberger
- Molecular Biology top 2%
- Physiology top 1%
- Endocrinology, Diabetes and Metabolism top 0.5%
- Aging top 0.1%
- Surgery top 5%
- Co-authors
- Joëlle DupontYves Le BoucPatricia LeneuveBertrand DucosPascale CerveraPatrick C. EvenAlain GéloënC. Ronald Kahn
- Topics
- Growth Hormone and Insulin-like Growth Factors (27 papers)Genetics, Aging, and Longevity in Model Organisms (14 papers)Adipose Tissue and Metabolism (12 papers)
- Partner nations
- FranceUnited StatesCanada
In The Last Decade
Martin Holzenberger
85 papers receiving 6.3k citations
Hit Papers
Peers
Comparison fields: 5 of 120
- Molecular Biology 2.9k
- Physiology 1.8k
- Endocrinology, Diabetes and Metabolism 1.5k
- Aging 1.3k
- Surgery 1.1k
Countries citing papers authored by Martin Holzenberger
This map shows the geographic impact of Martin Holzenberger's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Martin Holzenberger with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Martin Holzenberger more than expected).
Fields of papers citing papers by Martin Holzenberger
This network shows the impact of papers produced by Martin Holzenberger. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Martin Holzenberger. The network helps show where Martin Holzenberger may publish in the future.
Co-authorship network of co-authors of Martin Holzenberger
This figure shows the co-authorship network connecting the top 25 collaborators of Martin Holzenberger. A scholar is included among the top collaborators of Martin Holzenberger based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Martin Holzenberger. Martin Holzenberger is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 5 | |
| 2 | 6 | |
| 3 | 8 | |
| 4 | 13 | |
| 5 | 27 | |
| 6 | 66 | |
| 7 | 270 | |
| 8 | 121 | |
| 9 | 79 | |
| 10 | 17 | |
| 11 | 69 | |
| 12 | 9 | |
| 13 | 37 | |
| 14 | 54 | |
| 15 | 34 | |
| 16 | 230 | |
| 17 | 33 | |
| 18 | 138 | |
| 19 | 4 | |
| 20 | 22 |
About Martin Holzenberger
Martin Holzenberger is a scholar working on Aging, Endocrinology, Diabetes and Metabolism and Developmental Neuroscience, having authored 86 papers that have together received 6.4k indexed citations. Recurring topics across this work include Growth Hormone and Insulin-like Growth Factors (27 papers), Genetics, Aging, and Longevity in Model Organisms (14 papers) and Adipose Tissue and Metabolism (12 papers). The work is most often cited by research in Aging (1.3k citations), Endocrinology, Diabetes and Metabolism (1.5k citations) and Endocrine and Autonomic Systems (534 citations). Martin Holzenberger has collaborated with scholars based in France, United States and Canada. Frequent co-authors include Joëlle Dupont, Yves Le Bouc, Patricia Leneuve, Bertrand Ducos, Pascale Cervera, Patrick C. Even, Alain Géloën, C. Ronald Kahn, Saba Aïd and Zayna Chaker. Their work appears in journals such as Nature, Cell and Proceedings of the National Academy of Sciences.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.