Jo El J. Schultz

4.1k total citations
46 papers, 3.4k citations indexed

About

Jo El J. Schultz is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine and Pathology and Forensic Medicine. According to data from OpenAlex, Jo El J. Schultz has authored 46 papers receiving a total of 3.4k indexed citations (citations by other indexed papers that have themselves been cited), including 22 papers in Molecular Biology, 20 papers in Cardiology and Cardiovascular Medicine and 17 papers in Pathology and Forensic Medicine. Recurrent topics in Jo El J. Schultz's work include Cardiac Ischemia and Reperfusion (17 papers), Fibroblast Growth Factor Research (10 papers) and Nitric Oxide and Endothelin Effects (9 papers). Jo El J. Schultz is often cited by papers focused on Cardiac Ischemia and Reperfusion (17 papers), Fibroblast Growth Factor Research (10 papers) and Nitric Oxide and Endothelin Effects (9 papers). Jo El J. Schultz collaborates with scholars based in United States, Poland and Australia. Jo El J. Schultz's co-authors include Garrett J. Gross, Anna K. Hsu, Thomas Doetschman, Michelle L. Nieman, Thomas R. Kimball, Sandra A. Witt, Betty J. Glascock, Peter J. Reiser, Zhenhai Yao and John N. Lorenz and has published in prestigious journals such as Journal of Biological Chemistry, Circulation and Journal of Clinical Investigation.

In The Last Decade

Jo El J. Schultz

46 papers receiving 3.4k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Jo El J. Schultz United States	26	1.6k	1.3k	1.3k	740	544	46	3.4k
Takayuki Miki Japan	39	1.6k 1.0×	1.5k 1.1×	1.8k 1.4×	904 1.2×	484 0.9×	121	4.4k
Ryan M. Fryer United States	28	1.3k 0.8×	723 0.5×	1.6k 1.2×	756 1.0×	585 1.1×	58	3.2k
Sharon L. Hale United States	34	996 0.6×	1.4k 1.0×	1.4k 1.0×	796 1.1×	372 0.7×	101	3.8k
Dorothée Weihrauch United States	38	1.0k 0.7×	480 0.4×	1.3k 1.0×	651 0.9×	938 1.7×	99	3.3k
Hitoshi Takano Japan	30	848 0.5×	1.2k 0.9×	2.3k 1.7×	1.1k 1.5×	765 1.4×	127	4.0k
Javier Inserte Spain	34	1.0k 0.7×	952 0.7×	1.2k 0.9×	638 0.9×	261 0.5×	73	2.6k
Xi‐Ming Yang United States	30	1.0k 0.7×	1.0k 0.8×	3.0k 2.3×	1.8k 2.4×	1.2k 2.1×	44	4.0k
Andreas Skyschally Germany	34	826 0.5×	1.7k 1.3×	1.9k 1.4×	1.0k 1.4×	682 1.3×	74	3.6k
James D. McCully United States	37	2.8k 1.8×	522 0.4×	1.9k 1.4×	512 0.7×	512 0.9×	111	4.7k
Fabrice Prunier France	30	1.1k 0.7×	1.2k 0.9×	828 0.6×	396 0.5×	401 0.7×	101	2.9k

Countries citing papers authored by Jo El J. Schultz

Since Specialization

Citations

This map shows the geographic impact of Jo El J. Schultz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jo El J. Schultz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jo El J. Schultz more than expected).

Fields of papers citing papers by Jo El J. Schultz

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jo El J. Schultz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jo El J. Schultz. The network helps show where Jo El J. Schultz may publish in the future.

Co-authorship network of co-authors of Jo El J. Schultz

This figure shows the co-authorship network connecting the top 25 collaborators of Jo El J. Schultz. A scholar is included among the top collaborators of Jo El J. Schultz based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jo El J. Schultz. Jo El J. Schultz is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

Manning, Janet R., et al.. (2020). Phosphoproteomic analysis identifies phospho-Threonine-17 site of phospholamban important in low molecular weight isoform of fibroblast growth factor 2-induced protection against post-ischemic cardiac dysfunction. Journal of Molecular and Cellular Cardiology. 148. 1–14. 2 indexed citations

Bolte, Craig, Yufang Zhang, Allen J. York, et al.. (2012). Postnatal Ablation of Foxm1 from Cardiomyocytes Causes Late Onset Cardiac Hypertrophy and Fibrosis without Exacerbating Pressure Overload-Induced Cardiac Remodeling. PLoS ONE. 7(11). e48713–e48713. 31 indexed citations

Schulz, Emily, Robert N. Correll, Marco Stephan Lofrano‐Alves, et al.. (2012). Tropomyosin Dephosphorylation Results in Compensated Cardiac Hypertrophy. Journal of Biological Chemistry. 287(53). 44478–44489. 19 indexed citations

Liao, Siyun, Mohamad Azhar, Gilbert Newman, et al.. (2010). The influence of FGF2 high molecular weight (HMW) isoforms in the development of cardiac ischemia–reperfusion injury. Journal of Molecular and Cellular Cardiology. 48(6). 1245–1254. 25 indexed citations

Jones, W. Keith, Guo‐Chang Fan, Siyun Liao, et al.. (2009). Peripheral Nociception Associated With Surgical Incision Elicits Remote Nonischemic Cardioprotection Via Neurogenic Activation of Protein Kinase C Signaling. Circulation. 120(11_suppl_1). S1–9. 125 indexed citations

Bolte, Craig, Gilbert Newman, & Jo El J. Schultz. (2009). Kappa and delta opioid receptor signaling is augmented in the failing heart. Journal of Molecular and Cellular Cardiology. 47(4). 493–503. 25 indexed citations

Bolte, Craig, Gilbert Newman, & Jo El J. Schultz. (2009). Hypertensive state, independent of hypertrophy, exhibits an attenuated decrease in systolic function on cardiac κ-opioid receptor stimulation. American Journal of Physiology-Heart and Circulatory Physiology. 296(4). H967–H975. 18 indexed citations

Liao, Siyun, et al.. (2008). Biological functions of the low and high molecular weight protein isoforms of fibroblast growth factor‐2 in cardiovascular development and disease. Developmental Dynamics. 238(2). 249–264. 65 indexed citations

House, Stacey L., Susan J. Melhorn, Gilbert Newman, Thomas Doetschman, & Jo El J. Schultz. (2007). The protein kinase C pathway mediates cardioprotection induced by cardiac-specific overexpression of fibroblast growth factor-2. American Journal of Physiology-Heart and Circulatory Physiology. 293(1). H354–H365. 28 indexed citations

10.

Bolte, Craig, Siyun Liao, Garrett J. Gross, & Jo El J. Schultz. (2007). Remote Preconditioning- Endocrine Factors in Organ Protection Against Ischemic Injury. Endocrine Metabolic & Immune Disorders - Drug Targets. 7(3). 167–175. 14 indexed citations

11.

Liao, Siyun, et al.. (2006). The cardioprotective effect of the low molecular weight isoform of fibroblast growth factor-2: The role of JNK signaling. Journal of Molecular and Cellular Cardiology. 42(1). 106–120. 60 indexed citations

12.

Gumina, Richard J., et al.. (2005). Cardioprotective-Mimetics Reduce Myocardial Infarct Size in Animals Resistant to Ischemic Preconditioning. Cardiovascular Drugs and Therapy. 19(5). 315–322. 8 indexed citations

13.

Ji, Yong, Wen Zhao, Bailing Li, et al.. (2005). Targeted inhibition of sarcoplasmic reticulum CaMKII activity results in alterations of Ca²⁺homeostasis and cardiac contractility. American Journal of Physiology-Heart and Circulatory Physiology. 290(2). H599–H606. 20 indexed citations

14.

House, Stacey L., Kevin M. Branch, Gilbert Newman, Thomas Doetschman, & Jo El J. Schultz. (2005). Cardioprotection induced by cardiac-specific overexpression of fibroblast growth factor-2 is mediated by the MAPK cascade. American Journal of Physiology-Heart and Circulatory Physiology. 289(5). H2167–H2175. 49 indexed citations

15.

Schultz, Jo El J., Betty J. Glascock, Sandra A. Witt, et al.. (2004). Accelerated onset of heart failure in mice during pressure overload with chronically decreased SERCA2 calcium pump activity. American Journal of Physiology-Heart and Circulatory Physiology. 286(3). H1146–H1153. 77 indexed citations

16.

Dostanic, Iva, Jo El J. Schultz, John N. Lorenz, & Jerry B. Lingrel. (2004). The α1 Isoform of Na,K-ATPase Regulates Cardiac Contractility and Functionally Interacts and Co-localizes with the Na/Ca Exchanger in Heart. Journal of Biological Chemistry. 279(52). 54053–54061. 127 indexed citations

17.

Dostanic, Iva, John N. Lorenz, Jo El J. Schultz, et al.. (2003). The α2 Isoform of Na,K-ATPase Mediates Ouabain-induced Cardiac Inotropy in Mice. Journal of Biological Chemistry. 278(52). 53026–53034. 97 indexed citations

18.

Azhar, Mohamad, Jo El J. Schultz, Ingrid L. Grupp, et al.. (2003). Transforming growth factor beta in cardiovascular development and function. Cytokine & Growth Factor Reviews. 14(5). 391–407. 182 indexed citations

19.

Schultz, Jo El J., Sandra A. Witt, Betty J. Glascock, et al.. (2002). TGF-β1 mediates the hypertrophic cardiomyocyte growth induced by angiotensin II. Journal of Clinical Investigation. 109(6). 787–796. 373 indexed citations

20.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact