Jonathan Satin

4.7k total citations · 1 hit paper
65 papers, 3.7k citations indexed

About

Jonathan Satin is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine and Cellular and Molecular Neuroscience. According to data from OpenAlex, Jonathan Satin has authored 65 papers receiving a total of 3.7k indexed citations (citations by other indexed papers that have themselves been cited), including 59 papers in Molecular Biology, 51 papers in Cardiology and Cardiovascular Medicine and 31 papers in Cellular and Molecular Neuroscience. Recurrent topics in Jonathan Satin's work include Cardiac electrophysiology and arrhythmias (45 papers), Ion channel regulation and function (44 papers) and Neuroscience and Neural Engineering (19 papers). Jonathan Satin is often cited by papers focused on Cardiac electrophysiology and arrhythmias (45 papers), Ion channel regulation and function (44 papers) and Neuroscience and Neural Engineering (19 papers). Jonathan Satin collaborates with scholars based in United States, Hungary and Israel. Jonathan Satin's co-authors include Leanne L. Cribbs, Lior Gepstein, Gil Arbel, Douglas Andres, Izhak Kehat, Oren Caspi, Irit Huber, Elizabeth A. Schroder, Shawn M. Crump and Harry A. Fozzard and has published in prestigious journals such as Science, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Jonathan Satin

63 papers receiving 3.6k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Electromechanical integration of cardiomyocytes derived from human embryonic stem cells

2004 622 citations Jonathan Satin et al. profile →

Peers

Countries citing papers authored by Jonathan Satin

Since Specialization

Citations

This map shows the geographic impact of Jonathan Satin's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jonathan Satin with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jonathan Satin more than expected).

Fields of papers citing papers by Jonathan Satin

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jonathan Satin. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jonathan Satin. The network helps show where Jonathan Satin may publish in the future.

Co-authorship network of co-authors of Jonathan Satin

This figure shows the co-authorship network connecting the top 25 collaborators of Jonathan Satin. A scholar is included among the top collaborators of Jonathan Satin based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jonathan Satin. Jonathan Satin is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Work	Indexed citations
1	Pharmacological or genetic inhibition of LTCC promotes cardiomyocyte proliferation through inhibition of calcineurin activity 2025 ·npj Regenerative Medicine ·Lynn Devilée, (unknown), Jessica M. Miller, (unknown), Qinghui Ou, (unknown), (unknown), (unknown), Yibing Nong, Todd K. Rosengart, Douglas Andres, Jonathan Satin, Tamer Mohamed, James E. Hudson, Riham Abouleisa	5
2	Increased Retention of Cardiac Cells to a Glass Substrate through Streptavidin–Biotin Affinity 2021 ·ACS Omega ·(unknown), (unknown), Andrea Sebastian, (unknown), Christine A. Trinkle, Jonathan Satin, Ahmed Abdel‐Latif, Brad J. Berron	3
3	Myocardial-restricted ablation of the GTPase RAD results in a pro-adaptive heart response in mice 2019 ·Journal of Biological Chemistry ·(unknown), Bryana M. Levitan, Sudhakar Veeranki, (unknown), Nemat Ali, Andrea Sebastian, Wen Su, Ming Gong, Jiayang Li, Julian E. Stelzer, Douglas Andres, Jonathan Satin	22
4	Role of Gap Junction Channel in the Development of Beat-to-Beat Action Potential Repolarization Variability and Arrhythmias 2014 ·Current Pharmaceutical Design ·János Magyar, Tamás Bányász, Norbert Szentandrássy, Kornél Kistamás, Péter P. Nánási, Jonathan Satin	13
5	Mouse Strain Determines Cardiac Growth Potential 2013 ·PLoS ONE ·(unknown), Barry Grimes, Gary Van Zant, Jonathan Satin	21
6	Long Term Regulation of Cardiac L-Type Calcium Channel by Small G Proteins 2011 ·Current Medicinal Chemistry ·János Magyar, (unknown), Kornél Kistamás, (unknown), Péter P. Nánási, Jonathan Satin, Norbert Szentandrássy, Tamás Bányász	2
7	Rem GTPase interacts with the proximal Ca_V1.2 C-terminus and modulates calcium-dependent channel inactivation 2010 ·Channels ·(unknown), Shawn M. Crump, (unknown), Robert N. Correll, Brian S. Finlin, Jonathan Satin, Douglas Andres	21
8	L-Type Calcium Channel C Terminus Autoregulates Transcription 2009 ·Circulation Research ·Elizabeth A. Schroder, (unknown), Jonathan Satin	76
9	Autoregulation of Cardiac l-Type Calcium Channels 2009 ·Trends in Cardiovascular Medicine ·Jonathan Satin, Elizabeth A. Schroder	9
10	Steady-state coupling of plasma membrane calcium entry to extrusion revealed by novel L-type calcium channel block 2008 ·Cell Calcium ·(unknown), Elizabeth A. Schroder, Don E. Burgess, (unknown), Douglas Andres, Jonathan Satin	7
11	Calmodulin binding is dispensable for Rem-mediated Ca2+ channel inhibition 2007 ·Molecular and Cellular Biochemistry ·Robert N. Correll, (unknown), Dana M. Niedowicz, Jonathan Satin, Douglas Andres	4
12	Chronic verapamil treatment remodelsI_Ca,Lin mouse ventricle 2006 ·American Journal of Physiology-Heart and Circulatory Physiology ·Elizabeth A. Schroder, János Magyar, Don E. Burgess, Douglas Andres, Jonathan Satin	24
13	Divergent action potential morphologies reveal nonequilibrium properties of human cardiac Na channels 2004 ·Cardiovascular Research ·János Magyar, (unknown), Robert Dumaine, Diane J. Burgess, Tamás Bányász, Jonathan Satin	14
14	Regulation of voltage-gated calcium channel activity by the Rem and Rad GTPases 2003 ·Proceedings of the National Academy of Sciences ·Brian S. Finlin, Shawn M. Crump, Jonathan Satin, Douglas Andres	174
15	Monovalent Cations Contribute to T-type Calcium Channel (Ca V 3.1 and Ca V 3.2) Selectivity 2003 ·The Journal of Membrane Biology ·Brian P. Delisle, Jonathan Satin	7
16	pH Modification of Human T-Type Calcium Channel Gating 2000 ·Biophysical Journal ·Brian P. Delisle, Jonathan Satin	40
17	Divalent cation competition with [3H]saxitoxin binding to tetrodotoxin-resistant and -sensitive sodium channels. A two-site structural model of ion/toxin interaction. 1993 ·The Journal of General Physiology ·Donald D. Doyle, Yi Guo, S. Lustig, Jonathan Satin, R. B. Rogart, Harry A. Fozzard	56
18	Post-repolarization block of cardiac sodium channels by saxitoxin 1993 ·Biophysical Journal ·J. C. Makielski, Jonathan Satin, Zheng Fan	15
19	Functional expression of the rat heart I Na⁺ channel isoform Demonstration of properties characteristic of native cardiac Na⁺ channels 1990 ·FEBS Letters ·Leanne L. Cribbs, Jonathan Satin, Harry A. Fozzard, R. B. Rogart	59
20	Cell Interactions in Cardiac Development 1990 ·Development Growth & Differentiation ·Robert L. DeHaan, Shiroh Fujii, Jonathan Satin	11

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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