Raymond Pagliarini
- Molecular Biology top 5%
- Cell Biology top 2%
- Oncology top 5%
- Cancer Research top 5%
- Immunology top 10%
- Co-authors
- Tian XuTatsushi IgakiWilliam R. SellersWenlin ShaoAlexandra GrassianAjay SrivastavaFranklin ChungNickolas Papadopoulos
- Topics
- Cancer, Hypoxia, and Metabolism (4 papers)Glioma Diagnosis and Treatment (3 papers)Protein Degradation and Inhibitors (3 papers)
- Cited by
- Cell BiologyCancer ResearchAging
- Partner nations
- United StatesSwitzerlandChina
In The Last Decade
Raymond Pagliarini
19 papers receiving 2.3k citations
Peers
Comparison fields: 5 of 96
- Molecular Biology 1.6k
- Cell Biology 725
- Oncology 543
- Cancer Research 525
- Immunology 380
Countries citing papers authored by Raymond Pagliarini
This map shows the geographic impact of Raymond Pagliarini's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Raymond Pagliarini with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Raymond Pagliarini more than expected).
Fields of papers citing papers by Raymond Pagliarini
This network shows the impact of papers produced by Raymond Pagliarini. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Raymond Pagliarini. The network helps show where Raymond Pagliarini may publish in the future.
Co-authorship network of co-authors of Raymond Pagliarini
This figure shows the co-authorship network connecting the top 25 collaborators of Raymond Pagliarini. A scholar is included among the top collaborators of Raymond Pagliarini based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Raymond Pagliarini. Raymond Pagliarini is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 15 | |
| 3 | 221 | |
| 4 | 179 | |
| 5 | 211 | |
| 6 | 49 | |
| 7 | IDH1 Mutations Alter Citric Acid Cycle Metabolism and Increase Dependence on Oxidative Mitochondrial Metabolism | 83 |
| 8 | 12 | |
| 9 | 42 | |
| 10 | 46 | |
| 11 | 2 | |
| 12 | 49 | |
| 13 | 64 | |
| 14 | 83 | |
| 15 | 133 | |
| 16 | 230 | |
| 17 | 170 | |
| 18 | 300 | |
| 19 | 436 | |
| 20 | 11 |
About Raymond Pagliarini
Raymond Pagliarini is a scholar working on Cancer Research, Genetics and Cell Biology, having authored 20 papers that have together received 2.3k indexed citations. Recurring topics across this work include Cancer, Hypoxia, and Metabolism (4 papers), Glioma Diagnosis and Treatment (3 papers) and Protein Degradation and Inhibitors (3 papers). The work is most often cited by research in Cell Biology (725 citations), Cancer Research (525 citations) and Aging (45 citations). Raymond Pagliarini has collaborated with scholars based in United States, Switzerland and China. Frequent co-authors include Tian Xu, Tatsushi Igaki, William R. Sellers, Wenlin Shao, Alexandra Grassian, Ajay Srivastava, Franklin Chung, Nickolas Papadopoulos, Carlo Rago and Surojit Sur. Their work appears in journals such as Science, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.