Petra Mäkinen

1.3k total citations
32 papers, 807 citations indexed

About

Petra Mäkinen is a scholar working on Physiology, Cellular and Molecular Neuroscience and Neurology. According to data from OpenAlex, Petra Mäkinen has authored 32 papers receiving a total of 807 indexed citations (citations by other indexed papers that have themselves been cited), including 24 papers in Physiology, 9 papers in Cellular and Molecular Neuroscience and 9 papers in Neurology. Recurrent topics in Petra Mäkinen's work include Alzheimer's disease research and treatments (24 papers), Neuroinflammation and Neurodegeneration Mechanisms (9 papers) and Drug Transport and Resistance Mechanisms (4 papers). Petra Mäkinen is often cited by papers focused on Alzheimer's disease research and treatments (24 papers), Neuroinflammation and Neurodegeneration Mechanisms (9 papers) and Drug Transport and Resistance Mechanisms (4 papers). Petra Mäkinen collaborates with scholars based in Finland, United States and Germany. Petra Mäkinen's co-authors include Annakaisa Haapasalo, Mikko Hiltunen, Hilkka Soininen, Timo Sarajärvi, Heikki Tanila, Jayashree Viswanathan, Seppo Helisalmi, Teemu Natunen, Henna Koivisto and Saila Vepsäläinen and has published in prestigious journals such as Journal of Biological Chemistry, PLoS ONE and NeuroImage.

In The Last Decade

Petra Mäkinen

31 papers receiving 801 citations

Peers

Petra Mäkinen
Mari Takalo Finland
Hannah Tayler United Kingdom
Ronaldo Perez United States
Nurgül Aytan United States
Jordan B. Jahrling United States
Mari Takalo Finland
Petra Mäkinen
Citations per year, relative to Petra Mäkinen Petra Mäkinen (= 1×) peers Mari Takalo

Countries citing papers authored by Petra Mäkinen

Since Specialization
Citations

This map shows the geographic impact of Petra Mäkinen's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Petra Mäkinen with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Petra Mäkinen more than expected).

Fields of papers citing papers by Petra Mäkinen

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Petra Mäkinen. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Petra Mäkinen. The network helps show where Petra Mäkinen may publish in the future.

Co-authorship network of co-authors of Petra Mäkinen

This figure shows the co-authorship network connecting the top 25 collaborators of Petra Mäkinen. A scholar is included among the top collaborators of Petra Mäkinen based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Petra Mäkinen. Petra Mäkinen is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Martiskainen, Henna, Stephan A. Müller, Mari Takalo, et al.. (2025). Monoallelic TYROBP deletion is a novel risk factor for Alzheimer’s disease. Molecular Neurodegeneration. 20(1). 50–50.
2.
Kemppainen, Susanna, Nadine Huber, Ana Zamora, et al.. (2023). Organotypic Hippocampal Slice Cultures from Adult Tauopathy Mice and Theragnostic Evaluation of Nanomaterial Phospho-TAU Antibody-Conjugates. Cells. 12(10). 1422–1422. 1 indexed citations
3.
Leskelä, Stina, Ali Abdollahzadeh, Rebekka Wittrahm, et al.. (2020). BV-2 Microglial Cells Overexpressing C9orf72 Hexanucleotide Repeat Expansion Produce DPR Proteins and Show Normal Functionality but No RNA Foci. Frontiers in Neurology. 11. 550140–550140. 2 indexed citations
4.
Takalo, Mari, Rebekka Wittrahm, Benedikt Wefers, et al.. (2020). The Alzheimer’s disease-associated protective Plcγ2-P522R variant promotes immune functions. Molecular Neurodegeneration. 15(1). 52–52. 53 indexed citations
5.
Marttinen, Mikael, Jussi Paananen, Antonio Neme, et al.. (2018). A multiomic approach to characterize the temporal sequence in Alzheimer's disease-related pathology. Neurobiology of Disease. 124. 454–468. 39 indexed citations
6.
Gabbouj, Sami, Teemu Natunen, Hennariikka Koivisto, et al.. (2018). Intranasal insulin activates Akt2 signaling pathway in the hippocampus of wild-type but not in APP/PS1 Alzheimer model mice. Neurobiology of Aging. 75. 98–108. 23 indexed citations
7.
Leskelä, Stina, Mari Takalo, Mikael Marttinen, et al.. (2018). Interrelationship between the Levels of C9orf72 and Amyloid-β Protein Precursor and Amyloid-β in Human Cells and Brain Samples. Journal of Alzheimer s Disease. 62(1). 269–278. 3 indexed citations
8.
Martiskainen, Henna, Kaisa M. A. Paldanius, Teemu Natunen, et al.. (2017). DHCR24 exerts neuroprotection upon inflammation-induced neuronal death. Journal of Neuroinflammation. 14(1). 215–215. 39 indexed citations
9.
Sarajärvi, Timo, Mikael Marttinen, Teemu Natunen, et al.. (2015). Genetic Variation in δ-Opioid Receptor Associates with Increased β- and γ-Secretase Activity in the Late Stages of Alzheimer’s Disease. Journal of Alzheimer s Disease. 48(2). 507–516. 13 indexed citations
10.
Takalo, Mari, Annakaisa Haapasalo, Henna Martiskainen, et al.. (2014). High-fat diet increases tau expression in the brain of T2DM and AD mice independently of peripheral metabolic status. The Journal of Nutritional Biochemistry. 25(6). 634–641. 43 indexed citations
11.
Sarajärvi, Timo, Annakaisa Haapasalo, Lakshman Puli, et al.. (2014). P1‐082: INCREASED GAMMA‐SECRETASE ACTIVITY IN IDIOPATHIC NORMAL PRESSURE HYDROCEPHALUS PATIENTS WITH B‐AMYLOID PATHOLOGY. Alzheimer s & Dementia. 10(4S_Part_8). 1 indexed citations
13.
Sarajärvi, Timo, Annakaisa Haapasalo, Lakshman Puli, et al.. (2014). Increased γ-Secretase Activity in Idiopathic Normal Pressure Hydrocephalus Patients with β-Amyloid Pathology. PLoS ONE. 9(4). e93717–e93717. 11 indexed citations
14.
Vepsäläinen, Saila, Henna Koivisto, Petra Mäkinen, et al.. (2012). Anthocyanin-enriched bilberry and blackcurrant extracts modulate amyloid precursor protein processing and alleviate behavioral abnormalities in the APP/PS1 mouse model of Alzheimer's disease. The Journal of Nutritional Biochemistry. 24(1). 360–370. 120 indexed citations
15.
Hiltunen, Mikko, Vinoth Kumar Megraj Khandelwal, Nagendra Yaluri, et al.. (2011). Contribution of genetic and dietary insulin resistance to Alzheimer phenotype in APP/PS1 transgenic mice. Journal of Cellular and Molecular Medicine. 16(6). 1206–1222. 65 indexed citations
16.
Sarajärvi, Timo, Jussi Tuusa, Annakaisa Haapasalo, et al.. (2011). Cysteine 27 Variant of the δ-Opioid Receptor Affects Amyloid Precursor Protein Processing through Altered Endocytic Trafficking. Molecular and Cellular Biology. 31(11). 2326–2340. 29 indexed citations
17.
Sarajärvi, Timo, Annakaisa Haapasalo, Jayashree Viswanathan, et al.. (2009). Down-regulation of Seladin-1 Increases BACE1 Levels and Activity through Enhanced GGA3 Depletion during Apoptosis. Journal of Biological Chemistry. 284(49). 34433–34443. 55 indexed citations
18.
Hiltunen, Mikko, Petra Mäkinen, Sirpa Peräniemi, et al.. (2009). Focal cerebral ischemia in rats alters APP processing and expression of Aβ peptide degrading enzymes in the thalamus. Neurobiology of Disease. 35(1). 103–113. 53 indexed citations
19.
Turunen, Mari, Saila Vepsäläinen, Petra Mäkinen, et al.. (2009). No association between high temperature requirement 1 (HTRA1) gene polymorphisms and Alzheimer's disease. Neurobiology of Aging. 32(3). 547.e7–547.e9. 7 indexed citations
20.
Viswanathan, Jayashree, Petra Mäkinen, Seppo Helisalmi, et al.. (2008). An association study between granulin gene polymorphisms and Alzheimer's disease in Finnish population. American Journal of Medical Genetics Part B Neuropsychiatric Genetics. 150B(5). 747–750. 46 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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