Teemu Natunen
- Physiology top 5%
- Molecular Biology
- Neurology top 5%
- Cellular and Molecular Neuroscience top 10%
- Neurology top 10%
- Co-authors
- Mikko HiltunenAnnakaisa HaapasaloMari TakaloMikael MarttinenHenna MartiskainenSami GabboujRebekka WittrahmSusanna Kemppainen
- Topics
- Alzheimer's disease research and treatments (23 papers)Neuroinflammation and Neurodegeneration Mechanisms (10 papers)Parkinson's Disease Mechanisms and Treatments (4 papers)
- Journals
- PLoS ONENeuroImageBiochemistry
- Partner nations
- FinlandUnited KingdomUnited States
In The Last Decade
Teemu Natunen
42 papers receiving 1.1k citations
Hit Papers
Peers
Comparison fields: 5 of 92
- Physiology 481
- Molecular Biology 453
- Neurology 215
- Cellular and Molecular Neuroscience 168
- Neurology 164
Countries citing papers authored by Teemu Natunen
This map shows the geographic impact of Teemu Natunen's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Teemu Natunen with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Teemu Natunen more than expected).
Fields of papers citing papers by Teemu Natunen
This network shows the impact of papers produced by Teemu Natunen. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Teemu Natunen. The network helps show where Teemu Natunen may publish in the future.
Co-authorship network of co-authors of Teemu Natunen
This figure shows the co-authorship network connecting the top 25 collaborators of Teemu Natunen. A scholar is included among the top collaborators of Teemu Natunen based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Teemu Natunen. Teemu Natunen is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 4 | |
| 3 | 3 | |
| 4 | 3 | |
| 5 | 6 | |
| 6 | 10 | |
| 7 | 4 | |
| 8 | 2 | |
| 9 | Altered Insulin Signaling in Alzheimer’s Disease Brain – Special Emphasis on PI3K-Akt Pathwaybreakdown → | 300 |
| 10 | 70 | |
| 11 | 20 | |
| 12 | 34 | |
| 13 | 13 | |
| 14 | 66 | |
| 15 | 39 | |
| 16 | 23 | |
| 17 | 41 | |
| 18 | 39 | |
| 19 | 56 | |
| 20 | 13 |
About Teemu Natunen
Teemu Natunen is a scholar working on Neurology, Physiology and Developmental Neuroscience, having authored 44 papers that have together received 1.1k indexed citations. Recurring topics across this work include Alzheimer's disease research and treatments (23 papers), Neuroinflammation and Neurodegeneration Mechanisms (10 papers) and Parkinson's Disease Mechanisms and Treatments (4 papers). The work is most often cited by research in Neurology (215 citations), Biological Psychiatry (58 citations) and Physiology (481 citations). Teemu Natunen has collaborated with scholars based in Finland, United Kingdom and United States. Frequent co-authors include Mikko Hiltunen, Annakaisa Haapasalo, Mari Takalo, Mikael Marttinen, Henna Martiskainen, Sami Gabbouj, Rebekka Wittrahm, Susanna Kemppainen, Heikki Tanila and Simo Ryhänen. Their work appears in journals such as PLoS ONE, NeuroImage and Biochemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.