Mari Takalo

4.1k total citations · 1 hit paper
24 papers, 871 citations indexed

About

Mari Takalo is a scholar working on Physiology, Neurology and Molecular Biology. According to data from OpenAlex, Mari Takalo has authored 24 papers receiving a total of 871 indexed citations (citations by other indexed papers that have themselves been cited), including 16 papers in Physiology, 11 papers in Neurology and 7 papers in Molecular Biology. Recurrent topics in Mari Takalo's work include Alzheimer's disease research and treatments (16 papers), Neuroinflammation and Neurodegeneration Mechanisms (9 papers) and Amyotrophic Lateral Sclerosis Research (5 papers). Mari Takalo is often cited by papers focused on Alzheimer's disease research and treatments (16 papers), Neuroinflammation and Neurodegeneration Mechanisms (9 papers) and Amyotrophic Lateral Sclerosis Research (5 papers). Mari Takalo collaborates with scholars based in Finland, United States and Germany. Mari Takalo's co-authors include Annakaisa Haapasalo, Mikko Hiltunen, Teemu Natunen, Mikael Marttinen, Hilkka Soininen, Rebekka Wittrahm, Susanna Kemppainen, Henna Martiskainen, Sami Gabbouj and Heikki Tanila and has published in prestigious journals such as Neurobiology of Aging, Biochimica et Biophysica Acta (BBA) - Molecular Cell Research and Neurobiology of Disease.

In The Last Decade

Mari Takalo

23 papers receiving 863 citations

Hit Papers

Altered Insulin Signaling in Alzheimer’s Disease Brain – ... 2019 2026 2021 2023 2019 100 200 300
What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

  1. Altered Insulin Signaling in Alzheimer’s Disease Brain – Special Emphasis on PI3K-Akt Pathway
    2019 300 citations Sami Gabbouj, Simo Ryhänen et al. Frontiers in Neuroscience profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Mari Takalo Finland 12 396 367 190 129 102 24 871
Teemu Natunen Finland 18 481 1.2× 453 1.2× 215 1.1× 168 1.3× 112 1.1× 44 1.1k
Xiaoguang Li China 18 439 1.1× 387 1.1× 209 1.1× 232 1.8× 115 1.1× 39 1.0k
Jayoung Byun South Korea 10 416 1.1× 283 0.8× 114 0.6× 96 0.7× 97 1.0× 12 682
Petra Mäkinen Finland 18 422 1.1× 271 0.7× 192 1.0× 146 1.1× 71 0.7× 32 807
Mikael Marttinen Finland 12 344 0.9× 320 0.9× 187 1.0× 115 0.9× 88 0.9× 21 760
Long Wu United States 15 567 1.4× 630 1.7× 171 0.9× 147 1.1× 108 1.1× 21 1.1k
Jordan B. Jahrling United States 10 414 1.0× 252 0.7× 227 1.2× 109 0.8× 84 0.8× 10 747
Fabrício Simão Brazil 19 264 0.7× 339 0.9× 241 1.3× 191 1.5× 77 0.8× 30 1.0k
Masato Maesako United States 18 577 1.5× 347 0.9× 145 0.8× 200 1.6× 95 0.9× 39 910
Hannah Tayler United Kingdom 11 467 1.2× 281 0.8× 259 1.4× 112 0.9× 65 0.6× 17 828

Countries citing papers authored by Mari Takalo

Since Specialization
Citations

This map shows the geographic impact of Mari Takalo's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mari Takalo with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mari Takalo more than expected).

Fields of papers citing papers by Mari Takalo

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mari Takalo. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mari Takalo. The network helps show where Mari Takalo may publish in the future.

Co-authorship network of co-authors of Mari Takalo

This figure shows the co-authorship network connecting the top 25 collaborators of Mari Takalo. A scholar is included among the top collaborators of Mari Takalo based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mari Takalo. Mari Takalo is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Müller, Stephan A., Pinja Kettunen, Šárka Lehtonen, et al.. (2025). Inflammation-induced lysosomal dysfunction in human iPSC-derived microglia is exacerbated by APOE 4/4 genotype. Journal of Neuroinflammation. 22(1). 147–147. 3 indexed citations
2.
Martiskainen, Henna, Stephan A. Müller, Mari Takalo, et al.. (2025). Monoallelic TYROBP deletion is a novel risk factor for Alzheimer’s disease. Molecular Neurodegeneration. 20(1). 50–50.
3.
Kemppainen, Susanna, Nadine Huber, Ana Zamora, et al.. (2023). Organotypic Hippocampal Slice Cultures from Adult Tauopathy Mice and Theragnostic Evaluation of Nanomaterial Phospho-TAU Antibody-Conjugates. Cells. 12(10). 1422–1422. 1 indexed citations
4.
Wittrahm, Rebekka, Mari Takalo, Mikael Marttinen, et al.. (2021). MECP2 Increases the Pro-Inflammatory Response of Microglial Cells and Phosphorylation at Serine 423 Regulates Neuronal Gene Expression upon Neuroinflammation. Cells. 10(4). 860–860. 10 indexed citations
5.
Huber, Nadine, Dorit Hoffmann, Raisa Giniatullina, et al.. (2021). C9orf72 hexanucleotide repeat expansion leads to altered neuronal and dendritic spine morphology and synaptic dysfunction. Neurobiology of Disease. 162. 105584–105584. 4 indexed citations
6.
Leskelä, Stina, Nadine Huber, Dorit Hoffmann, et al.. (2021). Expression of C9orf72 hexanucleotide repeat expansion leads to formation of RNA foci and dipeptide repeat proteins but does not influence autophagy or proteasomal function in neuronal cells. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1868(7). 119021–119021. 5 indexed citations
7.
Takalo, Mari, Rebekka Wittrahm, Benedikt Wefers, et al.. (2020). The Alzheimer’s disease-associated protective Plcγ2-P522R variant promotes immune functions. Molecular Neurodegeneration. 15(1). 52–52. 53 indexed citations
8.
Leskelä, Stina, Ali Abdollahzadeh, Rebekka Wittrahm, et al.. (2020). BV-2 Microglial Cells Overexpressing C9orf72 Hexanucleotide Repeat Expansion Produce DPR Proteins and Show Normal Functionality but No RNA Foci. Frontiers in Neurology. 11. 550140–550140. 2 indexed citations
9.
Leskelä, Stina, Nadine Huber, Teemu Natunen, et al.. (2019). C9orf72 Proteins Regulate Autophagy and Undergo Autophagosomal or Proteasomal Degradation in a Cell Type-Dependent Manner. Cells. 8(10). 1233–1233. 20 indexed citations
10.
Gabbouj, Sami, Simo Ryhänen, Mikael Marttinen, et al.. (2019). Altered Insulin Signaling in Alzheimer’s Disease Brain – Special Emphasis on PI3K-Akt Pathway. Frontiers in Neuroscience. 13. 629–629. 300 indexed citations breakdown →
11.
Marttinen, Mikael, Jussi Paananen, Antonio Neme, et al.. (2018). A multiomic approach to characterize the temporal sequence in Alzheimer's disease-related pathology. Neurobiology of Disease. 124. 454–468. 39 indexed citations
12.
Marttinen, Mikael, Mari Takalo, Teemu Natunen, et al.. (2018). Molecular Mechanisms of Synaptotoxicity and Neuroinflammation in Alzheimer’s Disease. Frontiers in Neuroscience. 12. 963–963. 66 indexed citations
13.
Gabbouj, Sami, Teemu Natunen, Hennariikka Koivisto, et al.. (2018). Intranasal insulin activates Akt2 signaling pathway in the hippocampus of wild-type but not in APP/PS1 Alzheimer model mice. Neurobiology of Aging. 75. 98–108. 23 indexed citations
14.
Leskelä, Stina, Mari Takalo, Mikael Marttinen, et al.. (2018). Interrelationship between the Levels of C9orf72 and Amyloid-β Protein Precursor and Amyloid-β in Human Cells and Brain Samples. Journal of Alzheimer s Disease. 62(1). 269–278. 3 indexed citations
15.
Takalo, Mari, Teemu Natunen, Stina Leskelä, et al.. (2017). New Implications for the Role for Ubiquilin-1 in Molecular Mechanisms of Alzheime's Disease: Interrelationship with BACE1. Journal of Alzheimer’s Disease & Parkinsonism. 7(5). 1 indexed citations
16.
Martiskainen, Henna, Kaisa M. A. Paldanius, Teemu Natunen, et al.. (2017). DHCR24 exerts neuroprotection upon inflammation-induced neuronal death. Journal of Neuroinflammation. 14(1). 215–215. 39 indexed citations
17.
Takalo, Mari, Annakaisa Haapasalo, Henna Martiskainen, et al.. (2014). High-fat diet increases tau expression in the brain of T2DM and AD mice independently of peripheral metabolic status. The Journal of Nutritional Biochemistry. 25(6). 634–641. 43 indexed citations
18.
Koivisto, Hennariikka, Marcus O.W. Grimm, Tatjana L. Rothhaar, et al.. (2013). Special lipid-based diets alleviate cognitive deficits in the APPswe/PS1dE9 transgenic mouse model of Alzheimer's disease independent of brain amyloid deposition. The Journal of Nutritional Biochemistry. 25(2). 157–169. 46 indexed citations
19.
Takalo, Mari, Annakaisa Haapasalo, Teemu Natunen, et al.. (2013). Targeting ubiquilin-1 in Alzheimer's disease. Expert Opinion on Therapeutic Targets. 17(7). 795–810. 13 indexed citations
20.
Broersen, Laus M., Hennariikka Koivisto, Mari Takalo, Pasi Miettinen, & Heikki Tanila. (2011). P3‐420: Special lipid‐based diets alleviate cognitive deficits in the APPswe/PS1dE9 transgenic mouse model of Alzheimer's disease independent of brain amyloid deposition. Alzheimer s & Dementia. 7(4S_Part_19). 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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