Paul T. Francis

17.7k citations
249 papers · 12.3k indexed · 1 hit paper · h-index 61

Paul T. Francis

243 papers receiving 11.9k citations

Hit Papers

The cholinergic hypothesis of Alzheimer's disease: a revi...1.7k199920262008201750010001.5k

Peers

Paul T. Francis
Comparison fields: 5 of 164
  • Biological Psychiatry 733
  • Cellular and Molecular Neuroscience 3.8k
  • Pharmacology 3.5k
  • Neurology 1.7k
  • Physiology 4.7k
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Citations per year

Countries citing papers authored by Paul T. Francis

Since Specialization
Citations

This map shows the geographic impact of Paul T. Francis's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Paul T. Francis with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Paul T. Francis more than expected).

Fields of papers citing papers by Paul T. Francis

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Paul T. Francis. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Paul T. Francis. The network helps show where Paul T. Francis may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Paul T. Francis, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Paul T. Francis Line = papers co-authored together Paul T. Francis links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown
#Work
1 20234
2 202313
3 202215
4 202260
5 20214
6 2020110
7 20206
8 20198
9 201714
10 201332
11 20117
12 20107
13
Loss of serotonin 5-HT2A receptors in, the temporal cortex correlates with cognitive decline in Alzheimer's disease
20042
14
Serotonin 5-HT2A receptor alterations in the postmortem neocortex of behaviorally assessed Alzheimer patients
20032
15 1999103
16 19974
17 19957
18 199020
19 198967
20 1988113

About Paul T. Francis

Paul T. Francis is a scholar working on Cellular and Molecular Neuroscience, Physiology and Pharmacology, having authored 249 papers that have together received 12.3k indexed citations. Recurring topics across this work include Alzheimer's disease research and treatments (102 papers), Neuroscience and Neuropharmacology Research (65 papers), Cholinesterase and Neurodegenerative Diseases (53 papers), Memory and Neural Mechanisms (30 papers), Parkinson's Disease Mechanisms and Treatments (25 papers), Dementia and Cognitive Impairment Research (22 papers), Nicotinic Acetylcholine Receptors Study (21 papers) and Neurotransmitter Receptor Influence on Behavior (15 papers). The work is most often cited by research in Biological Psychiatry (733 citations), Cellular and Molecular Neuroscience (3.8k citations) and Pharmacology (3.5k citations). Paul T. Francis has collaborated with scholars based in United Kingdom, Singapore and United States. Frequent co-authors include Alan M. Palmer, Mike Snape, G K Wilcock, Margaret M. Esiri, Christopher Chen, David M. Bowen, Andrew Procter, D. M. Bowen, Clive Ballard and Mitchell K.P. Lai. Their work appears in journals such as Alzheimer s & Dementia, Neurobiology of Aging, Journal of Neurochemistry, Dementia and Geriatric Cognitive Disorders and Brain Research.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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