Emma Dempster
- Molecular Biology top 5%
- Genetics top 2%
- Pediatrics, Perinatology and Child Health top 2%
- Social Psychology top 5%
- Cognitive Neuroscience top 5%
- Co-authors
- Jonathan MillLeonard C. SchalkwykDavid CollierRobin MurrayRuth PidsleyCathy L. BarrJoana VianaBing Ren
- Topics
- Epigenetics and DNA Methylation (38 papers)Genetic Syndromes and Imprinting (14 papers)Genetics and Neurodevelopmental Disorders (11 papers)
- Journals
- CellNature CommunicationsPLoS ONE
- Partner nations
- United KingdomUnited StatesCanada
In The Last Decade
Emma Dempster
71 papers receiving 3.5k citations
Peers
Comparison fields: 5 of 133
- Molecular Biology 2.0k
- Genetics 1.1k
- Pediatrics, Perinatology and Child Health 478
- Social Psychology 411
- Cognitive Neuroscience 387
Countries citing papers authored by Emma Dempster
This map shows the geographic impact of Emma Dempster's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Emma Dempster with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Emma Dempster more than expected).
Fields of papers citing papers by Emma Dempster
This network shows the impact of papers produced by Emma Dempster. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Emma Dempster. The network helps show where Emma Dempster may publish in the future.
Co-authorship network of co-authors of Emma Dempster
This figure shows the co-authorship network connecting the top 25 collaborators of Emma Dempster. A scholar is included among the top collaborators of Emma Dempster based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Emma Dempster. Emma Dempster is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 1 | |
| 2 | 1 | |
| 3 | 0 | |
| 4 | 3 | |
| 5 | 5 | |
| 6 | 7 | |
| 7 | 3 | |
| 8 | 60 | |
| 9 | 8 | |
| 10 | 7 | |
| 11 | 84 | |
| 12 | 29 | |
| 13 | 26 | |
| 14 | 20 | |
| 15 | 63 | |
| 16 | 24 | |
| 17 | 100 | |
| 18 | 20 | |
| 19 | 66 | |
| 20 | Methylation analysis of a NGF1-A transcription factor binding-site in the promoter region of the human glucocorticoid receptor gene (NR3C1) | 1 |
About Emma Dempster
Emma Dempster is a scholar working on Genetics, Behavioral Neuroscience and Molecular Biology, having authored 74 papers that have together received 3.6k indexed citations. Recurring topics across this work include Epigenetics and DNA Methylation (38 papers), Genetic Syndromes and Imprinting (14 papers) and Genetics and Neurodevelopmental Disorders (11 papers). The work is most often cited by research in Biological Psychiatry (246 citations), Behavioral Neuroscience (238 citations) and Genetics (1.1k citations). Emma Dempster has collaborated with scholars based in United Kingdom, United States and Canada. Frequent co-authors include Jonathan Mill, Leonard C. Schalkwyk, David Collier, Robin Murray, Ruth Pidsley, Cathy L. Barr, Joana Viana, Bing Ren, Wei Xie and Ah Young Lee. Their work appears in journals such as Cell, Nature Communications and PLoS ONE.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.