Neil H. James
About
Neil H. James is a scholar working on Molecular Biology, Oncology and Cancer Research.
According to data from OpenAlex, Neil H. James has authored 28 papers receiving a total of 1.5k indexed citations (citations by other indexed papers that have themselves been cited), including 26 papers in Molecular Biology, 11 papers in Oncology and 7 papers in Cancer Research. Recurrent topics in Neil H. James's work include Peroxisome Proliferator-Activated Receptors (14 papers), PI3K/AKT/mTOR signaling in cancer (6 papers) and Drug Transport and Resistance Mechanisms (5 papers). Neil H. James is often cited by papers focused on Peroxisome Proliferator-Activated Receptors (14 papers), PI3K/AKT/mTOR signaling in cancer (6 papers) and Drug Transport and Resistance Mechanisms (5 papers). Neil H. James collaborates with scholars based in United Kingdom, United States and Japan. Neil H. James's co-authors include Ruth Roberts, Susan C. Hasmall, Sabina Cosulich, Neil Macdonald, Anthony R. Soames, Peter R. Holden, Eric B. Wheeldon, Jason H. Gill, Juliane M. Jürgensmeier and Neil R. Smith and has published in prestigious journals such as Hepatology, Oncogene and Clinical Cancer Research.
In The Last Decade
Neil H. James
27 papers receiving 1.4k citations
Peers
Neil H. James
Mario D’Andrea Italy
Vincent Wai‐Hin Yuen Hong Kong
Sheng-Chieh Hsu Taiwan
Dae‐Kee Kim South Korea
Michael Dukes United Kingdom
Sven A. Lang Germany
Elaine S. E. Stokes United Kingdom
Peihua Luo China
Cinzia Lanzi Italy
Anja J. Stauber United States
Citations per year, relative to Neil H. James Neil H. James (= 1×)
peers
Mario D’Andrea
Countries citing papers authored by Neil H. James
Since
Specialization
Citations
This map shows the geographic impact of Neil H. James's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Neil H. James with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Neil H. James more than expected).
Fields of papers citing papers by Neil H. James
Since
Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences
This network shows the impact of papers produced by Neil H. James. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Neil H. James. The network helps show where Neil H. James may publish in the future.
Co-authorship network of co-authors of Neil H. James
This figure shows the co-authorship network connecting the top 25 collaborators of Neil H. James. A scholar is included among the top collaborators of Neil H. James based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Neil H. James. Neil H. James is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
1.
Nakamura, Kyoko, Ankur Karmokar, Paul Farrington, et al.. (2021). Inhibition of DNA-PK with AZD7648 Sensitizes Tumor Cells to Radiotherapy and Induces Type I IFN-Dependent Durable Tumor Control. Clinical Cancer Research. 27(15). 4353–4366.
2.
Wijnhoven, Paul W.G., Antonio Ramos‐Montoya, Zena Wilson, et al.. (2020). Combined PARP and ATR inhibition potentiates genome instability and cell death in ATM-deficient cancer cells. Oncogene. 39(25). 4869–4883.
3.
Davies, Barry R., Armelle Logié, Claire Crafter, et al.. (2015). Tumors with AKT1E17K Mutations Are Rational Targets for Single Agent or Combination Therapy with AKT Inhibitors. Molecular Cancer Therapeutics. 14(11). 2441–2451.
4.
Crafter, Claire, John P. Vincent, Eric Tang, et al.. (2015). Combining AZD8931, a novel EGFR/HER2/HER3 signalling inhibitor, with AZD5363 limits AKT inhibitor induced feedback and enhances antitumour efficacy in HER2-amplified breast cancer models. International Journal of Oncology. 47(2). 446–454.
5.
Brave, Sandra R., Kirsty Ratcliffe, Zena Wilson, et al.. (2011). Assessing the Activity of Cediranib, a VEGFR-2/3 Tyrosine Kinase Inhibitor, against VEGFR-1 and Members of the Structurally Related PDGFR Family. Molecular Cancer Therapeutics. 10(5). 861–873.
6.
Smith, Neil R., Dawn Baker, Neil H. James, et al.. (2010). Vascular Endothelial Growth Factor Receptors VEGFR-2 and VEGFR-3 Are Localized Primarily to the Vasculature in Human Primary Solid Cancers. Clinical Cancer Research. 16(14). 3548–3561.
7.
Fox, Stephen B., Helen Turley, Cristina Blázquez, et al.. (2004). Phosphorylated KDR is expressed in the neoplastic and stromal elements of human renal tumours and shuttles from cell membrane to nucleus. The Journal of Pathology. 202(3). 313–320.
8.
Roberts, Ruth, Neil H. James, Sabina Cosulich, Susan C. Hasmall, & George M. Orphanides. (2001). Role of cytokines in non-genotoxic hepatocarcinogenesis: cause or effect?. Toxicology Letters. 120(1-3). 301–306.
9.
Hasmall, Susan C., et al.. (2001). Mouse hepatocyte response to peroxisome proliferators: dependency on hepatic nonparenchymal cells and peroxisome proliferator activated receptor α (PPARα). Archives of Toxicology. 75(6). 357–361.
10.
Holden, Peter R., Neil H. James, A. Nigel Brooks, et al.. (2000). Identification of a possible association between carbon tetrachloride-induced hepatotoxicity and interleukin-8 expression. Journal of Biochemical and Molecular Toxicology. 14(5). 283–290.
11.
Roberts, Ruth, Neil H. James, Susan C. Hasmall, et al.. (2000). Apoptosis and proliferation in nongenotoxic carcinogenesis: species differences and role of PPARα. Toxicology Letters. 112-113. 49–57.
12.
Hasmall, Susan C., Neil H. James, Neil Macdonald, et al.. (2000). Suppression of mouse hepatocyte apoptosis by peroxisome proliferators: role of PPARα and TNFα. Mutation research. Fundamental and molecular mechanisms of mutagenesis. 448(2). 193–200.
13.
Hasmall, Susan C., Neil H. James, Neil Macdonald, Anthony R. Soames, & Ruth Roberts. (2000). Species differences in response to diethylhexylphthalate: suppression of apoptosis, induction of DNA synthesis and peroxisome proliferator activated receptor alpha-mediated gene expression. Archives of Toxicology. 74(2). 85–91.
14.
Hasmall, Susan C., Neil H. James, Neil Macdonald, et al.. (1999). Suppression of apoptosis and induction of DNA synthesis in vitro by the phthalate plasticizers monoethylhexylphthalate (MEHP) and diisononylphthalate (DINP): a comparison of rat and human hepatocytes in vitro. Archives of Toxicology. 73(8-9). 451–456.
15.
James, Neil H., Jason H. Gill, Richard Brindle, et al.. (1998). Peroxisome proliferator-activated receptor (PPAR) alpha-regulated growth responses and their importance to hepatocarcinogenesis. Toxicology Letters. 102-103. 91–96.
16.
Tugwood, Jonathan, Peter R. Holden, Neil H. James, Rebecca A. Prince, & Ruth Roberts. (1998). A peroxisome proliferator-activated receptor-alpha (PPARα) cDNA cloned from guinea-pig liver encodes a protein with similar properties to the mouse PPARα: implications for species differences in responses to peroxisome proliferators. Archives of Toxicology. 72(3). 169–177.
17.
James, Neil H. & Ruth Roberts. (1996). Species differences in response to peroxisome proliferators correlate in vitro with induction of DNA synthesis rather than suppression of apoptosis. Carcinogenesis. 17(8). 1623–1632.
18.
Roberts, Ruth, Anthony R. Soames, Jason H. Gill, Neil H. James, & Eric B. Wheeldon. (1995). Non-genotoxic hepatocarcinogenesis stimulate DNA synthesis and their withdrawal induces apoptosis, but in different hepatocyte populations. Carcinogenesis. 16(8). 1693–1698.
19.
Roberts, Ruth, Anthony R. Soames, Neil H. James, Jason H. Gill, & Eric B. Wheeldon. (1995). Dosing-Induced Stress Causes Hepatocyte Apoptosis in Rats Primed by the Rodent Nongenotoxic Hepatocarcinogen Cyproterone Acetate. Toxicology and Applied Pharmacology. 135(2). 192–199.
20.
James, Neil H., et al.. (1992). An in vitro model of rodent nongenotoxic hepatocarcinogenesis. Experimental Cell Research. 203(2). 407–419.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.
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